of Medicine

Mr NORMAN BARNETT said that Mr Graham Hodgson had reported that there was no definite conclusion in his mind about his (Mr Barnett's) case ; he (Mr Hodgson) had never seen a similar one, and was uncertain whether it was a normal ear with unusual bone development, or whether there was a new growth of bone. He could detect no radiological evidence of the porosis of the cochlea which he had always seen in cases of otosclerosis. At present, however, negative X-ray evidence was not sufficient to rule out the possibility of otosclerosis. Mr T. B. LAYTON asked what the President proposed to do with the papilloma, if anything. Would he remove bone ? Sir JAMES DUNDAS-GRANT asked what was the result of probing in epithelioma simulating papilloma. He had himself found an enormous amount of denudation of bone beyond it. Mr SYDNEY SCOTT asked—In the second case, what was the patient's age ? In case the papilloma proved to be malignant, he suggested applying surgical diathermy. He preferred to have a pathologist present at the time of operation, to cut a frozen section. If it were malignant, he would at once perform a more extensive operation than one originally planned. The PRESIDENT (in reply) said that he proposed to carry out a modified mastoid operation, removing the posterior wall of the meatus, cartilage included, after removing some bone. After microscopical examination of a small portion, the growth was reported to be a papilloma.

[1]  R. Watson,et al.  A Specific Dileucine Motif Is Required for the GGA-dependent Entry of Newly Synthesized Insulin-responsive Aminopeptidase into the Insulin-responsive Compartment* , 2006, Journal of Biological Chemistry.

[2]  Ping Huang,et al.  Asymmetric phospholipid distribution drives in vitro reconstituted SNARE-dependent membrane fusion , 2006, Proceedings of the National Academy of Sciences.

[3]  J. Pessin,et al.  Dual Regulation of Rho and Rac by p120 Catenin Controls Adipocyte Plasma Membrane Trafficking* , 2006, Journal of Biological Chemistry.

[4]  P. Halban,et al.  Regulation of pancreatic β-cell insulin secretion by actin cytoskeleton remodelling: role of gelsolin and cooperation with the MAPK signalling pathway , 2006, Journal of Cell Science.

[5]  G. Valenti,et al.  Adipocytes support cAMP-dependent translocation of aquaporin-2 from intracellular sites distinct from the insulin-responsive GLUT4 storage compartment. , 2006, American journal of physiology. Renal physiology.

[6]  D. Boyle,et al.  Intracellular Trafficking and Secretion of Adiponectin Is Dependent on GGA-coated Vesicles* , 2006, Journal of Biological Chemistry.

[7]  R. Watson,et al.  Initial entry of IRAP into the insulin-responsive storage compartment occurs prior to basal or insulin-stimulated plasma membrane recycling. , 2005, American journal of physiology. Endocrinology and metabolism.

[8]  J. Pessin,et al.  Munc18c heterozygous knockout mice display increased susceptibility for severe glucose intolerance. , 2005, Diabetes.

[9]  S. Whiteheart,et al.  Platelets from Munc18c heterozygous mice exhibit normal stimulus-induced release , 2004, Thrombosis and Haemostasis.

[10]  M. Kanzaki,et al.  Phosphatidylinositol 4,5-Bisphosphate Regulates Adipocyte Actin Dynamics and GLUT4 Vesicle Recycling* , 2004, Journal of Biological Chemistry.

[11]  M. Kanzaki,et al.  Entry of newly synthesized GLUT4 into the insulin‐responsive storage compartment is GGA dependent , 2004, The EMBO journal.

[12]  J. Pessin,et al.  Functional characterization of an insulin-responsive glucose transporter (GLUT4) from fish adipose tissue. , 2004, American journal of physiology. Endocrinology and metabolism.

[13]  M. Kanzaki,et al.  Atypical protein kinase C (PKC / ) is a convergent downstream target of the insulin-stimulated phosphatidylinositol 3-kinase and TC10 signaling pathways , 2004 .

[14]  J. Pessin,et al.  Lipid Raft targeting of the TC10 amino terminal domain is responsible for disruption of adipocyte cortical actin. , 2003, Molecular biology of the cell.

[15]  S. Crosson,et al.  PTG gene deletion causes impaired glycogen synthesis and developmental insulin resistance. , 2003, The Journal of clinical investigation.

