NKG2A expression and impaired function of NK cells in patients with new onset of Graves' disease.

[1]  Yanfang Jiang,et al.  A high frequency of peripheral blood NKG2D+NK and NKT cells in euthyroid patients with new onset hashimoto’s thyroiditis—a pilot study , 2014, Immunological investigations.

[2]  C. Marcocci,et al.  Diagnosis and classification of Graves' disease. , 2014, Autoimmunity reviews.

[3]  E. Magen,et al.  Lymphocyte-Platelet Crosstalk in Graves’ Disease , 2014, The American journal of the medical sciences.

[4]  Y. Kuo,et al.  Hemoglobin, iron, and vitamin B12 deficiencies and high blood homocysteine levels in patients with anti-thyroid autoantibodies. , 2014, Journal of the Formosan Medical Association = Taiwan yi zhi.

[5]  P. Åsman,et al.  Increased TRAb and/or low anti-TPO titers at diagnosis of graves' disease are associated with an increased risk of developing ophthalmopathy after onset. , 2014, Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association.

[6]  Y. Tomer Mechanisms of autoimmune thyroid diseases: from genetics to epigenetics. , 2014, Annual review of pathology.

[7]  L. Moretta,et al.  KIR and KIR ligand polymorphism: a new area for clinical applications? , 2013, Tissue antigens.

[8]  P. M. van Hagen,et al.  The peripheral blood compartment in patients with Graves' disease: activated T lymphocytes and increased transitional and pre‐naive mature B lymphocytes , 2013, Clinical and experimental immunology.

[9]  Yanfang Jiang,et al.  A High Frequency of Circulating Th22 and Th17 Cells in Patients with New Onset Graves’ Disease , 2013, PloS one.

[10]  W. Yokoyama,et al.  Natural killer cells in human autoimmune disorders , 2013, Arthritis Research & Therapy.

[11]  M. Simmonds,et al.  GWAS in autoimmune thyroid disease: redefining our understanding of pathogenesis , 2013, Nature Reviews Endocrinology.

[12]  M. Altfeld,et al.  Control of human viral infections by natural killer cells. , 2013, Annual review of immunology.

[13]  Sumati Rajagopalan,et al.  Controlling natural killer cell responses: integration of signals for activation and inhibition. , 2013, Annual review of immunology.

[14]  R. Sun,et al.  Blocking the natural killer cell inhibitory receptor NKG2A increases activity of human natural killer cells and clears hepatitis B virus infection in mice. , 2013, Gastroenterology.

[15]  Yiming Li,et al.  Decrease in Proportion of CD19+CD24hiCD27+ B Cells and Impairment of Their Suppressive Function in Graves’ Disease , 2012, PloS one.

[16]  W. Held Nonclassical NK cell education , 2012, Nature Immunology.

[17]  Eric O Long,et al.  The adaptor protein Crk controls activation and inhibition of natural killer cells. , 2012, Immunity.

[18]  P. Conigliaro,et al.  Emerging role for NK cells in the pathogenesis of inflammatory arthropathies. , 2011, Autoimmunity reviews.

[19]  E. Alexander Autoimmunity: Thyroid autoantibodies and pregnancy risk , 2011, Nature Reviews Endocrinology.

[20]  J. Lünemann,et al.  Regulatory NK-Cell Functions in Inflammation and Autoimmunity , 2009, Molecular medicine.

[21]  Xue-guang Zhang,et al.  Association of Graves’ Disease and Prevalence of Circulating IFN-γ-producing CD28− T Cells , 2008, Journal of Clinical Immunology.

[22]  Eric Vivier,et al.  Functions of natural killer cells , 2008, Nature Immunology.

[23]  H. Young,et al.  In Vivo Regulation of Experimental Autoimmune Encephalomyelitis by NK Cells: Alteration of Primary Adaptive Responses12 , 2008, The Journal of Immunology.

[24]  J. Polańska,et al.  An analysis of the prevalence of thyroid autoantibodies: thyroid peroxidase antibodies (ATA) and thyroglobulin antibodies (ATG) in children with newly diagnosed diabetes mellitus type 1 during 2000–2004 in the Upper Silesia region, Poland , 2008, Acta Diabetologica.

