Network analysis reveals a role of the hippocampus in absence seizures: The effects of a cannabinoid agonist
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G. Luijtelaar | I. Sysoev | M. Sysoeva | L. Vinogradova | R. Ngomba | G. Kuznetsova | C. Rijn | A. Grishchenko
[1] G. Biagini,et al. Is the peroxisome proliferator-activated receptor gamma a putative target for epilepsy treatment? Current evidence and future perspectives. , 2022, Pharmacology & therapeutics.
[2] G. Luijtelaar,et al. Thalamo-Cortical and Thalamo-Thalamic Coupling During Sleep and Wakefulness in Rats , 2021, Brain Connect..
[3] G. Luijtelaar,et al. Altered SWD stopping mechanism in WAG/Rij rats subchronically treated with the cannabinoid agonist R(+)WIN55,212-2 , 2019, Epilepsy & Behavior.
[4] H. Stroink,et al. A network approach to investigate the bi-hemispheric synchrony in absence epilepsy , 2019, Clinical Neurophysiology.
[5] A. Lyon,et al. Dosage Related Efficacy and Tolerability of Cannabidiol in Children With Treatment-Resistant Epileptic Encephalopathy: Preliminary Results of the CARE-E Study , 2019, Front. Neurol..
[6] Ilya V. Sysoev,et al. The Brain Network in a Model of Thalamocortical Dysrhythmia , 2019, Brain Connect..
[7] I. Boileau,et al. A systematic review of phytocannabinoid exposure on the endocannabinoid system: Implications for psychosis , 2019, European Neuropsychopharmacology.
[8] H. Pape,et al. Regional specificity of cortico-thalamic coupling strength and directionality during waxing and waning of spike and wave discharges , 2019, Scientific Reports.
[9] Z. Maglóczky,et al. Alterations in hippocampal and cortical densities of functionally different interneurons in rat models of absence epilepsy , 2018, Epilepsy Research.
[10] Vincenzo Crunelli,et al. Cortical drive and thalamic feed-forward inhibition control thalamic output synchrony during absence seizures , 2018, Nature Neuroscience.
[11] Marina V. Sysoeva,et al. Modeling spike-wave discharges by a complex network of neuronal oscillators , 2018, Neural Networks.
[12] T. Medvedeva,et al. Optimization of Granger causation method parameters for the study of limbic epilepsy , 2018 .
[13] C. D. De Zeeuw,et al. Synchronicity and Rhythmicity of Purkinje Cell Firing during Generalized Spike-and-Wave Discharges in a Natural Mouse Model of Absence Epilepsy , 2017, Front. Cell. Neurosci..
[14] Shuyang Huang,et al. Quantify neuromagnetic network changes from pre-ictal to ictal activities in absence seizures , 2017, Neuroscience.
[15] Benjamin J. Whalley,et al. Therapeutic effects of cannabinoids in animal models of seizures, epilepsy, epileptogenesis, and epilepsy-related neuroprotection , 2017, Epilepsy & Behavior.
[16] Orrin Devinsky,et al. Cannabinoids in treatment-resistant epilepsy: A review , 2017, Epilepsy & Behavior.
[17] V. Kitchigina,et al. Endocannabinoid-dependent protection against kainic acid-induced long-term alteration of brain oscillations in guinea pigs , 2017, Brain Research.
[18] L. Vinogradova,et al. The cannabinoid receptor agonist WIN55.212 reduces consequences of status epilepticus in rats , 2016, Neuroscience.
[19] Naoki Yamawaki,et al. A Corticocortical Circuit Directly Links Retrosplenial Cortex to M2 in the Mouse , 2016, The Journal of Neuroscience.
[20] Kara L. Agster,et al. Subcortical connections of the perirhinal, postrhinal, and entorhinal cortices of the rat. II. efferents , 2016, Hippocampus.
[21] E. Luijtelaar,et al. Dynamics of directional coupling underlying spike-wave discharges , 2016, Neuroscience.
[22] B. Bezruchko,et al. Choosing the optimal model parameters for Granger causality in application to time series with main timescale , 2016 .
[23] I. Sysoev,et al. [Thalamo-Cortical Mechanisms of Initiation, Maintenance and Termination of Spike-wave Discharges at WAG/Rij rats]. , 2016, Zhurnal vyssheĭ nervnoĭ deiatelnosti imeni I P Pavlova.
[24] C. V. van Rijn,et al. Unilateral and Bilateral Cortical Resection: Effects on Spike-Wave Discharges in a Genetic Absence Epilepsy Model , 2015, PloS one.
[25] I. Sysoev,et al. Detecting changes in coupling with Granger causality method from time series with fast transient processes , 2015 .
[26] C. V. van Rijn,et al. Long-term disease-modifying effect of the endocannabinoid agonist WIN55,212-2 in a rat model of audiogenic epilepsy , 2015, Pharmacological reports : PR.
[27] Gilles van Luijtelaar,et al. Dynamics of networks during absence seizure's on- and offset in rodents and man , 2015, Front. Physiol..