[16]  P. Halban,et al.  Glucose-stimulated insulin secretion is coupled to the interaction of actin with the t-SNARE (target membrane soluble N-ethylmaleimide-sensitive factor attachment protein receptor protein) complex. , 2003, Molecular endocrinology.

[17]  R. Watson,et al.  The Adipocyte Plasma Membrane Caveolin Functional/Structural Organization Is Necessary for the Efficient Endocytosis of GLUT4* , 2003, The Journal of Biological Chemistry.

[18]  M. Kanzaki,et al.  The Exocytotic Trafficking of TC10 Occurs through both Classical and Nonclassical Secretory Transport Pathways in 3T3L1 Adipocytes , 2003, Molecular and Cellular Biology.

[19]  R. Watson,et al.  The Requirement of Specific Membrane Domains for Raf-1 Phosphorylation and Activation* , 2003, The Journal of Biological Chemistry.

[20]  M. Kanzaki,et al.  A Crk-II/TC10 Signaling Pathway Is Required For Osmotic Shock-stimulated Glucose Transport* , 2002, The Journal of Biological Chemistry.

[21]  A. Saltiel,et al.  The Insulin Receptor Catalyzes the Tyrosine Phosphorylation of Caveolin-1* , 2002, The Journal of Biological Chemistry.

[22]  A. Jeromin,et al.  NCS-1 Inhibits Insulin-stimulated GLUT4 Translocation in 3T3L1 Adipocytes through a Phosphatidylinositol 4-Kinase-dependent Pathway* , 2002, The Journal of Biological Chemistry.

[23]  M. Kanzaki,et al.  Caveolin-associated Filamentous Actin (Cav-actin) Defines a Novel F-actin Structure in Adipocytes* , 2002, The Journal of Biological Chemistry.

[24]  J. Pessin,et al.  Rate and extent of phagocytosis in macrophages lacking vamp3 , 2002, Journal of leukocyte biology.

[25]  M. Kanzaki,et al.  Small GTP-binding protein TC10 differentially regulates two distinct populations of filamentous actin in 3T3L1 adipocytes. , 2002, Molecular biology of the cell.

[26]  M. Kanzaki,et al.  Intracellular insulin-responsive glucose transporter (GLUT4) distribution but not insulin-stimulated GLUT4 exocytosis and recycling are microtubule dependent. , 2002, Molecular endocrinology.

[27]  M. Kanzaki,et al.  Functional Dissection of Lipid and Protein Kinase Signals of PIKfyve Reveals the Role of PtdIns 3,5-P2 Production for Endomembrane Integrity* , 2002, The Journal of Biological Chemistry.

[28]  M. Kanzaki,et al.  Insulin Stimulates Actin Comet Tails on Intracellular GLUT4-containing Compartments in Differentiated 3T3L1 Adipocytes* 210 , 2001, The Journal of Biological Chemistry.

[29]  M. Kanzaki,et al.  Insulin-stimulated GLUT4 Translocation in Adipocytes Is Dependent upon Cortical Actin Remodeling* 210 , 2001, The Journal of Biological Chemistry.

[30]  M. Kanzaki,et al.  Lipid raft microdomain compartmentalization of TC10 is required for insulin signaling and GLUT4 translocation , 2001, The Journal of cell biology.

[31]  R. Watson,et al.  Transmembrane domain length determines intracellular membrane compartment localization of syntaxins 3, 4, and 5. , 2001, American journal of physiology. Cell physiology.

[32]  G. Shulman,et al.  Syntaxin 4 heterozygous knockout mice develop muscle insulin resistance. , 2001, The Journal of clinical investigation.

[33]  J. Pessin,et al.  Differentiated 3T3L1 Adipocytes Are Composed of Heterogenous Cell Populations with Distinct Receptor Tyrosine Kinase Signaling Properties* , 2001, The Journal of Biological Chemistry.

[34]  J. Pessin,et al.  VAMP3 Null Mice Display Normal Constitutive, Insulin- and Exercise-Regulated Vesicle Trafficking , 2001, Molecular and Cellular Biology.

[35]  J. Pessin,et al.  Munc18c Regulates Insulin-stimulated GLUT4 Translocation to the Transverse Tubules in Skeletal Muscle* , 2001, The Journal of Biological Chemistry.

[36]  J. Pessin,et al.  Discrimination of GLUT4 vesicle trafficking from fusion using a temperature‐sensitive Munc18c mutant , 2000, The EMBO journal.

