Immunopathogenic Background of Pars Planitis
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[1] M. Mochizuki,et al. Suppression of IL-22-producing T helper 22 cells by RPE cells via PD-L1/PD-1 interactions. , 2013, Investigative ophthalmology & visual science.
[2] J. Fernández. Diagnosis and Treatment of Uveitis , 2013 .
[3] M. Mochizuki,et al. Mature dendritic cell suppression by IL-1 receptor antagonist on retinal pigment epithelium cells. , 2013, Investigative ophthalmology & visual science.
[4] M. Schwartz,et al. Orchestrated leukocyte recruitment to immune-privileged sites: absolute barriers versus educational gates , 2013, Nature Reviews Immunology.
[5] B. Takahashi,et al. Experimental models of autoimmune inflammatory ocular diseases. , 2012, Arquivos brasileiros de oftalmologia.
[6] J. Fung,et al. Myeloid suppressor cells induced by retinal pigment epithelial cells inhibit autoreactive T-cell responses that lead to experimental autoimmune uveitis. , 2012, Investigative ophthalmology & visual science.
[7] M. Mochizuki,et al. Suppression of interleukin-17-producing T-helper 17 cells by retinal pigment epithelial cells , 2011, Japanese Journal of Ophthalmology.
[8] M. Mochizuki,et al. Inhibition of B-cell activation by retinal pigment epithelium. , 2010, Investigative ophthalmology & visual science.
[9] J. Bernabeu-Wittel,et al. Características clínicas, epidemiológicas y asociación con el sistema HLA en pacientes con pars planitis , 2010 .
[10] C. Peters,et al. Acquisition of T Regulatory Function in Cathepsin L-Inhibited T Cells by Eye-Derived CTLA-2α during Inflammatory Conditions1 , 2009, The Journal of Immunology.
[11] N. Acharya,et al. Ranibizumab for refractory uveitis-related macular edema. , 2009, American journal of ophthalmology.
[12] A. Rosenthal,et al. Course and complications of intermediate uveitis. , 2009, Acta ophthalmologica Scandinavica.
[13] R. Nussenblatt,et al. CD4+Foxp3+ T-regulatory cells in noninfectious uveitis. , 2009, Archives of ophthalmology.
[14] Vanita Gupta,et al. Search for a functional glucocorticoid receptor in the mammalian lens. , 2009, Experimental eye research.
[15] H. Aburatani,et al. Retinal Pigment Epithelium-Derived CTLA-2α Induces TGFβ-Producing T Regulatory Cells12 , 2008, The Journal of Immunology.
[16] E. Ling,et al. Blood–retinal barrier in hypoxic ischaemic conditions: Basic concepts, clinical features and management , 2008, Progress in Retinal and Eye Research.
[17] Andrew D. Dick,et al. The dynamics of leukocyte infiltration in experimental autoimmune uveoretinitis , 2008, Progress in Retinal and Eye Research.
[18] M. Hudson,et al. Anterior and intermediate uveitis cases referred to a tertiary centre in Alberta. , 2007, Canadian journal of ophthalmology. Journal canadien d'ophtalmologie.
[19] J. Pulido,et al. Pars planitis: a 20-year study of incidence, clinical features, and outcomes. , 2007, American journal of ophthalmology.
[20] J. Forrester,et al. Inducible nitric oxide synthase isoform is a key mediator of leukostasis and blood-retinal barrier breakdown in diabetic retinopathy. , 2007, Investigative ophthalmology & visual science.
[21] J. Nickerson,et al. Hydrodynamic Vaccination with DNA Encoding an Immunologically Privileged Retinal Antigen Protects from Autoimmunity through Induction of Regulatory T Cells1 , 2007, The Journal of Immunology.
[22] K. Lew,et al. Interaction of retinal pigmented epithelial cells and CD4 T cells leads to T-cell anergy. , 2007, Investigative ophthalmology & visual science.
[23] R. Lascuraín,et al. Pars planitis is associated with an increased frequency of effector-memory CD57+ T cells , 2007, British Journal of Ophthalmology.
[24] H. Kaplan,et al. Minimally activated CD8 autoreactive T cells specific for IRBP express a high level of Foxp3 and are functionally suppressive. , 2007, Investigative ophthalmology & visual science.
[25] M. Zierhut,et al. High incidence of antibodies to lens proteins in sera from patients with uveitis , 2007, Graefe's Archive for Clinical and Experimental Ophthalmology.
