Identification of Networks of Co-Occurring, Tumor-Related DNA Copy Number Changes Using a Genome-Wide Scoring Approach
暂无分享,去创建一个
Marcel J. T. Reinders | Lodewyk F. A. Wessels | David J. Adams | Christiaan Klijn | Jan Bot | Jos Jonkers | M. Reinders | D. Adams | L. Wessels | J. Jonkers | J. Bot | C. Klijn
[1] Chris Sander,et al. An integrated genomic analysis of lung cancer reveals loss of DUSP4 in EGFR-mutant tumors , 2009, Oncogene.
[2] Thorsten Heinzel,et al. Ligand-independent repression by the thyroid hormone receptor mediated by a nuclear receptor co-repressor , 1995, Nature.
[3] P S Gaynon,et al. Prognostic significance of cytogenetic abnormalities of chromosome arm 12p in childhood acute lymphoblastic leukemia , 2000, Cancer.
[4] A. Berns,et al. Haplo-insufficiency? Let me count the ways. , 2001, Genes & development.
[5] M. Leid,et al. Identification of Nuclear Receptor Corepressor as a Peroxisome Proliferator-activated Receptor α Interacting Protein* , 1999, The Journal of Biological Chemistry.
[6] Marcel J. T. Reinders,et al. Co-occurrence analysis of insertional mutagenesis data reveals cooperating oncogenes , 2007, ISMB/ECCB.
[7] Amit Verma,et al. AML1-ETO Decreases ETO-2 (MTG16) Interactions with Nuclear Receptor Corepressor, an Effect That Impairs Granulocyte Differentiation , 2004, Cancer Research.
[8] D. Pinkel,et al. Array comparative genomic hybridization and its applications in cancer , 2005, Nature Genetics.
[9] Li Zhao,et al. Nuclear Receptor Corepressor Is a Novel Regulator of Phosphatidylinositol 3-Kinase Signaling , 2007, Molecular and Cellular Biology.
[10] Gouri Nanjangud,et al. Array comparative genomic hybridization reveals genomic copy number changes associated with outcome in diffuse large B-cell lymphomas. , 2006, Blood.
[11] M. Nowak,et al. Dynamics of cancer progression , 2004, Nature Reviews Cancer.
[12] Göran Roos,et al. Chromosomal Imbalances in Diffuse Large B-Cell Lymphoma Detected by Comparative Genomic Hybridization , 2002, Modern Pathology.
[13] M. Fiegl,et al. Presence of a p53 gene deletion in patients with multiple myeloma predicts for short survival after conventional-dose chemotherapy. , 1998, Blood.
[14] P S Gaynon,et al. CLINICAL OBSERVATIONS, INTERVENTIONS, AND THERAPEUTIC TRIALS Association of Chromosome Arm 9p Abnormalities With Adverse Risk in Childhood Acute Lymphoblastic Leukemia: A Report From the Children's Cancer Group , 2016 .
[15] O. Sieber,et al. Genomic instability — the engine of tumorigenesis? , 2003, Nature Reviews Cancer.
[16] A Benner,et al. p53 gene deletion predicts for poor survival and non-response to therapy with purine analogs in chronic B-cell leukemias. , 1995, Blood.
[17] L. Bullinger,et al. Genetics of chronic lymphocytic leukemia: genomic aberrations and VH gene mutation status in pathogenesis and clinical course , 2002, Leukemia.
[18] Lawrence S. Hon,et al. High-resolution analysis of copy number alterations and associated expression changes in ovarian tumors , 2009, BMC Medical Genomics.
[19] T. Hubbard,et al. A census of human cancer genes , 2004, Nature Reviews Cancer.
[20] Béatrice Desvergne,et al. Peroxisome-proliferator-activated receptors and cancers: complex stories , 2004, Nature Reviews Cancer.
[21] O. Rath,et al. MAP kinase signalling pathways in cancer , 2007, Oncogene.
[22] D. Hanahan,et al. The Hallmarks of Cancer , 2000, Cell.
[23] G. Ehninger,et al. The prognostic impact of 17p (p53) deletion in 2272 adults with acute myeloid leukemia , 2009, Leukemia.
[24] Takeshi Suzuki,et al. Tumor suppressor gene identification using retroviral insertional mutagenesis in Blm‐deficient mice , 2006, The EMBO journal.
[25] A. Jetten,et al. Characterization of the Repressor Function of the Nuclear Orphan Receptor Retinoid Receptor-related Testis-associated Receptor/Germ Cell Nuclear Factor* , 2000, The Journal of Biological Chemistry.
[26] Christian von Mering,et al. STRING 8—a global view on proteins and their functional interactions in 630 organisms , 2008, Nucleic Acids Res..
[27] Brian T Chait,et al. The N-CoR-HDAC3 nuclear receptor corepressor complex inhibits the JNK pathway through the integral subunit GPS2. , 2002, Molecular cell.
[28] T. Hubbard,et al. Large-Scale Mutagenesis in p19ARF- and p53-Deficient Mice Identifies Cancer Genes and Their Collaborative Networks , 2008, Cell.
[29] Jun Qin,et al. Purification and functional characterization of the human N‐CoR complex: the roles of HDAC3, TBL1 and TBLR1 , 2003, The EMBO journal.
[30] F. Ross,et al. Genome complexity in acute lymphoblastic leukemia is revealed by array-based comparative genomic hybridization , 2007, Oncogene.
[31] Martin A. Nowak,et al. The significance of unstable chromosomes in colorectal cancer , 2003, Nature Reviews Cancer.
[32] P Meltzer,et al. Transgenic Targeting of a Dominant Negative Corepressor to Liver Blocks Basal Repression by Thyroid Hormone Receptor and Increases Cell Proliferation* , 2001, The Journal of Biological Chemistry.
[33] F Menestrina,et al. Insights into the multistep transformation process of lymphomas: IgH-associated translocations and tumor suppressor gene mutations in clonally related composite Hodgkin's and non-Hodgkin's lymphomas , 2005, Leukemia.
[34] S. Gabriel,et al. High-throughput oncogene mutation profiling in human cancer , 2007, Nature Genetics.
[35] J. Stockman,et al. A Network Model of a Cooperative Genetic Landscape in Brain Tumors , 2011 .
[36] J. Downing,et al. Genome-wide profiling of genetic alterations in acute lymphoblastic leukemia: recent insights and future directions , 2009, Leukemia.
[37] Kristen Jepsen,et al. Combinatorial Roles of the Nuclear Receptor Corepressor in Transcription and Development , 2000, Cell.