[37]  J. Pessin,et al.  The MEF2A Isoform Is Required for Striated Muscle-specific Expression of the Insulin-responsive GLUT4 Glucose Transporter* , 2000, The Journal of Biological Chemistry.

[38]  J. Pessin,et al.  Functional comparison of the role of dynamin 2 splice variants on GLUT-4 endocytosis in 3T3L1 adipocytes. , 2000, American journal of physiology. Endocrinology and metabolism.

[39]  R. Watson,et al.  Calmodulin antagonists inhibit insulin-stimulated GLUT4 (glucose transporter 4) translocation by preventing the formation of phosphatidylinositol 3,4,5-trisphosphate in 3T3L1 adipocytes. , 2000, Molecular endocrinology.

[40]  R. Watson,et al.  Functional Cooperation of Two Independent Targeting Domains in Syntaxin 6 Is Required for Its Efficient Localization in thetrans-Golgi Network of 3T3L1 Adipocytes* , 2000, The Journal of Biological Chemistry.

[41]  M. Kanzaki,et al.  Munc18c Function Is Required for Insulin-Stimulated Plasma Membrane Fusion of GLUT4 and Insulin-Responsive Amino Peptidase Storage Vesicles , 2000, Molecular and Cellular Biology.

[42]  J. Pessin,et al.  Temporal Separation of Insulin-stimulated GLUT4/IRAP Vesicle Plasma Membrane Docking and Fusion in 3T3L1 Adipocytes* , 1999, The Journal of Biological Chemistry.

[43]  J. Pessin,et al.  Dynamin Is Required for Recombinant Adeno-Associated Virus Type 2 Infection , 1999, Journal of Virology.

[44]  R. Fucini,et al.  Osmotic Shock Inhibits Insulin Signaling by Maintaining Akt/Protein Kinase B in an Inactive Dephosphorylated State , 1999, Molecular and Cellular Biology.

[45]  R. Fucini,et al.  Insulin-induced Desensitization of Extracellular Signal-regulated Kinase Activation Results from an Inhibition of Raf Activity Independent of Ras Activation and Dissociation of the Grb2-SOS Complex* , 1999, The Journal of Biological Chemistry.

[46]  A. Saltiel,et al.  Aldolase Mediates the Association of F-actin with the Insulin-responsive Glucose Transporter GLUT4* , 1999, The Journal of Biological Chemistry.

[47]  M. Kanzaki,et al.  Synip: a novel insulin-regulated syntaxin 4-binding protein mediating GLUT4 translocation in adipocytes. , 1999, Molecular cell.

[48]  J. Pessin,et al.  Regulation of Insulin-stimulated GLUT4 Translocation by Munc18c in 3T3L1 Adipocytes* , 1998, The Journal of Biological Chemistry.

[49]  J. Pessin,et al.  Adenovirus-mediated transfer of a modified human proinsulin gene reverses hyperglycemia in diabetic mice. , 1998, American journal of physiology. Endocrinology and metabolism.

[50]  J. Pessin,et al.  Expression of a Dominant Interfering Dynamin Mutant in 3T3L1 Adipocytes Inhibits GLUT4 Endocytosis without Affecting Insulin Signaling* , 1998, The Journal of Biological Chemistry.

[51]  J. Pessin,et al.  Inhibition of Clathrin-Mediated Endocytosis Selectively Attenuates Specific Insulin Receptor Signal Transduction Pathways , 1998, Molecular and Cellular Biology.

[52]  S. Guruswamy,et al.  Myocyte enhancer factor 2 (MEF2)-binding site is required for GLUT4 gene expression in transgenic mice. Regulation of MEF2 DNA binding activity in insulin-deficient diabetes. , 1998, The Journal of biological chemistry.

[53]  D. Chen,et al.  Guanosine 5′-O-(3-Thiotriphosphate) (GTPγS) Stimulation of GLUT4 Translocation is Tyrosine Kinase-dependent* , 1998, The Journal of Biological Chemistry.

[54]  T. Pawson,et al.  Insulin regulates the dynamic balance between Ras and Rap1 signaling by coordinating the assembly states of the Grb2–SOS and CrkII–C3G complexes , 1998, The EMBO journal.

[55]  J. Pessin,et al.  Regulation of glucose transporters GLUT-4 and GLUT-1 gene transcription in denervated skeletal muscle. , 1998, Journal of applied physiology.

[56]  J. Pessin,et al.  Insulin and Epidermal Growth Factor Stimulate a Conformational Change in Rap1 and Dissociation of the CrkII-C3G Complex* , 1997, The Journal of Biological Chemistry.

