Landscape of the PARKIN-dependent ubiquitylome in response to mitochondrial depolarization
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Steven P. Gygi | Edward L. Huttlin | J. Wade Harper | S. Gygi | M. Sowa | J. Harper | E. Huttlin | Shireen A. Sarraf | M. Raman | Malavika Raman | Virginia Guarani-Pereira | Mathew E. Sowa | Virginia Guarani‐Pereira | Malavika Raman | J. W. Harper
[1] Rachel E. Klevit,et al. UbcH7 reactivity profile reveals Parkin and HHARI to be RING/HECT hybrids , 2011, Nature.
[2] S. Gygi,et al. Defining the Human Deubiquitinating Enzyme Interaction Landscape , 2009, Cell.
[3] Steven P Gygi,et al. A probability-based approach for high-throughput protein phosphorylation analysis and site localization , 2006, Nature Biotechnology.
[4] T. Dawson,et al. The role of parkin in familial and sporadic Parkinson's disease , 2010, Movement disorders : official journal of the Movement Disorder Society.
[5] Steven P Gygi,et al. The SCX/IMAC enrichment approach for global phosphorylation analysis by mass spectrometry , 2008, Nature Protocols.
[6] Miratul M. K. Muqit,et al. PINK1 is activated by mitochondrial membrane potential depolarization and stimulates Parkin E3 ligase activity by phosphorylating Serine 65 , 2012, Open Biology.
[7] Maki Maeda,et al. Fis1 acts as a mitochondrial recruitment factor for TBC1D15 that is involved in regulation of mitochondrial morphology , 2013, Journal of Cell Science.
[8] R. Youle,et al. Role of PINK1 binding to the TOM complex and alternate intracellular membranes in recruitment and activation of the E3 ligase Parkin. , 2012, Developmental cell.
[9] W. Saxton,et al. Parkinson's Disease–Associated Kinase PINK1 Regulates Miro Protein Level and Axonal Transport of Mitochondria , 2012, PLoS genetics.
[10] Brad T. Sherman,et al. Systematic and integrative analysis of large gene lists using DAVID bioinformatics resources , 2008, Nature Protocols.
[11] Xinnan Wang,et al. PINK1 and Parkin Target Miro for Phosphorylation and Degradation to Arrest Mitochondrial Motility , 2011, Cell.
[12] Hyeseong Cho,et al. Mitofusin 1 is degraded at G2/M phase through ubiquitylation by MARCH5 , 2012, Cell Division.
[13] Edward L. Huttlin,et al. A Tissue-Specific Atlas of Mouse Protein Phosphorylation and Expression , 2010, Cell.
[14] Fabienne C. Fiesel,et al. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 , 2010, Nature Cell Biology.
[15] R. Youle,et al. Proteasome and p97 mediate mitophagy and degradation of mitofusins induced by Parkin , 2010, The Journal of cell biology.
[16] Edward L. Huttlin,et al. Systematic and quantitative assessment of the ubiquitin-modified proteome. , 2011, Molecular cell.
[17] Michael Lazarou,et al. PINK1 drives Parkin self-association and HECT-like E3 activity upstream of mitochondrial binding , 2013, The Journal of cell biology.
[18] R. Youle,et al. Mechanisms of mitophagy , 2010, Nature Reviews Molecular Cell Biology.
[19] N. Mizushima,et al. Parkin Mediates Proteasome-dependent Protein Degradation and Rupture of the Outer Mitochondrial Membrane*♦ , 2011, The Journal of Biological Chemistry.
[20] J. Yates,et al. An approach to correlate tandem mass spectral data of peptides with amino acid sequences in a protein database , 1994, Journal of the American Society for Mass Spectrometry.
[21] Angela C. Poole,et al. The Mitochondrial Fusion-Promoting Factor Mitofusin Is a Substrate of the PINK1/Parkin Pathway , 2010, PloS one.
[22] A. Vashisht,et al. Voltage-dependent Anion Channels (VDACs) Recruit Parkin to Defective Mitochondria to Promote Mitochondrial Autophagy* , 2012, The Journal of Biological Chemistry.
[23] A. Whitworth,et al. Drosophila Parkin requires PINK1 for mitochondrial translocation and ubiquitinates Mitofusin , 2010, Proceedings of the National Academy of Sciences.
[24] R. Youle,et al. Mitochondrial quality control mediated by PINK1 and Parkin: links to parkinsonism. , 2012, Cold Spring Harbor perspectives in biology.
[25] Brendan K Faherty,et al. Optimization and Use of Peptide Mass Measurement Accuracy in Shotgun Proteomics*S , 2006, Molecular & Cellular Proteomics.
[26] Sonja Hess,et al. Broad activation of the ubiquitin–proteasome system by Parkin is critical for mitophagy , 2011, Human molecular genetics.
[27] H. Walden,et al. Autoregulation of Parkin activity through its ubiquitin‐like domain , 2011, The EMBO journal.
[28] S. Bloor,et al. LC3C, Bound Selectively by a Noncanonical LIR Motif in NDP52, Is Required for Antibacterial Autophagy , 2012, Molecular cell.
[29] N. Hattori,et al. Parkin binds the Rpn10 subunit of 26S proteasomes through its ubiquitin‐like domain , 2003, EMBO reports.
[30] L. Chin,et al. Phosphorylation of parkin by Parkinson disease-linked kinase PINK1 activates parkin E3 ligase function and NF-kappaB signaling. , 2010, Human molecular genetics.
[31] Sarah Sonnay,et al. Parkin promotes the ubiquitination and degradation of the mitochondrial fusion factor mitofusin 1 , 2011, Journal of neurochemistry.
[32] R. Youle,et al. p62/SQSTM1 is required for Parkin-induced mitochondrial clustering but not mitophagy; VDAC1 is dispensable for both , 2010, Autophagy.
[33] Y. Yoon,et al. Control of Mitochondrial Morphology Through Differential Interactions of Mitochondrial Fusion and Fission Proteins , 2011, PloS one.
[34] David S. Park,et al. ROS-dependent regulation of Parkin and DJ-1 localization during oxidative stress in neurons. , 2012, Human molecular genetics.
[35] Sebastian A. Wagner,et al. A Proteome-wide, Quantitative Survey of In Vivo Ubiquitylation Sites Reveals Widespread Regulatory Roles* , 2011, Molecular & Cellular Proteomics.
[36] R. Youle,et al. Parkin is recruited selectively to impaired mitochondria and promotes their autophagy , 2008, The Journal of cell biology.