[28] Mukesh Dhamala,et al. Application of high‐frequency Granger causality to analysis of epileptic seizures and surgical decision making , 2014, Epilepsia.
[29] Marina V. Sysoeva,et al. Application of adaptive nonlinear Granger causality: Disclosing network changes before and after absence seizure onset in a genetic rat model , 2014, Journal of Neuroscience Methods.
[30] G. van Luijtelaar,et al. Progress and outlooks in a genetic absence epilepsy model (WAG/Rij). , 2014, Current medicinal chemistry.
[31] M. Avoli,et al. Is there such a thing as "generalized" epilepsy? , 2014, Advances in experimental medicine and biology.
[32] F. Hyder,et al. Anti-epileptogenesis: Electrophysiology, diffusion tensor imaging and behavior in a genetic absence model , 2013, Neurobiology of Disease.
[33] Benjamin J. Whalley,et al. CB1 agonists, locally applied to the cortico-thalamic circuit of rats with genetic absence epilepsy, reduce epileptic manifestations , 2013, Epilepsy Research.
[34] S. Nasoohi,et al. Dual Role of PPAR-γ in Induction and Expression of Behavioral Sensitization to Cannabinoid Receptor Agonist WIN55,212-2 , 2013, NeuroMolecular Medicine.
[35] F. H. Lopes da Silva,et al. Epileptic Neuronal Networks: Methods of Identification and Clinical Relevance , 2012, Front. Neurol..
[36] D. Smirnov,et al. Spurious causalities due to low temporal resolution: Towards detection of bidirectional coupling from time series , 2012 .
[37] Annika Lüttjohann,et al. The dynamics of cortico-thalamo-cortical interactions at the transition from pre-ictal to ictal LFPs in absence epilepsy , 2012, Neurobiology of Disease.
[38] T. Freund,et al. Multiple functions of endocannabinoid signaling in the brain. , 2012, Annual review of neuroscience.
[39] Massimo Avoli,et al. A brief history on the oscillating roles of thalamus and cortex in absence seizures , 2012, Epilepsia.
[40] Marina V. Sysoeva,et al. Mathematical modeling of encephalogram dynamics during epileptic seizure , 2012 .
[41] A. Coenen,et al. Cytokines and Absence Seizures in a Genetic Rat Model , 2012, Neurophysiology.
[42] Pauly P. W. Ossenblok,et al. Space–time network connectivity and cortical activations preceding spike wave discharges in human absence epilepsy: a MEG study , 2011, Medical & Biological Engineering & Computing.
[43] Evgenia Sitnikova,et al. On the Origin and Suddenness of Absences in Genetic Absence Models , 2011, Clinical EEG and neuroscience.
[44] John T. E. Richardson,et al. Eta Squared and Partial Eta Squared as Measures of Effect Size in Educational Research. , 2011 .
[45] Stephen P. H. Alexander,et al. International Union of Basic and Clinical Pharmacology. LXXIX. Cannabinoid Receptors and Their Ligands: Beyond CB1 and CB2 , 2010, Pharmacological Reviews.
[46] S. Gaetani,et al. WAG/Rij rats show a reduced expression of CB1 receptors in thalamic nuclei and respond to the CB1 receptor agonist, R(+)WIN55,212‐2, with a reduced incidence of spike‐wave discharges , 2010, Epilepsia.
[47] P. Ossenblok,et al. Onset and propagation of spike and slow wave discharges in human absence epilepsy: A MEG study , 2009, Epilepsia.
[48] Max Kleiman-Weiner,et al. A gain in GABAA receptor synaptic strength in thalamus reduces oscillatory activity and absence seizures , 2009, Proceedings of the National Academy of Sciences.
[49] Evgenia Sitnikova,et al. Electroencephalographic precursors of spike-wave discharges in a genetic rat model of absence epilepsy: Power spectrum and coherence EEG analyses , 2009, Epilepsy Research.
[50] C. Segebarth,et al. Identifying Neural Drivers with Functional MRI: An Electrophysiological Validation , 2008, PLoS biology.
[51] A. Karson,et al. Hippocampal kindling in rats with absence epilepsy resembles amygdaloid kindling , 2008, Epilepsy Research.
[52] H. Siebner,et al. Simultaneous EEG‐fMRI in drug‐naive children with newly diagnosed absence epilepsy , 2008, Epilepsia.
[53] Joshua E. Motelow,et al. Early treatment suppresses the development of spike‐wave epilepsy in a rat model , 2008, Epilepsia.
[54] G. van Luijtelaar,et al. The Effect of Generalized Absence Seizures on the Progression of Kindling in the Rat , 2007, Epilepsia.
[55] P. Somogyi,et al. Neuronal Diversity in GABAergic Long-Range Projections from the Hippocampus , 2007, The Journal of Neuroscience.
[56] S. Charpier,et al. Deep Layer Somatosensory Cortical Neurons Initiate Spike-and-Wave Discharges in a Genetic Model of Absence Seizures , 2007, The Journal of Neuroscience.