[57]  B. Margolis,et al.  The 66-kDa Shc Isoform Is a Negative Regulator of the Epidermal Growth Factor-stimulated Mitogen-activated Protein Kinase Pathway* , 1997, The Journal of Biological Chemistry.

[58]  D. Chen,et al.  Osmotic Shock Stimulates GLUT4 Translocation in 3T3L1 Adipocytes by a Novel Tyrosine Kinase Pathway* , 1997, The Journal of Biological Chemistry.

[59]  K. Guan,et al.  Signaling molecules involved in coupling growth hormone receptor to mitogen-activated protein kinase activation. , 1997, Endocrinology.

[60]  J. Pessin,et al.  Insulin stimulates the phosphorylation of the 66- and 52-kilodalton Shc isoforms by distinct pathways. , 1997, Endocrinology.

[61]  C. Torrance,et al.  Characterization of a low affinity thyroid hormone receptor binding site within the rat GLUT4 gene promoter. , 1997, Endocrinology.

[62]  J. Pessin,et al.  Insulin and Epidermal Growth Factor Receptors Regulate Distinct Pools of Grb2-SOS in the Control of Ras Activation* , 1996, The Journal of Biological Chemistry.

[63]  J. Pessin,et al.  Insulin Stimulates the Serine Phosphorylation of the Signal Transducer and Activator of Transcription (STAT3) Isoform (*) , 1996, The Journal of Biological Chemistry.

[64]  A. Saltiel,et al.  Epidermal Growth Factor Receptor Targeting Prevents Uncoupling of the Grb2-SOS Complex (*) , 1996, The Journal of Biological Chemistry.

[65]  J. Pessin,et al.  SOS Phosphorylation and Disassociation of the Grb2-SOS Complex by the ERK and JNK Signaling Pathways (*) , 1996, The Journal of Biological Chemistry.

[66]  J. Pessin,et al.  Insulin stimulation of a MEK-dependent but ERK-independent SOS protein kinase , 1996, Molecular and cellular biology.

[67]  J. Pessin,et al.  Transcriptional Regulation of the Human GLUT4 Gene Promoter in Diabetic Transgenic Mice (*) , 1995, The Journal of Biological Chemistry.

[68]  K. Guan,et al.  Desensitization of Ras Activation by a Feedback Disassociation of the SOS-Grb2 Complex (*) , 1995, The Journal of Biological Chemistry.

[69]  P. Cuatrecasas,et al.  Mitogen-activated Protein Kinase Kinase Inhibition Does Not Block the Stimulation of Glucose Utilization by Insulin (*) , 1995, The Journal of Biological Chemistry.

[70]  J. Pessin,et al.  Shc Isoform-specific Tyrosine Phosphorylation by the Insulin and Epidermal Growth Factor Receptors (*) , 1995, The Journal of Biological Chemistry.

[71]  A. Saltiel,et al.  Identification of the Major SHPTP2-binding Protein That Is Tyrosine-phosphorylated in Response to Insulin (*) , 1995, The Journal of Biological Chemistry.

[72]  J. Pessin,et al.  Epidermal Growth Factor-induced Association of the SHPTP2 Protein Tyrosine Phosphatase with a 115-kDa Phosphotyrosine Protein (*) , 1995, The Journal of Biological Chemistry.

[73]  J. Pessin,et al.  Insulin-stimulated disassociation of the SOS-Grb2 complex , 1995, Molecular and cellular biology.

[74]  J. Pessin,et al.  Divergent Insulin and Platelet-derived Growth Factor Regulation of Focal Adhesion Kinase (pp125FAK) Tyrosine Phosphorylation, and Rearrangement of Actin Stress Fibers (*) , 1995, The Journal of Biological Chemistry.

[75]  J. Pessin,et al.  Glycemic improvement in diabetic db/db mice by overexpression of the human insulin-regulatable glucose transporter (GLUT4). , 1995, The Journal of clinical investigation.

[76]  A. Saltiel,et al.  Protein-tyrosine-phosphatase SHPTP2 is a required positive effector for insulin downstream signaling. , 1995, Proceedings of the National Academy of Sciences of the United States of America.

[77]  J. Pessin,et al.  Skeletal muscle glucose transport and metabolism are enhanced in transgenic mice overexpressing the Glut4 glucose transporter. , 1995, The Journal of biological chemistry.

