Maturation of kainic acid seizure-brain damage syndrome in the rat. i. clinical, electrographic and metabolic observations
暂无分享,去创建一个
[1] Y. Ben-Ari. Limbic seizure and brain damage produced by kainic acid: Mechanisms and relevance to human temporal lobe epilepsy , 1985, Neuroscience.
[2] Y. Ben‐Ari,et al. Usefulness of parenteral kainic acid as a model of temporal lobe epilepsy. , 1984, Revue d'electroencephalographie et de neurophysiologie clinique.
[3] Y. Ben‐Ari,et al. Maturation of kainic acid seizure-brain damage syndrome in the rat. III. Postnatal development of kainic acid binding sites in the limbic system , 1984, Neuroscience.
[4] Y. Ben‐Ari,et al. Maturation of kainic acid seizure-brain damage syndrome in the rat. II. Histopathological sequelae , 1984, Neuroscience.
[5] N. Geschwind,et al. Temporal lobe epilepsy , 1984, Neurology.
[6] Y. Ben‐Ari,et al. Kainic acid seizure syndrome and binding sites in developing rats. , 1984, Brain research.
[7] S. Kish,et al. Kainic acid induced seizures: Neurochemical and histopathological changes , 1983, Neuroscience.
[8] D. Hopkins,et al. Direct amygdaloid projections to the dorsal motor nucleus of the vagus nerve: a light and electron microscopic study in the rat , 1983, Brain Research.
[9] S. Feldblum,et al. Role of the amygdala in development of hippocampal kindling in the rat , 1983, Experimental Neurology.
[10] D. Kristt. Acetylcholinesterase in the ventrobasal thalamus: Transience and patterning during ontogenesis , 1983, Neuroscience.
[11] Y. Ben-Ari,et al. Autoradiographic visualization of [3H]kainic acid receptor subtypes in the rat hippocampus , 1983, Neuroscience Letters.
[12] T. Scott,et al. The postnatal metabolic development of the nucleus commissuralis and nucleus medialis of the nucleus tractus solitarius. , 1983, Brain research.
[13] E. Cherubini,et al. Behavioral and electrographic patterns induced by systemic administration of kainic acid in developing rats. , 1983, Brain research.
[14] D. Riche,et al. Amygdaloid lesion increases the toxicity of intrahippocampal kainic acid injection and reduces the late occurrence of spontaneous recurrent seizures in rats , 1983, Brain Research.
[15] O. P. Ottersen,et al. Intra-amygdaloid injections of kainic acid: Regional metabolic changes and their relation to the pathological alterations , 1983, Neuroscience.
[16] J. Mcculloch,et al. A potential error in modifications of the [14C]2-deoxyglucose technique , 1983, Brain Research.
[17] J. Wamsley,et al. Autoradiographic localization of high-affinity [3H]kainic acid binding sites in the rat forebrain. , 1983, European journal of pharmacology.
[18] C. Cotman,et al. The distribution of [3H]kainic acid binding sites in rat CNS as determined by autoradiography , 1982, Brain Research.
[19] E. Cherubini,et al. A simple method for implanting electrodes in freely moving neonatal rats , 1982, Journal of Neuroscience Methods.
[20] D. Riche,et al. Long-term effects of intrahippocampal kainic acid injection in rats: a method for inducing spontaneous recurrent seizures. , 1982, Electroencephalography and clinical neurophysiology.
[21] S. Deadwyler,et al. Kainic acid produces depolarization of CA3 pyramidal cells in the in vitro hippocampal slice , 1981, Brain Research.
[22] R. C. Collins,et al. Kainic acid induced limbic seizures: metabolic, behavioral, electroencephalographic and neuropathological correlates , 1981, Brain Research.
[23] Y. Ben-Ari,et al. Electrographic, clinical and pathological alterations following systemic administration of kainic acid, bicuculline or pentetrazole: Metabolic mapping using the deoxyglucose method with special reference to the pathology of epilepsy , 1981, Neuroscience.
[24] S. D. Glick,et al. The ontogeny of hippocampal energy metabolism , 1981, Brain Research.
[25] C. Pilgrim,et al. Improving the Resolution of the 2-Deoxy-D-Glucose Method 1 , 2 , 1981, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.
[26] J. Nadler,et al. Kainic acid neurotoxicity toward hippocampal formation: Dependence on specific excitatory pathways , 1980, Brain Research.
[27] S. D. Glick,et al. Intraperitoneal administration and other modifications of the 2-deoxy-d-glucose technique , 1980, Brain Research.
[28] O. Ottersen,et al. The role of epileptic activity in hippocampal and ‘remote’ cerebral lesions induced by kainic acid , 1980, Brain Research.
[29] J. Price,et al. Widespread patterns of neuronal damage following systemic or intracerebral injections of kainic acid: A histological study , 1980, Neuroscience.
[30] O. P. Ottersen,et al. Injections of kainic acid into the amygdaloid complex of the rat: An electrographic, clinical and histological study in relation to the pathology of epilepsy , 1980, Neuroscience.