[57] G. Luijtelaar,et al. Absence seizures are reduced by the enhancement of GABA-ergic inhibition in the hippocampus in WAG/Rij rats , 2007, Neuroscience Letters.
[58] Gilles van Luijtelaar,et al. Time–frequency analysis of spike-wave discharges using a modified wavelet transform , 2006, Journal of Neuroscience Methods.
[59] Terence J O'Brien,et al. Cellular and network mechanisms of genetically-determined absence seizures. , 2005, Thalamus & related systems.
[60] C. Meshul,et al. Glutamate and GABA immunocytochemical electron microscopy in the hippocampal dentate gyrus of normal and genetic absence epilepsy rats , 2005, Brain Research.
[61] F. H. Lopes da Silva,et al. Evolving concepts on the pathophysiology of absence seizures: the cortical focus theory. , 2005, Archives of neurology.
[62] F. L. D. Silva,et al. Dynamics of non-convulsive epileptic phenomena modeled by a bistable neuronal network , 2004, Neuroscience.
[63] D. Tucker,et al. Are “Generalized” Seizures Truly Generalized? Evidence of Localized Mesial Frontal and Frontopolar Discharges in Absence , 2004, Epilepsia.
[64] B. Alger. Endocannabinoids and Their Implications for Epilepsy , 2004, Epilepsy currents.
[65] G. Rangarajan,et al. Analyzing multiple nonlinear time series with extended Granger causality , 2004, nlin/0405016.
[66] B. Martin,et al. The Endogenous Cannabinoid System Regulates Seizure Frequency and Duration in a Model of Temporal Lobe Epilepsy , 2003, Journal of Pharmacology and Experimental Therapeutics.
[67] S. Roper,et al. Cannabinoid receptor-1 activation suppresses inhibitory synaptic activity in human dentate gyrus , 2003, Neuropharmacology.
[68] J. Huguenard,et al. Dynamic GABAA Receptor Subtype-Specific Modulation of the Synchrony and Duration of Thalamic Oscillations , 2003, The Journal of Neuroscience.
[69] W. Hesse,et al. The use of time-variant EEG Granger causality for inspecting directed interdependencies of neural assemblies , 2003, Journal of Neuroscience Methods.
[70] F. H. Lopes da Silva,et al. Cortical Focus Drives Widespread Corticothalamic Networks during Spontaneous Absence Seizures in Rats , 2002, The Journal of Neuroscience.
[71] A. Coenen,et al. Electrophysiological and pharmacological characteristics of two types of spike-wave discharges in WAG/Rij rats , 2001, Brain Research.
[72] N. Bowery,et al. Hippocampal extracellular amino acids and EEG spectral analysis in a genetic rat model of absence epilepsy , 2000, Neuropharmacology.
[73] T. Freund,et al. GABAergic interneurons are the targets of cannabinoid actions in the human hippocampus , 2000, Neuroscience.
[74] C. Deransart,et al. The role of basal ganglia in the control of generalized absence seizures , 1998, Epilepsy Research.
[75] M. Witter,et al. Entorhinal cortex of the rat: Cytoarchitectonic subdivisions and the origin and distribution of cortical efferents , 1998, Hippocampus.
[76] R. J. Meijer,et al. Collateral projections from the rat hippocampal formation to the lateral and medial prefrontal cortex , 1997, Hippocampus.
[77] W. Staines,et al. Efferent projections of the anterior perirhinal cortex in the rat , 1996, The Journal of comparative neurology.
[78] G. Buzsáki,et al. Lack of hippocampal involvement in a rat model of petit mal epilepsy , 1996, Epilepsy Research.
[79] A. Coenen,et al. Kappa opioid receptor agonists suppress absence seizures in WAG/Rij rats , 1995, Neuroscience Letters.
[80] Jacques Duysens,et al. Thalamic multiple-unit activity underlying spike-wave discharges in anesthetized rats , 1993, Brain Research.
[81] A. Coenen,et al. Endogenous opioid peptides in brain and pituitary of rats with absence epilepsy , 1992, Neuropeptides.
[82] T. Jay,et al. Distribution of hippocampal CA1 and subicular efferents in the prefrontal cortex of the rat studied by means of anterograde transport of Phaseolus vulgaris‐leucoagglutinin , 1991, The Journal of comparative neurology.
[83] Antoine Depaulis,et al. Mapping of spontaneous spike and wave discharges in Wistar rats with genetic generalized non-convulsive epilepsy , 1990, Brain Research.
[84] M. Herkenham,et al. Cannabinoid receptor localization in brain. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[85] A. Coenen,et al. Two types of electrocortical paroxysms in an inbred strain of rats , 1986, Neuroscience Letters.
[86] L. Swanson,et al. A direct projection from Ammon's horn to prefrontal cortex in the rat , 1981, Brain Research.
[87] James P. Crutchfield,et al. Geometry from a Time Series , 1980 .
[88] G. Schwarz. Estimating the Dimension of a Model , 1978 .