[78]  J. Pessin,et al.  Insulin receptor substrate-1 (IRS1) and Shc compete for a limited pool of Grb2 in mediating insulin downstream signaling. , 1994, The Journal of biological chemistry.

[79]  R. McPherson,et al.  Enhanced peripheral glucose utilization in transgenic mice expressing the human GLUT4 gene. , 1994, The Journal of biological chemistry.

[80]  J. Pessin,et al.  Myocyte enhancer factor 2 (MEF2) binding site is essential for C2C12 myotube-specific expression of the rat GLUT4/muscle-adipose facilitative glucose transporter gene. , 1994, The Journal of biological chemistry.

[81]  J. Pessin,et al.  Enhancement or inhibition of insulin signaling by insulin receptor substrate 1 is cell context dependent , 1994, Molecular and cellular biology.

[82]  L. Olson,et al.  Phosphatidylinositol 3-kinase activation is mediated by high-affinity interactions between distinct domains within the p110 and p85 subunits , 1994, Molecular and cellular biology.

[83]  R. McPherson,et al.  Transgenic mice expressing the human GLUT4/muscle-fat facilitative glucose transporter protein exhibit efficient glycemic control. , 1993, Proceedings of the National Academy of Sciences of the United States of America.

[84]  J. Pessin,et al.  Functional expression of insulin receptor substrate-1 is required for insulin-stimulated mitogenic signaling. , 1993, The Journal of biological chemistry.

[85]  J. Pessin,et al.  Identification of a skeletal muscle-specific regulatory domain in the rat GLUT4/muscle-fat gene. , 1993, The Journal of biological chemistry.

[86]  J. Pessin,et al.  Phosphatidylinositol 3-kinase functions upstream of Ras and Raf in mediating insulin stimulation of c-fos transcription. , 1993, The Journal of biological chemistry.

[87]  J. Pessin,et al.  Regulation of the GLUT4/muscle-fat glucose transporter mRNA in adipose tissue of insulin-deficient diabetic rats. , 1993, The Journal of biological chemistry.

[88]  J. Pessin,et al.  Time-dependent regulation of rat adipose tissue glucose transporter (GLUT4) mRNA and protein by insulin in streptozocin-diabetic and normal rats. , 1992, Metabolism: clinical and experimental.

[89]  S. Seino,et al.  Human GLUT4/Muscle-Fat Glucose-Transporter Gene: Characterization and Genetic Variation , 1992, Diabetes.

[90]  A. Marette,et al.  Abundance, localization, and insulin-induced translocation of glucose transporters in red and white muscle. , 1992, The American journal of physiology.

[91]  G. Bell,et al.  Expression and regulation of the human GLUT4/muscle-fat facilitative glucose transporter gene in transgenic mice. , 1992, The Journal of biological chemistry.

[92]  J. Treadway,et al.  Differential regulation of glucose transporter activity and expression in red and white skeletal muscle. , 1991, The Journal of biological chemistry.

[93]  J. Treadway,et al.  The endogenous functional turkey erythrocyte and rat liver insulin receptor is an α2β2 heterotetrameric complex , 1990 .

[94]  A. Klip,et al.  Glucose transport activity in L6 muscle cells is regulated by the coordinate control of subcellular glucose transporter distribution, biosynthesis, and mRNA transcription. , 1990, The Journal of biological chemistry.

[95]  J. Treadway,et al.  Assembly of insulin/insulin-like growth factor-1 hybrid receptors in vitro. , 1989, The Journal of biological chemistry.

[96]  J. Treadway,et al.  Regulation of the insulin receptor kinase by hyperinsulinism. , 1989, The Journal of biological chemistry.

[97]  J. Pessin,et al.  Polylysine specifically activates the insulin-dependent insulin receptor protein kinase. , 1989, The Journal of biological chemistry.

[98]  M. Swanson,et al.  High affinity insulin binding in the human placenta insulin receptor requires αβ heterodimeric subunit interactions , 1989, The Journal of Membrane Biology.

[99]  A. Klip,et al.  Insulin and glucose-dependent regulation of the glucose transport system in the rat L6 skeletal muscle cell line. , 1989, The Journal of biological chemistry.

[100]  J. Pessin,et al.  Regulation of the glucose transporter in developing rat brain. , 1989, Endocrinology.

[101]  J. Pessin,et al.  Glucose-dependent regulation of glucose transport activity, protein, and mRNA in primary cultures of rat brain glial cells. , 1988, The Journal of biological chemistry.

