Down-regulation of CXCL1 inhibits tumor growth in colorectal liver metastasis.

[1]  A. Boulesteix,et al.  Differential expression of the chemokines GRO-2, GRO-3, and interleukin-8 in colon cancer and their impact on metastatic disease and survival , 2010, International Journal of Colorectal Disease.

[2]  R. Strieter,et al.  CXC chemokines in cancer angiogenesis and metastases. , 2010, Advances in cancer research.

[3]  P. Schirmacher,et al.  Transcriptional activation of the β‐catenin gene at the invasion front of colorectal liver metastases , 2009, The Journal of pathology.

[4]  P. Allavena,et al.  Expression of chemokines and chemokine receptors in human colon cancer. , 2009, Methods in enzymology.

[5]  P. Schirmacher,et al.  Cross-species comparison of biological themes and underlying genes on a global gene expression scale in a mouse model of colorectal liver metastasis and in clinical specimens , 2008, BMC Genomics.

[6]  P. Johnston,et al.  Chemotherapy-Induced CXC-Chemokine/CXC-Chemokine Receptor Signaling in Metastatic Prostate Cancer Cells Confers Resistance to Oxaliplatin through Potentiation of Nuclear Factor-κB Transcription and Evasion of Apoptosis , 2008, Journal of Pharmacology and Experimental Therapeutics.

[7]  J. Van Damme,et al.  The role of CXC chemokines and their receptors in cancer. , 2008, Cancer letters.

[8]  P. Schirmacher,et al.  Invasion front-specific overexpression of tissue inhibitor of metalloproteinase-1 in liver metastases from colorectal cancer. , 2008, Anticancer research.

[9]  G. Tsujimoto,et al.  Secreted CXCL1 Is a Potential Mediator and Marker of the Tumor Invasion of Bladder Cancer , 2008, Clinical Cancer Research.

[10]  A. Krüger,et al.  Distinct Functionality of Tumor Cell–Derived Gelatinases during Formation of Liver Metastases , 2008, Molecular Cancer Research.

[11]  A. Krüger,et al.  Tissue inhibitor of metalloproteinases-1 promotes liver metastasis by induction of hepatocyte growth factor signaling. , 2007, Cancer research.

[12]  D. Notterman,et al.  GROα Is Highly Expressed in Adenocarcinoma of the Colon and Down-Regulates Fibulin-1 , 2006, Clinical Cancer Research.

[13]  R. Strieter,et al.  CXCL1 induced by prostaglandin E2 promotes angiogenesis in colorectal cancer , 2006, The Journal of experimental medicine.

[14]  M. Burdick,et al.  Cancer CXC chemokine networks and tumour angiogenesis. , 2006, European journal of cancer.

[15]  H. Herzel,et al.  Global analysis of host tissue gene expression in the invasive front of colorectal liver metastases. , 2005, International journal of cancer.

[16]  Yan Zhou,et al.  The chemokine GRO-α (CXCL1) confers increased tumorigenicity to glioma cells , 2005 .

[17]  U. Hopt,et al.  Chemokines in human colorectal carcinoma. , 2005, Anticancer research.

[18]  F. Hsieh,et al.  Clinicopathologic Correlation of Up-regulated Genes Identified Using cDNA Microarray and Real-time Reverse Transcription-PCR in Human Colorectal Cancer , 2005, Cancer Epidemiology Biomarkers & Prevention.

[19]  Yan Zhou,et al.  The chemokine GRO-alpha (CXCL1) confers increased tumorigenicity to glioma cells. , 2005, Carcinogenesis.

[20]  Aihua Li,et al.  Constitutive expression of growth regulated oncogene (gro) in human colon carcinoma cells with different metastatic potential and its role in regulating their metastatic phenotype , 2005, Clinical & Experimental Metastasis.

[21]  A. Krüger,et al.  Adenovirus‐mediated overexpression of tissue inhibitor of metalloproteinases‐1 in the liver: efficient protection against T‐cell lymphoma and colon carcinoma metastasis , 2004, The journal of gene medicine.

[22]  G. Opdenakker,et al.  The countercurrent principle in invasion and metastasis of cancer cells. Recent insights on the roles of chemokines. , 2004, The International journal of developmental biology.

[23]  P. Dhawan,et al.  Role of CXCL1 in tumorigenesis of melanoma , 2002, Journal of leukocyte biology.

[24]  B. Hyman,et al.  GROα/KC, a chemokine receptor CXCR2 ligand, can be a potent trigger for neuronal ERK1/2 and PI-3 kinase pathways and for tau hyperphosphorylation—a role in Alzheimer's disease? , 2002, Journal of Neuroimmunology.

[25]  B. Hyman,et al.  GROalpha/KC, a chemokine receptor CXCR2 ligand, can be a potent trigger for neuronal ERK1/2 and PI-3 kinase pathways and for tau hyperphosphorylation-a role in Alzheimer's disease? , 2002, Journal of neuroimmunology.

[26]  L. Chin,et al.  Induction of melanoma in murine macrophage inflammatory protein 2 transgenic mice heterozygous for inhibitor of kinase/alternate reading frame. , 2001, Cancer research.

[27]  A. Richmond,et al.  Nuclear Factor-κB Activation by the CXC Chemokine Melanoma Growth-stimulatory Activity/Growth-regulated Protein Involves the MEKK1/p38 Mitogen-activated Protein Kinase Pathway* , 2001, The Journal of Biological Chemistry.

[28]  D. Wang,et al.  Nuclear factor-kappa B activation by the CXC chemokine melanoma growth-stimulatory activity/growth-regulated protein involves the MEKK1/p38 mitogen-activated protein kinase pathway. , 2001, The Journal of biological chemistry.

[29]  K. Brand,et al.  Treatment of colorectal liver metastases by adenoviral transfer of tissue inhibitor of metalloproteinases-2 into the liver tissue. , 2000, Cancer research.

[30]  M. Burdick,et al.  Distinct CXC chemokines mediate tumorigenicity of prostate cancer cells. , 1999, The American journal of pathology.

[31]  M. Burdick,et al.  Mechanism and biological significance of constitutive expression of MGSA/GRO chemokines in malignant melanoma tumor progression , 1997, Journal of leukocyte biology.

[32]  J. Grem.  Current treatment approaches in colorectal cancer. , 1991, Seminars in oncology.

[33]  Gustav Wagner,et al.  Krebsatlas der Bundesrepublik Deutschland / Cancer Atlas of the Federal Republic of Germany , 1979 .