Postprandial hypertriglyceridemia impairs endothelial function by enhanced oxidant stress.

[1]  H. Hein,et al.  Triglyceride concentration and ischemic heart disease: an eight-year follow-up in the Copenhagen Male Study. , 1998, Circulation.

[2]  A. Gotto,et al.  Triglyceride: the forgotten risk factor. , 1998, Circulation.

[3]  J. Hokanson,et al.  Hypertriglyceridemia as a cardiovascular risk factor. , 1998, The American journal of cardiology.

[4]  K. Kostner,et al.  Is oxidative stress causally linked to unstable angina pectoris? A study in 100 CAD patients and matched controls. , 1997, Cardiovascular research.

[5]  G. Plotnick,et al.  Effect of antioxidant vitamins on the transient impairment of endothelium-dependent brachial artery vasoactivity following a single high-fat meal. , 1997, JAMA.

[6]  K. Schenck-Gustafsson,et al.  Transient triglyceridemia decreases vascular reactivity in young, healthy men without risk factors for coronary heart disease. , 1997, Circulation.

[7]  D. Sgoutas,et al.  Vitamins C and E inhibit O2- production in the pig coronary artery. , 1997, Circulation.

[8]  M. Reilly,et al.  Modulation of oxidant stress in vivo in chronic cigarette smokers. , 1996, Circulation.

[9]  M. Hori,et al.  Endothelial dysfunction in the early stage of atherosclerosis precedes appearance of intimal lesions assessable with intravascular ultrasound. , 1996, American heart journal.

[10]  D. Harrison,et al.  Dietary correction of hypercholesterolemia in the rabbit normalizes endothelial superoxide anion production. , 1995, Circulation.

[11]  J. Keaney,et al.  Dietary probucol preserves endothelial function in cholesterol-fed rabbits by limiting vascular oxidative stress and superoxide generation. , 1995, The Journal of clinical investigation.

[12]  J. Hodgson,et al.  Evidence that selective endothelial dysfunction may occur in the absence of angiographic or ultrasound atherosclerosis in patients with risk factors for atherosclerosis. , 1994, Journal of the American College of Cardiology.

[13]  D. Celermajer,et al.  Cigarette Smoking Is Associated With Dose‐Related and Potentially Reversible Impairment of Endothelium‐Dependent Dilation in Healthy Young Adults , 1993, Circulation.

[14]  A. Nitenberg,et al.  Impairment of Coronary Vascular Reserve and ACh-Induced Coronary Vasodilation in Diabetic Patients With Angiographically Normal Coronary Arteries and Normal Left Ventricular Systolic Function , 1993, Diabetes.

[15]  D. Harrison,et al.  Hypercholesterolemia increases endothelial superoxide anion production. , 1993, The Journal of clinical investigation.

[16]  J. Manson,et al.  Vitamin E consumption and the risk of coronary disease in women. , 1993, The New England journal of medicine.

[17]  E. Rimm,et al.  Vitamin E consumption and the risk of coronary heart disease in men. , 1993, The New England journal of medicine.

[18]  K. Prasad,et al.  Oxygen free radicals and hypercholesterolemic atherosclerosis: effect of vitamin E. , 1993, American heart journal.

[19]  A. Quyyumi,et al.  Endothelial Dysfunction in Patients With Chest Pain and Normal Coronary Arteries , 1992, Circulation.

[20]  J. K. Lloyd,et al.  Non-invasive detection of endothelial dysfunction in children and adults at risk of atherosclerosis , 1992, The Lancet.

[21]  S. Solomon,et al.  Impaired endothelium-mediated vasodilation in the peripheral vasculature of patients with congestive heart failure. , 1992, Journal of the American College of Cardiology.

[22]  B. Hoffman,et al.  Inactivation of endothelial derived relaxing factor by oxidized lipoproteins. , 1992, The Journal of clinical investigation.

[23]  E. Coligan.  Current protocols in immunology , 1991 .

[24]  C. Packard,et al.  Postprandial lipemia, fenofibrate and coronary artery disease. , 1990, Atherosclerosis.

[25]  B. Pannier,et al.  Flow-dependent vasodilation of brachial artery in essential hypertension. , 1990, The American journal of physiology.

[26]  H. Drexler,et al.  Flow-dependent coronary artery dilatation in humans. , 1989, Circulation.

[27]  S. Arimori,et al.  Increased Superoxide Production by Mononuclear Cells of Patients With Hypertriglyceridemia and Diabetes , 1988, Diabetes.

[28]  D. Steinberg,et al.  Macrophage Oxidation of Low Density Lipoprotein Generates a Modified Form Recognized by the Scavenger Receptor , 1986, Arteriosclerosis.

[29]  A. Yen,et al.  Increased c-myc RNA levels associated with the precommitment state during HL-60 myeloid differentiation. , 1986, Cancer research.

[30]  D. Morel,et al.  Low density lipoprotein cytotoxicity induced by free radical peroxidation of lipid. , 1983, Journal of lipid research.

[31]  D. B. Zilversmit.  Atherogenesis: a postprandial phenomenon. , 1979, Circulation.

[32]  P. McCay,et al.  Evidence that peroxidation of lysosomal membranes is initiated by hydroxyl free radicals produced during flavin enzyme activity. , 1973, The Journal of biological chemistry.

[33]  R. Levy,et al.  Estimation of the concentration of low-density lipoprotein cholesterol in plasma, without use of the preparative ultracentrifuge. , 1972, Clinical chemistry.

[34]  M. Albrink,et al.  Serum triglycerides in coronary artery disease. , 1958, Transactions of the Association of American Physicians.