[26] J. Forrester,et al. CD4+CD25+ T regulatory cells induced by LPS-activated bone marrow dendritic cells suppress experimental autoimmune uveoretinitis in vivo , 2007, Graefe's Archive for Clinical and Experimental Ophthalmology.
[27] M. Mochizuki,et al. Retinal and ciliary body pigment epithelium suppress activation of T lymphocytes via transforming growth factor beta. , 2006, Experimental eye research.
[28] M. Mochizuki,et al. B7+ iris pigment epithelial cells convert T cells into CTLA-4+, B7-expressing CD8+ regulatory T cells. , 2006, Investigative ophthalmology & visual science.
[29] J. Streilein,et al. Pigment epithelial growth factor suppresses inflammation by modulating macrophage activation. , 2006, Investigative ophthalmology & visual science.
[30] T. Hattori,et al. Supplementation of CD4+CD25+ regulatory T cells suppresses experimental autoimmune uveoretinitis , 2006, British Journal of Ophthalmology.
[31] A. Rothova,et al. Distinct cytokine and chemokine profiles in the aqueous of patients with uveitis and cystoid macular edema. , 2006, American journal of ophthalmology-glaucoma.
[32] R. Gress,et al. B7+ iris pigment epithelium induce CD8+ T regulatory cells; both suppress CTLA-4+ T cells , 2006, The Journal of Immunology.
[33] S. Sugita,et al. B7+ Iris Pigment Epithelium Induce CD8+ T Regulatory Cells; Both Suppress CTLA-4+ T Cells12 , 2006, The Journal of Immunology.
[34] F. Falciani,et al. Multiplex bead immunoassay analysis of aqueous humor reveals distinct cytokine profiles in uveitis. , 2005, Investigative ophthalmology & visual science.
[35] Y. Yu,et al. CD8brightCD56+ T Cells Are Cytotoxic Effectors in Patients with Active Behçet’s Uveitis1 , 2005, The Journal of Immunology.
[36] Douglas A Jabs,et al. Standardization of uveitis nomenclature for reporting clinical data. Results of the First International Workshop. , 2005, American journal of ophthalmology.
[37] P. Sfikakis,et al. Regression of neovascular age-related macular degeneration following infliximab therapy. , 2005, American journal of ophthalmology-glaucoma.
[38] D. Gritz,et al. Incidence and prevalence of uveitis in Northern California , 2004 .
[39] A. Rothova,et al. Endothelial cell activation and hypercoagulability in ocular Behçet's disease. , 2004, American journal of ophthalmology.
[40] J. Schwartzkopff,et al. CTLA-4+CD8+ T Cells That Encounter B7-2+ Iris Pigment Epithelial Cells Express Their Own B7-2 to Achieve Global Suppression of T Cell Activation1 , 2004, The Journal of Immunology.
[41] D. Gritz,et al. Incidence and prevalence of uveitis in Northern California; the Northern California Epidemiology of Uveitis Study. , 2004, Ophthalmology.
[42] A. Dick,et al. Systemic CD4+ T cell phenotype and activation status in intermediate uveitis , 2004, British Journal of Ophthalmology.
[43] D. Green,et al. Presence of a transcriptionally active glucocorticoid receptor α in Lens epithelial cells , 2003 .
[44] J. Streilein. Ocular immune privilege: therapeutic opportunities from an experiment of nature , 2003, Nature Reviews Immunology.
[45] C. Gorodezky,et al. Classic Pars Planitis: strong correlation of class II genes with gender and some clinical features in Mexican Mestizos. , 2003, Human immunology.
[46] S. Sugita,et al. Iris Pigment Epithelium Expressing CD86 (B7-2) Directly Suppresses T Cell Activation In Vitro via Binding to Cytotoxic T Lymphocyte–associated Antigen 4 , 2003, The Journal of experimental medicine.
[47] J. Streilein,et al. Participation of pigment epithelium in ocular immune privilege. 3. Epithelia cultured from iris, ciliary body, and retina suppress T-cell activation by partially non-overlapping mechanisms , 2003, Ocular immunology and inflammation.
[48] S. Urbaniak,et al. Genetic Polymorphism of HLA DR in a Scottish Population of Patients with Pars Planitis , 2002, European journal of ophthalmology.
