Increased levels of the excitotoxin quinolinic acid in spinal cord following contusion injury
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[1] S. Markey,et al. 4-Chloro-3-hydroxyanthranilate, 6-chlorotryptophan and norharmane attenuate quinolinic acid formation by interferon-gamma-stimulated monocytes (THP-1 cells). , 1993, The Biochemical journal.
[2] S. Markey,et al. Mechanism of Delayed Increases in Kynurenine Pathway Metabolism in Damaged Brain Regions Following Transient Cerebral Ischemia , 1993, Journal of neurochemistry.
[3] M. Demitrack,et al. Quinolinic acid and kynurenine pathway metabolism in inflammatory and non-inflammatory neurological disease. , 1992, Brain : a journal of neurology.
[4] D. Jacobowitz,et al. Poliovirus induces indoleamine‐2,3‐dioxygenase and quinolinic acid synthesis in macaque brain , 1992, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[5] J. Wrathall,et al. Evidence that local non-NMDA receptors contribute to functional deficits in contusive spinal cord injury , 1992, Brain Research.
[6] E. Hall,et al. Biochemistry and pharmacology of lipid antioxidants in acute brain and spinal cord injury. , 1992, Journal of neurotrauma.
[7] C H Tator,et al. Review of the secondary injury theory of acute spinal cord trauma with emphasis on vascular mechanisms. , 1991, Journal of neurosurgery.
[8] A. Blight. Morphometric analysis of a model of spinal cord injury in guinea pigs, with behavioral evidence of delayed secondary pathology , 1991, Journal of the Neurological Sciences.
[9] D. McAdoo,et al. Excitatory amino acids rise to toxic levels upon impact injury to the rat spinal cord , 1991, Brain Research.
[10] D. Giulian,et al. Secretion of neurotoxins by mononuclear phagocytes infected with HIV-1 , 1990, Science.
[11] A. Blight,et al. Cutaneus trunci muscle reflex of the guinea pig , 1990, The Journal of comparative neurology.
[12] L. Noble,et al. Effects of competetive and non-competetive NMDA receptor antagonists in spinal cord injury , 1990 .
[13] C. Robertson,et al. Inhibition of mononuclear phagocytes reduces ischemic injury in the spinal cord , 1990, Annals of neurology.
[14] K. Spicer,et al. Vascular permeability in experimental spinal cord injury , 1985, Journal of the Neurological Sciences.
[15] E. Means,et al. The effect of laminectomy on spinal cord blood flow, energy metabolism and ATPase activity , 1985, Paraplegia.
[16] H. Wiśniewski,et al. Primary demyelination as a nonspecific consequence of a cell-mediated immune reaction , 1975, The Journal of experimental medicine.
[17] Blight Ar. Remyelination, revascularization, and recovery of function in experimental spinal cord injury. , 1993 .
[18] A. Blight. Delayed demyelination and macrophage invasion: a candidate for secondary cell damage in spinal cord injury. , 1985, Central nervous system trauma : journal of the American Paralysis Association.