Uptake by mouse peritoneal macrophages of large cholesteryl ester-rich particles isolated from human atherosclerotic lesions.
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[1] H F Hoff,et al. A low density lipoprotein-sized particle isolated from human atherosclerotic lesions is internalized by macrophages via a non-scavenger-receptor mechanism. , 1986, Journal of lipid research.
[2] Y Morino,et al. Scavenger receptor for aldehyde-modified proteins. , 1986, The Journal of biological chemistry.
[3] P. Edwards,et al. Receptor-mediated uptake of remnant lipoproteins by cholesterol-loaded human monocyte-macrophages. , 1985, The Journal of biological chemistry.
[4] C. Minick,et al. Insoluble low-density lipoprotein-proteoglycan complexes enhance cholesteryl ester accumulation in macrophages. , 1985, The American journal of pathology.
[5] H. Shio,et al. Lipoprotein-heparin-fibronectin-denatured collagen complexes enhance cholesteryl ester accumulation in macrophages , 1984, The Journal of cell biology.
[6] B. Clevidence,et al. Cholesterol esterification in macrophages. Stimulation by lipoproteins containing apo B isolated from human aortas. , 1984, Arteriosclerosis.
[7] R. Mahley,et al. The receptor-binding domain of human apolipoprotein E. Binding of apolipoprotein E fragments. , 1983, The Journal of biological chemistry.
[8] R. Gerrity,et al. Quantitation of apolipoprotein B in aortas of hypercholesterolemic swine. , 1983, Laboratory investigation; a journal of technical methods and pathology.
[9] M. Brown,et al. Lipoprotein metabolism in the macrophage: implications for cholesterol deposition in atherosclerosis. , 1983, Annual review of biochemistry.
[10] J. Gaubatz,et al. Isolation, purification, and characterization of a lipoprotein containing Apo B from the human aorta. , 1982, Atherosclerosis.
[11] R. Hawkes,et al. A dot-immunobinding assay for monoclonal and other antibodies. , 1982, Analytical biochemistry.
[12] D. Steinberg,et al. Enhanced macrophage degradation of low density lipoprotein previously incubated with cultured endothelial cells: recognition by receptors for acetylated low density lipoproteins. , 1981, Proceedings of the National Academy of Sciences of the United States of America.
[13] R. Gerrity,et al. The role of the monocyte in atherogenesis: II. Migration of foam cells from atherosclerotic lesions. , 1981, The American journal of pathology.
[14] M. Brown,et al. Stimulation of Cholesteryl Ester Synthesis in Macrophages by Extracts of Atherosclerotic Human Aortas and Complexes of Albumin/Cholesteryl Esters , 1981, Arteriosclerosis.
[15] L. Lorand,et al. Fibronectin-mediated uptake of gelatin-coated latex particles by peritoneal macrophages , 1980, The Journal of cell biology.
[16] F. Grinnell. Fibroblast receptor for cell-substratum adhesion: studies on the interaction of baby hamster kidney cells with latex beads coated by cold insoluble globulin (plasma fibronectin) , 1980, The Journal of cell biology.
[17] P. Edwards,et al. Malondialdehyde alteration of low density lipoproteins leads to cholesteryl ester accumulation in human monocyte-macrophages. , 1980, Proceedings of the National Academy of Sciences of the United States of America.
[18] H. Shio,et al. Characterization of lipid-laden aortic cells from cholesterol-fed rabbits. IV. Investigation of macrophage-like properties of aortic cell populations. , 1979, Laboratory investigation; a journal of technical methods and pathology.
[19] R. Mahley,et al. Altered metabolism (in vivo and in vitro) of plasma lipoproteins after selective chemical modification of lysine residues of the apoproteins. , 1979, The Journal of clinical investigation.
[20] H. Towbin,et al. Electrophoretic transfer of proteins from polyacrylamide gels to nitrocellulose sheets: procedure and some applications. , 1979, Proceedings of the National Academy of Sciences of the United States of America.
[21] W. A. Bradley,et al. Characterization of low density lipoprotein-like particle in the human aorta from grossly normal and atherosclerotic regions. , 1979, Biochimica et biophysica acta.
[22] W. Hollander,et al. Lipoproteins in human atherosclerotic vessels. I. Biochemical properties of arterial low density lipoproteins, very low density lipoproteins, and high density lipoproteins. , 1979, Experimental and molecular pathology.
[23] M. Brown,et al. Binding site on macrophages that mediates uptake and degradation of acetylated low density lipoprotein, producing massive cholesterol deposition. , 1979, Proceedings of the National Academy of Sciences of the United States of America.
[24] J. Avigan,et al. Procedure for determination of free and total cholesterol in micro- or nanogram amounts suitable for studies with cultured cells. , 1978, Journal of lipid research.
[25] G. Peterson,et al. A simplification of the protein assay method of Lowry et al. which is more generally applicable. , 1977, Analytical biochemistry.
[26] M. A. Hayat,et al. Principles and Techniques of Electron Microscopy , 1975 .
[27] H. Hoff. Differential interference contrast microscopy of human atherosclerotic lesions. , 1973, The American journal of pathology.
[28] D. Bilheimer,et al. The metabolism of very low density lipoprotein proteins. I. Preliminary in vitro and in vivo observations. , 1972, Biochimica et biophysica acta.
[29] Z. Werb,et al. CHOLESTEROL METABOLISM IN THE MACROPHAGE III. INGESTION AND INTRACELLULAR FATE OF CHOLESTEROL AND CHOLESTEROL ESTERS , 1972 .
[30] Mcgill Hc. Fatty streaks in the coronary arteries and aorta. , 1968 .
[31] F. Hatch. Practical methods for plasma lipoprotein analysis. , 1968, Advances in lipid research.
[32] A. Mcfarlane,et al. Efficient Trace-labelling of Proteins with Iodine , 1958, Nature.
[33] O. H. Lowry,et al. Protein measurement with the Folin phenol reagent. , 1951, The Journal of biological chemistry.