[49] G. Adamus,et al. EXPERIMENTAL AUTOIMMUNE UVEITIDES: MULTIPLE ANTIGENS, DIVERSE DISEASES , 2002, International reviews of immunology.
[50] K. Geboes,et al. An immunohistochemical study of the ‘snowbank’ in a case of pars planitis , 2002, Ocular immunology and inflammation.
[51] A. Dick,et al. CD69 expression on peripheral CD4+ T cells parallels disease activity and is reduced by mycophenolate mofetil therapy in uveitis. , 2001, Investigative ophthalmology & visual science.
[52] H. Kaplan,et al. The association of HLA class II with pars planitis. , 2001, American journal of ophthalmology.
[53] R. Solana,et al. Increased expression of NK cell markers on T lymphocytes in aging and chronic activation of the immune system reflects the accumulation of effector/senescent T cells , 2001, Mechanisms of Ageing and Development.
[54] M. Calonge,et al. Pars planitis: epidemiology, treatment, and association with multiple sclerosis , 2001, Ocular immunology and inflammation.
[55] T. Kezuka,et al. Participation of pigment epithelium of iris and ciliary body in ocular immune privilege. 2. Generation of TGF-beta-producing regulatory T cells. , 2000, Investigative ophthalmology & visual science.
[56] T. Kivelä,et al. Search for autoantibodies against the HNK-1 carbohydrate epitope in the human eye in intermediate uveitis. , 2000, Acta ophthalmologica.
[57] M. Mańczak,et al. Epitope recognition and T cell receptors in recurrent autoimmune anterior uveitis in Lewis rats immunized with myelin basic protein , 2000, Journal of Neuroimmunology.
[58] M. Takeuchi,et al. Participation of pigment epithelium of iris and ciliary body in ocular immune privilege. 1. Inhibition of T-cell activation in vitro by direct cell-to-cell contact. , 2000, Investigative ophthalmology & visual science.
[59] C. Chan,et al. Inhibition of experimental melanin protein-induced uveitis (EMIU) by targeting nitric oxide via phosphatidylcholine-specific phospholipase C. , 1999, Journal of autoimmunity.
[60] S. Lightman,et al. Increased CD4+ expression and decreased IL-10 in the anterior chamber in idiopathic uveitis. , 1999, Investigative ophthalmology & visual science.
[61] A. Rothova,et al. Soluble ICAM-1 serum levels in patients with intermediate uveitis , 1999, The British journal of ophthalmology.
[62] N. Bressler,et al. Pars planitis: clinical features and class II HLA associations. , 1999, Ophthalmology.
[63] G. Adamus,et al. EAE TCR motifs and antigen recognition in myelin basic protein-induced anterior uveitis in Lewis rats. , 1998, Journal of immunology.
[64] C. Hack,et al. Elevated serum IL-8 levels are associated with disease activity in idiopathic intermediate uveitis , 1998, The British journal of ophthalmology.
[65] J. E. Dickerson,et al. Steroid-induced cataract: new perspective from in vitro and lens culture studies. , 1997, Experimental eye research.
[66] J. Hillert,et al. Distribution of HLA‐Dw2 in optic neuritis and multiple sclerosis indicates heterogeneity , 1996, Acta neurologica Scandinavica.
[67] C. Foster,et al. Referral patterns of uveitis in a tertiary eye care center. , 1996, Archives of ophthalmology.
[68] C. Chan,et al. Endotoxin induced uveitis in the mouse: susceptibility and genetic control. , 1995, Experimental eye research.
[69] K. Barton,et al. The kinetics of cytokine mRNA expression in the retina during experimental autoimmune uveoretinitis. , 1995, Cellular immunology.
[70] S. Lightman,et al. Oligoclonal activation of CD4+ T lymphocytes in posterior uveitis , 1995, Clinical and experimental immunology.
[71] T. Rème,et al. CD57+ T lymphocytes are derived from CD57− precursors by differentiation occurring in late immune responses , 1994, European journal of immunology.
[72] J. Folk,et al. The association of HLA-B8, B51, DR2, and multiple sclerosis in pars planitis. , 1993, Ophthalmology.
[73] J. Folk,et al. Long-term visual outcome and complications associated with pars planitis. , 1993, Ophthalmology.
[74] R. Nussenblatt,et al. Uveitis: Fundamentals and Clinical Practice , 1988 .
