Sodium nitroprusside in patients with compromised left ventricular function undergoing coronary bypass: reduction of cardiac proinflammatory substances.

[1]  S. Zahler,et al.  Significant leukocyte and platelet retention during pulmonary passage after declamping of the aorta in CABG patients. , 1999, European journal of medical research.

[2]  S. Zahler,et al.  Sodium nitroprusside during coronary artery bypass grafting: evidence for an antiinflammatory action. , 1999, The Annals of thoracic surgery.

[3]  O. Kohmoto,et al.  IS013 Cellular Basis for the Acute Inhibitory Effects of IL-6-and TNF-α on Excitation-Contraction Coupling , 1999 .

[4]  S. Zahler,et al.  Acute cardiac inflammatory responses to postischemic reperfusion during cardiopulmonary bypass. , 1999, Cardiovascular research.

[5]  J. Cavaillon,et al.  Reduced ex vivo interleukin-8 production by neutrophils in septic and nonseptic systemic inflammatory response syndrome. , 1998, Blood.

[6]  S. Ishiwata,et al.  Postangioplasty restenosis: platelet activation and the coagulation-fibrinolysis system as possible factors in the pathogenesis of restenosis. , 1997, American heart journal.

[7]  J. Hammon,et al.  Basal nitric oxide expresses endogenous cardioprotection during reperfusion by inhibition of neutrophil-mediated damage after surgical revascularization. , 1997, The Journal of thoracic and cardiovascular surgery.

[8]  N. Maulik,et al.  Critical timing of nitric oxide supplementation in cardioplegic arrest and reperfusion. , 1996, Circulation.

[9]  G. von Bernuth,et al.  Effect of sodium nitroprusside on complement activation induced by cardiopulmonary bypass: a clinical and experimental study. , 1996, The Journal of thoracic and cardiovascular surgery.

[10]  S. Zahler,et al.  Nitric oxide mitigates leukocyte adhesion and vascular leak after myocardial ischemia. , 1996, Journal of molecular and cellular cardiology.

[11]  J. Fitch,et al.  Amplification of the inflammatory response: adhesion molecules associated with platelet/white cell responses. , 1996, Journal of cardiovascular pharmacology.

[12]  G. Hill,et al.  Aprotinin and methylprednisolone equally blunt cardiopulmonary bypass-induced inflammation in humans. , 1995, The Journal of thoracic and cardiovascular surgery.

[13]  AlbertSchömig,et al.  Cardiac Release of Cytokines and Inflammatory Responses in Acute Myocardial Infarction , 1995 .

[14]  T. Tanabe,et al.  [Effect of ulinastatin on the free radical during cardiopulmonary bypass]. , 1995, Masui. The Japanese journal of anesthesiology.

[15]  B. F. Becker,et al.  Nitric Oxide Accounts for Postischemic Cardioprotection Resulting from Angiotensin‐Converting Enzyme Inhibition: Indirect Evidence for a Radical Scavenger Effect in Isolated Guinea Pig Heart , 1995, Journal of cardiovascular pharmacology.

[16]  D. Smith,et al.  Respiratory function after cardiopulmonary bypass: a comparison of bubble and membrane oxygenators. , 1994, Journal of cardiothoracic and vascular anesthesia.

[17]  K. Miyagi,et al.  [Extravascular lung water in patients after cardiac surgery]. , 1993, [Zasshi] [Journal]. Nihon Kyobu Geka Gakkai.