Attenuation by intravenous 2-chloroadenosine of acute lung injury induced by live escherichia coli or latex particles added to endotoxin in the neutropenic state.

[1]  R. Saura,et al.  2‐Chloroadenosine but not adenosine induces apoptosis in rheumatoid fibroblasts independently of cell surface adenosine receptor signalling , 2002, British journal of pharmacology.

[2]  A. Ohta,et al.  Role of G-protein-coupled adenosine receptors in downregulation of inflammation and protection from tissue damage , 2001, Nature.

[3]  B. Cronstein,et al.  Inflammatory Cytokines Regulate Function and Expression of Adenosine A2A Receptors in Human Monocytic THP-1 Cells1 , 2001, The Journal of Immunology.

[4]  E. Deitch,et al.  Adenosine inhibits IL-12 and TNF-a production via adenosine A 2 a receptor-dependent and independent mechanisms , 2000 .

[5]  K. Messmer,et al.  Effects of adenosine on the functions of circulating polymorphonuclear leukocytes during hyperdynamic endotoxemia , 1997, Infection and immunity.

[6]  C. Edwards,et al.  Adenosine A3 receptor agonists inhibit murine macrophage tumor necrosis factor-alpha production in vitro and in vivo. , 1997, Cellular and molecular biology.

[7]  E. Vizi,et al.  Adenosine receptor agonists differentially regulate IL-10, TNF-alpha, and nitric oxide production in RAW 264.7 macrophages and in endotoxemic mice. , 1996, Journal of immunology.

[8]  A. Ishizaka,et al.  Augmentation of endotoxin-induced pulmonary responses by mononuclear cell phagocytosis in the reticuloendothelial system. , 1996, Critical care medicine.

[9]  A. Ishizaka,et al.  Granulocyte colony-stimulating factor exacerbates acute lung injury induced by intratracheal endotoxin in guinea pigs. , 1994, American journal of respiratory and critical care medicine.

[10]  D. Morrison,et al.  Adenosine and a related carbocyclic nucleoside analogue selectively inhibit tumor necrosis factor-alpha production and protect mice against endotoxin challenge. , 1993, Journal of immunology.

[11]  H. Reynolds,et al.  Macrophages and polymorphonuclear neutrophils in lung defense and injury. , 1990, The American review of respiratory disease.

[12]  E. Brown,et al.  Adenosine receptors are expressed during differentiation of monocytes to macrophages in vitro. Implications for regulation of phagocytosis. , 1989, Journal of immunology.

[13]  C. Nathan,et al.  Adenosine regulates the respiratory burst of cytokine-triggered human neutrophils adherent to biologic surfaces. , 1989, Journal of immunology.

[14]  T. Raffin,et al.  Granulocyte depletion prevents tumor necrosis factor-mediated acute lung injury in guinea pigs. , 1988, The American review of respiratory disease.

[15]  R. Centor,et al.  The adult respiratory distress syndrome. Cell populations and soluble mediators in the air spaces of patients at high risk. , 1987, The American review of respiratory disease.

[16]  G. Weissmann,et al.  Adenosine: an endogenous inhibitor of neutrophil-mediated injury to endothelial cells. , 1986, The Journal of clinical investigation.

[17]  F. Ognibene,et al.  Adult respiratory distress syndrome in patients with severe neutropenia. , 1986, The New England journal of medicine.

[18]  R. Simon,et al.  Adult respiratory distress syndrome in neutropenic patients. , 1986, The American journal of medicine.

[19]  P. Dorinsky,et al.  Lung neutrophils in the adult respiratory distress syndrome. Clinical and pathophysiologic significance. , 1986, The American review of respiratory disease.

[20]  T. Saito,et al.  2-Chloroadenosine: a selective lethal effect to mouse macrophages and its mechanism. , 1985, Journal of immunology.

[21]  C. Griscelli,et al.  Separation of a population of human T lymphocytes that bind prostaglandin E2 and exert a suppressor activity. , 1985, Journal of immunology.

[22]  H. Walzl,et al.  Studies of lung infections caused by Pseudomonas aeruginosa in mice treated with cyclophosphamide , 1983, Infection.

[23]  Y. Kumazawa,et al.  Inhibition of macrophage function by 2-chloroadenosine. , 1982, Journal of the Reticuloendothelial Society.

[24]  K. Brigham,et al.  Prevention by granulocyte depletion of increased vascular permeability of sheep lung following endotoxemia. , 1981, The Journal of clinical investigation.

[25]  R. Wallace,et al.  Proteins iodinated by the chloramine-T method appear to be degraded at an abnormally rapid rate after endocytosis. , 1980, Proceedings of the National Academy of Sciences of the United States of America.

[26]  L. Chauvelot‐Moachon,et al.  Inhibition of human monocyte TNF production by adenosine receptor agonists. , 1993, Life sciences.

[27]  A. Ishizaka,et al.  Granulocyte colony-stimulating factor does not enhance endotoxin-induced acute lung injury in guinea pigs. , 1992, The American review of respiratory disease.

[28]  R. Furth Origin and turnover of monocytes and macrophages. , 1989 .

[29]  D. Adams,et al.  Cell Kinetics of the Inflammatory Reaction , 1989, Current Topics in Pathology.

[30]  E. W. Hall,et al.  Pulmonary edema after Escherichia coli peritonitis correlates with thiobarbituric-acid-reactive materials in bronchoalveolar lavage fluid. , 1988, The American review of respiratory disease.

[31]  E. Chi,et al.  Neutrophil depletion does not prevent lung edema after endotoxin infusion in goats. , 1987, Journal of applied physiology.

[32]  John E. Bennett,et al.  Principles and practice of infectious diseases. Vols 1 and 2. , 1979 .