[75] A. Friedman,et al. A clinical and histopathological review of intermediate uveitis ("pars planitis"). , 1988, Bulletin of the New York Academy of Medicine.
[76] D. Char,et al. Activated T lymphocytes in uveitis. , 1988, The British journal of ophthalmology.
[77] R. Nussenblatt,et al. Clinical and immunopathological studies of pars planitis in a family. , 1988, The British journal of ophthalmology.
[78] R. E. Smith,et al. Changing patterns of uveitis. , 1987, American journal of ophthalmology.
[79] R. Nussenblatt,et al. International Uveitis Study Group recommendations for the evaluation of intraocular inflammatory disease. , 1987, American journal of ophthalmology.
[80] D. Wakefield,et al. Uveitis: aetiology and disease associations in an Australian population. , 1986, Australian and New Zealand journal of ophthalmology.
[81] R. Nussenblatt,et al. Kinetics of T-lymphocyte subsets in the eyes of Lewis rats with experimental autoimmune uveitis. , 1985, Cellular immunology.
[82] R. Nussenblatt,et al. T-lymphocyte subsets in experimental autoimmune uveitis. , 1985, Clinical immunology and immunopathology.
[83] R. Brockhurst,et al. Neovascular fundus abnormalities in peripheral uveitis. , 1982, Archives of ophthalmology.
[84] W. Green,et al. Pathology of pars planitis. , 1978, American journal of ophthalmology.
[85] W. Sibley,et al. Uveitis, perivenous sheathing and multiple sclerosis , 1978, Neurology.
[86] R. E. Smith,et al. Complications of chronic cyclitis. , 1976, American journal of ophthalmology.
[87] R. Pruett,et al. Fluorescein angiography of peripheral uveitis. , 1974, American journal of ophthalmology.
[88] R. Porter. Uveitis in association with multiple sclerosis. , 1972, The British journal of ophthalmology.
[89] C. L. Giles. Peripheral uveitis in patients with multiple sclerosis. , 1970, American journal of ophthalmology.
[90] C. Schepens,et al. Uveitis. IV. Peripheral uveitis: the complications of retinal detachment. , 1968, Archives of ophthalmology.
[91] J. Sánchez-Román,et al. [Clinical and epidemiological features and HLA association in patients with pars planitis]. , 2010, Medicina clínica (Ed. impresa).
[92] S. Sugita,et al. Role of ocular pigment epithelial cells in immune privilege , 2009, Archivum Immunologiae et Therapiae Experimentalis.
[93] H. Aburatani,et al. Retinal pigment epithelium-derived CTLA-2alpha induces TGFbeta-producing T regulatory cells. , 2008, Journal of immunology.
[94] D. Green,et al. Presence of a transcriptionally active glucocorticoid receptor alpha in lens epithelial cells. , 2003, Investigative Ophthalmology and Visual Science.
[95] L. Goodglick,et al. BetaB1-crystallin: identification of a candidate ciliary body uveitis antigen. , 2003, Investigative ophthalmology & visual science.
[96] E. Bettelli,et al. Arch.Immunol.Ther.Exp. , 2000 .
[97] Dennis P. Han,et al. The association of HLA-DR15 and intermediate uveitis. , 1997, American journal of ophthalmology.
[98] J. Winston,et al. Causes of uveitis in the general practice of ophthalmology. UCLA Community-Based Uveitis Study Group. , 1996, American journal of ophthalmology.
[99] J. Winston,et al. Causes of Uveitis in the General Practice of Ophthalmology , 1996 .
[100] S. Yoser,et al. Pathology of intermediate uveitis. , 1992, Developments in ophthalmology.
[101] J. Forrester,et al. Immunocytochemical analysis of blood lymphocytes in uveitis , 1992, Eye.
[102] R. Nussenblatt,et al. Immunology of intermediate uveitis. , 1992, Developments in ophthalmology.
[103] D. E. Henderly,et al. Pars planitis. , 1986, Transactions of the ophthalmological societies of the United Kingdom.
[104] R. Nussenblatt,et al. Long-term culture of Muller cells from adult rats in the presence of activated lymphocytes/monocytes products. , 1985, Current eye research.
[105] Y. de Kozak,et al. S antigen-induced experimental autoimmune uveo-retinitis in rats. , 1981, Current eye research.
[106] W. Green,et al. Fibroglial proliferation in pars planitis. , 1975, Transactions of the ophthalmological societies of the United Kingdom.