[25]  Z. Tian,et al.  NK3-Like NK Cells Are Involved in Protective Effect of Polyinosinic-Polycytidylic Acid on Type 1 Diabetes in Nonobese Diabetic Mice1 , 2007, The Journal of Immunology.

[26]  S. Welte,et al.  Mutual activation of natural killer cells and monocytes mediated by NKp80-AICL interaction , 2006, Nature Immunology.

[27]  G. Burmester,et al.  Immunopathologic role of B lymphocytes in rheumatoid arthritis: rationale of B cell-directed therapy. , 2006, Autoimmunity reviews.

[28]  L. Hegedüs,et al.  The rationale for B lymphocyte depletion in Graves' disease. Monoclonal anti-CD20 antibody therapy as a novel treatment option. , 2006, European journal of endocrinology.

[29]  K. Tung,et al.  Requirements of NK Cells and Proinflammatory Cytokines in T Cell-Dependent Neonatal Autoimmune Ovarian Disease Triggered by Immune Complex1 , 2004, The Journal of Immunology.

[30]  Christophe Benoist,et al.  Natural killer cells distinguish innocuous and destructive forms of pancreatic islet autoimmunity. , 2004, Proceedings of the National Academy of Sciences of the United States of America.

[31]  D. Greiner,et al.  Deficiencies in Gut NK Cell Number and Function Precede Diabetes Onset in BB Rats1 , 2004, The Journal of Immunology.

[32]  R. Tan,et al.  Regulation of Autoimmune Diabetes by Complete Freund’s Adjuvant Is Mediated by NK Cells , 2004, The Journal of Immunology.

[33]  D. Raulet Roles of the NKG2D immunoreceptor and its ligands , 2003, Nature Reviews Immunology.

[34]  V. Triggiani,et al.  Modifications of the immune responsiveness in patients with autoimmune thyroiditis: evidence for a systemic immune alteration. , 2003, Current pharmaceutical design.

[35]  A. Bossowski,et al.  Analysis of Circulating T γ/δ Lymphocytes and CD16/56 Cell Populations in Children and Adolescents with Graves' Disease , 2003, Pediatric Research.

[36]  M. Caligiuri,et al.  The biology of human natural killer-cell subsets. , 2001, Trends in immunology.

[37]  F. Shi,et al.  Natural killer cells determine the outcome of B cell–mediated autoimmunity , 2000, Nature Immunology.

[38]  H. Schleusener,et al.  Natural killer cell activity in patients with Graves' disease and Hashimoto's thyroiditis. , 1998, Thyroid : official journal of the American Thyroid Association.

[39]  I. Gavilán,et al.  Serial analysis of the effects of methimazole or radical therapy on circulating CD16/56 subpopulations in Graves' disease. , 1998, European journal of endocrinology.

[40]  K. Takeda,et al.  The development of autoimmunity in C57BL/6 lpr mice correlates with the disappearance of natural killer type 1-positive cells: evidence for their suppressive action on bone marrow stem cell proliferation, B cell immunoglobulin secretion, and autoimmune symptoms , 1993, The Journal of experimental medicine.

[41]  F. Karlsson,et al.  Immunomodulation by methimazole therapy in Graves' disease: rapid changes in activation stage of circulating regulatory T cell subsets, B cells and NK cells. , 1988, Clinical and experimental immunology.

[42]  N. Ishikawa,et al.  Natural killer and natural killer-like cell activity of peripheral blood and intrathyroidal mononuclear cells from patients with Graves' disease. , 1988, The Journal of clinical endocrinology and metabolism.

[43]  S. Targan,et al.  NK and T cell subsets regulate antibody production by human in vivo antigen-induced lymphoblastoid B cells. , 1984, Journal of immunology.

[44]  E. Aktas,et al.  Relationship between CD107a expression and cytotoxic activity. , 2009, Cellular immunology.

[45]  Lewis L Lanier,et al.  NK cell recognition. , 2005, Annual review of immunology.