p16INK4a gene promoter variation and differential binding of a repressor, the ras-responsive zinc-finger transcription factor, RREB
暂无分享,去创建一个
Douglas R Lowy | D. Lowy | E. Ramsay | B. Mock | Xiaolan Qian | Shuling Zhang | X. Qian | Beverly A Mock | Chanelle Redman | Valeri Bliskovski | Edward S Ramsay | Shuling Zhang | C. Redman | V. Bliskovski
[1] R. T. Brown,et al. An Acute Phase Response Factor/NF-B Site Downstream of the junB Gene That Mediates Responsiveness to Interleukin-6 in a Murine Plasmacytoma (*) , 1995, The Journal of Biological Chemistry.
[2] M. Potter,et al. Genetic Considerations in Plasmacytomas of BALB/c, NZB, and (BALB/c X NZB) F1 Mice , 1980 .
[3] D. Carrasco,et al. Loss of p16Ink4a with retention of p19Arf predisposes mice to tumorigenesis , 2001, Nature.
[4] S. Baylin,et al. Sequence-specific DNA Binding Activity of RNA Helicase A to thep16INK4a Promoter* , 2001, The Journal of Biological Chemistry.
[5] J. Stone,et al. RAS signalling is abnormal in a c-raf1 MEK1 double mutant , 1995, Molecular and cellular biology.
[6] M. You,et al. The 5′-flanking region of the E1 α form of the murine p16INK4a (MTS1) gene ☆ , 1996 .
[7] Peter A. Jones,et al. The fundamental role of epigenetic events in cancer , 2002, Nature Reviews Genetics.
[8] J. Sambrook,et al. Molecular Cloning: A Laboratory Manual , 2001 .
[9] S. Baylin,et al. Altered methylation patterns in cancer cell genomes: cause or consequence? , 2002, Cancer cell.
[10] M. You,et al. Cdkn2a encodes functional variation of p16INK4a but not p14ARF, which confers selection in mouse lung tumorigenesis , 1999, Molecular carcinogenesis.
[11] David M. Livingston,et al. Functional interactions of the retinoblastoma protein with mammalian D-type cyclins , 1993, Cell.
[12] Y. Qian,et al. The retinoblastoma gene product regulates progression through the G1 phase of the cell cycle , 1991, Cell.
[13] T. Tong,et al. Characterization of Regulatory Elements on the Promoter Region of p16INK4a That Contribute to Overexpression of p16 in Senescent Fibroblasts* , 2001, The Journal of Biological Chemistry.
[14] B. Mock,et al. The role of p16INK4a (Cdkn2a) in mouse plasma cell tumors. , 1999, Current topics in microbiology and immunology.
[15] M. Potter,et al. The plasmacytoma resistance gene, Pctr2, delays the onset of tumorigenesis and resides in the telomeric region of chromosome 4. , 1997, Blood.
[16] A. Berns,et al. Loss of p16Ink4a confers susceptibility to metastatic melanoma in mice , 2001, Nature.
[17] M. Ewen,et al. Direct binding of cyclin D to the retinoblastoma gene product (pRb) and pRb phosphorylation by the cyclin D-dependent kinase CDK4. , 1993, Genes & development.
[18] E. Wagner,et al. JunB suppresses cell proliferation by transcriptional activation of p16INK4a expression , 2000, The EMBO journal.
[19] D. Dilworth,et al. Germline CDKN2A mutation implicated in predisposition to multiple myeloma. , 2000, Blood.
[20] W. Clark,et al. Germline p16 mutations in familial melanoma , 1994, Nature Genetics.
[21] S. Shurtleff,et al. D-type cyclin-dependent kinase activity in mammalian cells , 1994, Molecular and cellular biology.
[22] G. G. Stokes. "J." , 1890, The New Yale Book of Quotations.
[23] M Mabry,et al. RREB-1, a novel zinc finger protein, is involved in the differentiation response to Ras in human medullary thyroid carcinomas , 1996, Molecular and cellular biology.
[24] B. Mock,et al. Genetic mapping of tumor susceptibility genes involved in mouse plasmacytomagenesis. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[25] E. Ramsay,et al. Cdkn2a, the cyclin-dependent kinase inhibitor encoding p16INK4a and p19ARF, is a candidate for the plasmacytoma susceptibility locus, Pctr1. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[26] S. Rudikoff,et al. Interleukin 6 is essential for in vivo development of B lineage neoplasms , 1995, The Journal of experimental medicine.
[27] G. Peters,et al. Opposing effects of Ets and Id proteins on p16INK4a expression during cellular senescence , 2001, Nature.
[28] G. Gores,et al. p16INK4a promoter mutations are frequent in primary sclerosing cholangitis (PSC) and PSC-associated cholangiocarcinoma. , 2002, Gastroenterology.
[29] R. Alani,et al. Id1 regulation of cellular senescence through transcriptional repression of p16/Ink4a , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[30] R. Bataille,et al. IL‐6 up‐regulates Mcl‐1 in human myeloma cells through JAK / STAT rather than Ras / MAP kinase pathway , 1999, European journal of immunology.
[31] R. Nordan,et al. A macrophage-derived factor required by plasmacytomas for survival and proliferation in vitro. , 1986, Science.
[32] R. DePinho,et al. Efficiency Alleles of the Pctr1 Modifier Locus for Plasmacytoma Susceptibility , 2001, Molecular and Cellular Biology.
[33] M. Potter,et al. Induction of plasmacytomas with silicone gel in genetically susceptible strains of mice. , 1994, Journal of the National Cancer Institute.
[34] M. Potter,et al. Induction of Plasma-Cell Neoplasms in Strain BALB/c Mice with Mineral Oil and Mineral Oil Adjuvants , 1962, Nature.
[35] D. Lowy,et al. Ras-Specific Exchange Factor GRF: Oligomerization through Its Dbl Homology Domain and Calcium-Dependent Activation of Raf , 1999, Molecular and Cellular Biology.