Accumulation of extracellular ATP protects against acute reperfusion injury in rat heart endothelial cells.

[1]  F. Härtel,et al.  Opposite effect of cAMP signaling in endothelial barriers of different origin. , 2004, American journal of physiology. Cell physiology.

[2]  F. Härtel,et al.  MEK/MAPK as a signaling element in ATP control of endothelial myosin light chain. , 2004, American journal of physiology. Cell physiology.

[3]  J. Kruskal,et al.  Beneficial effects of CD39/ecto-nucleoside triphosphate diphosphohydrolase-1 in murine intestinal ischemia-reperfusion injury , 2004, Thrombosis and Haemostasis.

[4]  F. Hirche,et al.  ATP antagonism of thrombin-induced endothelial barrier permeability. , 2003, Cardiovascular research.

[5]  C. Schäfer,et al.  Inhibition of contractile activation reduces reoxygenation-induced endothelial gap formation. , 2003, Cardiovascular research.

[6]  J. Sévigny,et al.  Differential catalytic properties and vascular topography of murine nucleoside triphosphate diphosphohydrolase 1 (NTPDase1) and NTPDase2 have implications for thromboregulation. , 2002, Blood.

[7]  E. Schwiebert,et al.  Extracellular ATP signaling and P2X nucleotide receptors in monolayers of primary human vascular endothelial cells. , 2002, American journal of physiology. Cell physiology.

[8]  S J Allen,et al.  Myocardial fluid balance. , 2001, European journal of cardio-thoracic surgery : official journal of the European Association for Cardio-thoracic Surgery.

[9]  G Burnstock,et al.  Evidence That Release of Adenosine Triphosphate From Endothelial Cells During Increased Shear Stress Is Vesicular , 2001, Journal of cardiovascular pharmacology.

[10]  Geoffrey Burnstock,et al.  Purinergic Signalling: ATP Release , 2001, Neurochemical Research.

[11]  I. Rubinstein,et al.  Adenosine A(1) receptors mediate plasma exudation from the oral mucosa. , 2001, Journal of applied physiology.

[12]  T. Noll,et al.  ATP induces dephosphorylation of myosin light chain in endothelial cells. , 2000, American journal of physiology. Cell physiology.

[13]  H. Zimmermann.  Extracellular metabolism of ATP and other nucleotides , 2000, Naunyn-Schmiedeberg's Archives of Pharmacology.

[14]  G. Burnstock,et al.  Effect of shear stress on the release of soluble ecto‐enzymes ATPase and 5′‐nucleotidase along with endogenous ATP from vascular endothelial cells , 2000, British journal of pharmacology.

[15]  Patricia D. Christie,et al.  Targeted disruption of cd39/ATP diphosphohydrolase results in disordered hemostasis and thromboregulation , 1999, Nature Medicine.

[16]  S. Hourani,et al.  Differential effects of adenine nucleotide analogues on shape change and aggregation induced by adenosine 5′‐diphosphate (ADP) in human platelets , 1999, British Journal of Pharmacology.

[17]  Deniz Gündüz,et al.  ATP reduces macromolecule permeability of endothelial monolayers despite increasing [Ca2+]i. , 1999, American journal of physiology. Heart and circulatory physiology.

[18]  T. Noll,et al.  Effect of Factor XIII on Endothelial Barrier Function , 1999, The Journal of experimental medicine.

[19]  E. Schwiebert,et al.  Bioluminescence detection of ATP release mechanisms in epithelia. , 1998, The American journal of physiology.

[20]  G. Burnstock,et al.  Increased release of ATP from endothelial cells during acute inflammation , 1998, Inflammation Research.

[21]  D. Statile,et al.  [Effect of A1 adenosine receptor blockade on postischemic damage to the coronary microcirculation]. , 1998, Cardiologia.

[22]  F. Bach,et al.  Identification and Characterization of CD39/Vascular ATP Diphosphohydrolase* , 1996, The Journal of Biological Chemistry.

[23]  J. Greelish,et al.  A1 adenosine receptor antagonists block ischemia-reperfusion injury of the heart. , 1996, Circulation.

[24]  T. Noll,et al.  Functional antagonism between cAMP and cGMP on permeability of coronary endothelial monolayers. , 1996, The American journal of physiology.

[25]  C. Kennedy,et al.  Enhancement of sympathetic purinergic neurotransmission in the guinea‐pig isolated vas deferens by the novel ecto‐ATPase inhibitor ARL 67156 , 1996, British journal of pharmacology.

[26]  R B Wysolmerski,et al.  Myosin light chain kinase-regulated endothelial cell contraction: the relationship between isometric tension, actin polymerization, and myosin phosphorylation , 1995, The Journal of cell biology.

[27]  I. Keith,et al.  A1 adenosine receptor antagonists block ischemia-reperfusion injury of the lung. , 1995, The American journal of physiology.

[28]  A. IJzerman,et al.  Pharmacological and biochemical analysis of FPL 67156, a novel, selective inhibitor of ecto‐ATPase , 1995, British journal of pharmacology.

[29]  D. Garcia-Dorado,et al.  Myocardial oedema: a preventable cause of reperfusion injury? , 1993, Cardiovascular research.

[30]  P. Schwartz,et al.  A2-adenosine receptor stimulation increases macromolecule permeability of coronary endothelial cells. , 1992, The American journal of physiology.

[31]  D. Heistad,et al.  Role of protein kinase C in bradykinin-induced increases in microvascular permeability. , 1991, Circulation research.

[32]  R. Spahr,et al.  Macromolecule permeability of coronary and aortic endothelial monolayers under energy depletion. , 1991, The American journal of physiology.

[33]  J. Pearson,et al.  The hydrolysis of extracellular adenine nucleotides by cultured endothelial cells from pig aorta. Feed-forward inhibition of adenosine production at the cell surface. , 1986, The Journal of biological chemistry.

[34]  W. Durán,et al.  Dose‐Related Effects of Adenosine and Bradykinin on Microvascular Permselectivity to Macromolecules in the Hamster Cheek Pouch , 1986, Circulation research.

[35]  D. Durrer,et al.  Tissue Osmolality, Cell Swelling, and Reperfusion in Acute Regional Myocardial Ischemia in the Isolated Porcine Heart , 1981, Circulation research.

[36]  T. Forrester,et al.  Appearance of adenosine triphosphate in the coronary sinus effluent from isolated working rat heart in response to hypoxia. , 1981, The Journal of physiology.

[37]  D. Garcia-Dorado,et al.  Cardiovascular Research , 1966 .

[38]  G. Burnstock,et al.  Cellular distribution and functions of P2 receptor subtypes in different systems. , 2004, International review of cytology.

[39]  F. Curry,et al.  Microvascular permeability. , 1999, Physiological reviews.

[40]  T. Forrester,et al.  Release of ATP from human erythrocytes in response to a brief period of hypoxia and hypercapnia. , 1992, Cardiovascular research.