[31] S. Bayer,et al. Development of the hippocampal region in the rat II. Morphogenesis during embryonic and early postnatal life , 1980, The Journal of comparative neurology.
[32] J. Talairach,et al. Role du noyau amygdalien dans la survenue de manifestations oro-alimentaires au cours des crises épileptiques chez l'homme* , 1979 .
[33] J. Olney,et al. Effects of morphine or naloxone on kainic acid neurotoxicity. , 1979, Life sciences.
[34] J. Holaday,et al. Endorphins may function in heat adaptation. , 1978, Proceedings of the National Academy of Sciences of the United States of America.
[35] J. Zimmer,et al. Laminar differentiation of the hippocampus, fascia dentata and subiculum in developing rats, observed with the timm sulphide silver method , 1978, The Journal of comparative neurology.
[36] J. Holaday,et al. Unique behavioral effects of beta endorphin and their relationship to thermoregulation and hypothalamic function. , 1978, Life sciences.
[37] C. Cotman,et al. Intraventricular kainic acid preferentially destroys hippocampal pyramidal cells , 1978, Nature.
[38] J. Aicardi,et al. Convulsive Disorders in the First Year of Life: Neurological and Mental Outcome and Mortality , 1978, Epilepsia.
[39] F. Sharp,et al. Autoradiographic maps of regional brain glucose consumption in resting, awake rats using [14c] 2‐deoxyglucose , 1978, The Journal of comparative neurology.
[40] W. Cowan,et al. An autoradiographic study of the development of the entorhinal and commissural afferents to the dentate gyrus of the Rat , 1977, The Journal of comparative neurology.
[41] M. Reivich,et al. THE [14C]DEOXYGLUCOSE METHOD FOR THE MEASUREMENT OF LOCAL CEREBRAL GLUCOSE UTILIZATION: THEORY, PROCEDURE, AND NORMAL VALUES IN THE CONSCIOUS AND ANESTHETIZED ALBINO RAT 1 , 1977, Journal of neurochemistry.
[42] G. Lynch,et al. Development of afferent lamination in the fascia dentata of the rat , 1977, Brain Research.
[43] G. Dahlquist,et al. The Rate of Cerebral Utilization of Glucose, Ketone Bodies, and Oxygen: A Comparative in Vivo Study of Infant and Adult Rats , 1976, Pediatric Research.
[44] H. Gastaut,et al. Relative Frequency of Different Types of Epilepsy: A Study Employing the Classification of the International League Against Epilepsy , 1975, Epilepsia.
[45] H. Kraus,et al. Developmental Changes of Cerebral Ketone Body Utilization in Human Infants , 1974, Hoppe-Seyler's Zeitschrift fur physiologische Chemie.
[46] R. Racine,et al. Modification of seizure activity by electrical stimulation. II. Motor seizure. , 1972, Electroencephalography and clinical neurophysiology.
[47] J. Aicardi,et al. Convulsive Status Epilepticus in Infants and Children , 1970, Epilepsia.
[48] G. V. Goddard,et al. A permanent change in brain function resulting from daily electrical stimulation. , 1969, Experimental neurology.
[49] S. Jarman,et al. Topographic brain chemistry: R.L. FRIEDE. Academic Press, New York, 1966. 543 pp. $22 , 1967 .
[50] J H Margerison,et al. Epilepsy and the temporal lobes. A clinical, electroencephalographic and neuropathological study of the brain in epilepsy, with particular reference to the temporal lobes. , 1966, Brain : a journal of neurology.
[51] S. C. Wang,et al. Intravenous, cortical and intraventricular dose-effect relationship of pentylenetetrazol, picrotoxin and deslanoside in dogs. , 1962, Electroencephalography and clinical neurophysiology.
[52] W. Scholz. The Contribution of Patho‐Anatomical Research to the Problem of Epilepsy , 1959 .
[53] N. Malamud,et al. Clinical significance of sclerosis of the cornu ammonis: ictal psychic phenomena. , 1953, A.M.A. archives of neurology and psychiatry.
[54] G. Capovilla,et al. Nosological classification of epilepsies in the first three years of life. , 1983, Progress in clinical and biological research.
[55] G. le gal la Salle. Amygdaloid organization related to the kindling effect. , 1982, Electroencephalography and clinical neurophysiology. Supplement.
[56] G. Lynch,et al. Development of cholinergic innervation in the hippocampal formation of the rat. I. Histochemical demonstration of acetylcholinesterase activity. , 1974, Developmental biology.
[57] J Bancaud,et al. Depth Recordings in Man in Temporal Lobe Epilepsy , 1973 .
[58] M. Falconer. The pathological substrate of temporal lobe epilepsy. , 1970, Guy's Hospital reports.
[59] Falconer Ma. The pathological substrate of temporal lobe epilepsy. , 1970 .
[60] J. E. Cremer. Journal of Cerebral Blood Flow and Metabolism Substrate Utilization and Brain Development , 2022 .