[102]  M. Jennings,et al.  Heterogeneity in the human erythrocyte band 3 anion-transporter revealed by Triton X-114 phase partitioning. , 1988, The Biochemical journal.

[103]  J. Pessin,et al.  Transfer of functional insulin receptors to receptor-deficient target cells. , 1988, Endocrinology.

[104]  J. Pessin,et al.  Differential sensitivity of the insulin-receptor kinase to thiol and oxidizing agents in the absence and presence of insulin. , 1987, The Biochemical journal.

[105]  J. Pessin,et al.  Phospholipid activation of the insulin receptor kinase: regulation by phosphatidylinositol , 1987, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[106]  J. Pessin,et al.  Insulin stimulation of the insulin receptor kinase can occur in the complete absence of beta subunit autophosphorylation. , 1987, The Journal of biological chemistry.

[107]  M. Czech,et al.  Regulation of insulin receptor kinase by multisite phosphorylation. , 1985, Biochimie.

[108]  J. Pessin,et al.  Subunit structure of the purified human placental insulin receptor. Intramolecular subunit dissociation by sodium dodecyl sulfate-polyacrylamide gel electrophoresis. , 1985, The Journal of biological chemistry.

[109]  J. Pessin,et al.  Phorbol ester induces desensitization of adenylate cyclase and phosphorylation of the beta-adrenergic receptor in turkey erythrocytes. , 1984, Proceedings of the National Academy of Sciences of the United States of America.

[110]  K. Isselbacher,et al.  Photoaffinity labeling of the stereospecific D-glucose transport system with cytochalasin B. , 1984, Federation proceedings.

[111]  M. Czech,et al.  The relationship of microvesicles to the plasmalemma of rat adipocytes. , 1983, European journal of cell biology.

[112]  M. Czech,et al.  beta-Adrenergic regulation of insulin and epidermal growth factor receptors in rat adipocytes. , 1983, The Journal of biological chemistry.

[113]  J. Massagué,et al.  Insulin action rapidly modulates the apparent affinity of the insulin-like growth factor II receptor. , 1983, The Journal of biological chemistry.

[114]  J. Pessin,et al.  Proximate charge effects. 2. Enthalpies of solvent transfer in the choline-anhydrocholine equilibrium , 1982 .

[115]  M. Czech,et al.  Photoaffinity labeling of the human erythrocyte D-glucose transporter. , 1982, The Journal of biological chemistry.

[116]  K. Isselbacher,et al.  Identification of the stereospecific hexose transporter from starved and fed chicken embryo fibroblasts. , 1982, Proceedings of the National Academy of Sciences of the United States of America.

[117]  J. Pessin,et al.  Budding of Rous sarcoma virus and vesicular stomatitis virus from localized lipid regions in the plasma membrane of chicken embryo fibroblasts. , 1980, The Journal of biological chemistry.

[118]  J. Pessin,et al.  Use of a fluorescent probe to compare the plasma membrane properties in normal and transformed cells. Evaluation of the interference by triacylglycerols and alkyldiacylglycerols. , 1978, Biochemistry.

[119]  M. Weber,et al.  Modification of the lipid composition of normal and Rous sarcoma virus-infected cells. Effects on transformation-associated membrane properties. , 1977, The Journal of biological chemistry.

[120]  M. Kanzaki,et al.  The trimeric GTP-binding protein (G(q)/G(11)) alpha subunit is required for insulin-stimulated GLUT4 translocation in 3T3L1 adipocytes. , 2000, The Journal of biological chemistry.

[121]  J. Pessin,et al.  Regulation of c-fos expression in adipose and muscle tissue of diabetic rats. , 1994, Endocrinology.

[122]  J. Cimino,et al.  Formation of the envelope of rous sarcoma virus and vesicular stomatitis virus from localized lipid regions in the plasma membrane. , 1982, Biophysical journal.

[123]  D. Storm,et al.  Effect of membrane phospholipid composition changes on adenylate cyclase activity in normal and rous-sarcoma-transformed chicken embryo fibroblasts. , 1980, Biochimica et biophysica acta.

[124]  J. Pessin,et al.  Printed in U.S.A. Copyright © 2001 by The Endocrine Society The MEF2A and MEF2D Isoforms Are Differentially Regulated in Muscle and Adipose Tissue during States of Insulin Deficiency* , 2022 .