Senescence-accelerated mouse (SAM): a biogerontological resource in aging research

[1]  P. Jansen-Dürr,et al.  Diseases of aging. , 2000, Vaccine.

[2]  A. Morley,et al.  Accelerated accumulation of somatic mutations in the senescence-accelerated mouse , 1998, Nature Genetics.

[3]  A. Parfitt,et al.  Increased Adipogenesis and Myelopoiesis in the Bone Marrow of SAMP6, a Murine Model of Defective Osteoblastogenesis and Low Turnover Osteopenia , 1997, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

[4]  M. Hosokawa,et al.  Pathobiology of the senescence-accelerated mouse (SAM) , 1997, Experimental Gerontology.

[5]  M. Kishikawa,et al.  Herbal medicine and the study of aging in senescence-accelerated mice (SAMP1TA/Ngs) , 1997, Experimental Gerontology.

[6]  D. Butterfield,et al.  Free radical oxidation of brain proteins in accelerated senescence and its modulation by N-tert-butyl-α-phenylnitrone , 1997 .

[7]  M. Chung,et al.  Oxidative status in senescence-accelerated mice. , 1996, The journals of gerontology. Series A, Biological sciences and medical sciences.

[8]  T. Takeda.  [Senescence-accelerated mouse (SAM): with special reference to age-associated pathologies and their modulation]. , 1996, Nihon eiseigaku zasshi. Japanese journal of hygiene.

[9]  A. Parfitt,et al.  Linkage of decreased bone mass with impaired osteoblastogenesis in a murine model of accelerated senescence. , 1996, The Journal of clinical investigation.

[10]  D. Harrison.  The SAM model of senescence Edited by Toshio Takeda, Elsevier Science B. V., P. O. Box 211, 1000 AE Amsterdam, The Netherlands, 1994, ISBN 0-444-81695-x, 458 pp , 1995, Experimental Gerontology.

[11]  L. Packer,et al.  The Spin-Trap N-tert-α-Phenylbutylnitrone Prolongs the Life Span of the Senescence-Accelerated Mouse , 1995 .

[12]  K. Meguro,et al.  Effects of thioperamide, a histamine H3 antagonist, on the step-through passive avoidance response and histidine decarboxylase activity in senescence-accelerated mice , 1995, Pharmacology Biochemistry and Behavior.

[13]  H. Tojo,et al.  Dietary α-linolenate/linoleate balance influences learning and memory in the senescence-accelerated mouse (SAM) , 1995, Brain Research.

[14]  T. Heya,et al.  Effects of a sustained release formulation of thyrotropin-releasing hormone on behavioral abnormalities in senescence-accelerated mice. , 1994, European journal of pharmacology.

[15]  M. Hosokawa,et al.  Accelerated aging of dermal fibroblast-like cells from senescence-accelerated mouse (SAM). 1. Acceleration of population aging in vitro , 1994, Mechanisms of Ageing and Development.

[16]  T. Yamamuro,et al.  Effective intervention of low peak bone mass and bone modeling in the spontaneous murine model of senile osteoporosis, SAM-P/6, by Ca supplement and hormone treatment. , 1994, Bone.

[17]  A. Shimada,et al.  Genetic analysis of murine senile amyloidosis. , 1993, Laboratory investigation; a journal of technical methods and pathology.

[18]  K. Kitagawa,et al.  Mouse senile amyloidosis , 1991, Virchows Archiv. B, Cell pathology including molecular pathology.

[19]  K. Kitagawa,et al.  Polymorphism of apolipoprotein A-II (apoA-II) among inbred strains of mice. Relationship between the molecular type of apoA-II and mouse senile amyloidosis. , 1991, The Biochemical journal.

[20]  M. Hosokawa,et al.  Senescence‐Accelerated Mouse (SAM): A Novel Murine Model of Accelerated Senescence , 1991, Journal of the American Geriatrics Society.

[21]  K. Higuchi,et al.  Kinetic analysis of amyloid fibril polymerization in vitro. , 1991, Laboratory investigation; a journal of technical methods and pathology.

[22]  Y. Kitamura,et al.  Effects of bifemelane hydrochloride, a brain function improver, on muscarinic receptors in the CNS of senescence-accelerated mouse. , 1991, Japanese journal of pharmacology.

[23]  M. Hosokawa,et al.  Effects of dietary restriction on age-related immune dysfunction in the senescence accelerated mouse (SAM). , 1990, The Journal of nutrition.

[24]  M. Sasaki,et al.  Acceleration of chromosome aberrations in senescence-accelerated strains of mice. , 1990, Mutation research.

[25]  P. Anthony.  Robbins' Pathologic Basis of Disease , 1990 .

[26]  T. Yamamuro,et al.  Decreased endosteal formation during cortical bone modelling in SAM-P/6 mice with a low peak bone mass. , 1989, Bone and mineral.

[27]  T. Iizuka,et al.  Age-related changes in the temporomandibular joint of the senescence accelerated mouse. SAM-P/3 as a new murine model of degenerative joint disease. , 1989, The American journal of pathology.

[28]  M. Hosokawa,et al.  Fluorometric determination of amyloid fibrils in vitro using the fluorescent dye, thioflavin T1. , 1989, Analytical biochemistry.

[29]  K. Higuchi,et al.  A molecular-pathologic approach to murine senile amyloidosis. Serum precursor-apolipoprotein A-II variant (Pro5----Gln) presents only in the senile amyloidosis-prone SAM-P/1 and SAM-P/2 mice. , 1987, Laboratory investigation; a journal of technical methods and pathology.

[30]  M. Hosokawa,et al.  Purification and characterization of a senile amyloid-related antigenic substance (apoSASSAM) from mouse serum. apoSASSAM is an apoA-II apolipoprotein of mouse high density lipoproteins. , 1986, The Journal of biological chemistry.

[31]  K. Higuchi,et al.  High homology is present in the primary structures between murine senile amyloid protein (ASSAM) and human apolipoprotein A‐II , 1986, FEBS letters.

[32]  S. Aota,et al.  Molecular cloning and nucleotide sequence of cDNA for murine senile amyloid protein: nucleotide substitutions found in apolipoprotein A-II cDNA of senescence accelerated mouse (SAM). , 1986, Nucleic acids research.

[33]  M. Hosokawa,et al.  Chronic food restriction modulates the advance of senescence in the senescence accelerated mouse (SAM). , 1985, The Journal of nutrition.

[34]  M. Hosokawa,et al.  Grading score system: A method for evaluation of the degree of senescence in Senescence Accelerated Mouse (SAM) , 1984, Mechanisms of Ageing and Development.

[35]  L. Melton,et al.  Evidence for two distinct syndromes of involutional osteoporosis. , 1983, The American journal of medicine.

[36]  M. Hosokawa,et al.  Systemic senile amyloid in senescence-accelerated mice. A unique fibril protein demonstrated in tissues from various organs by the unlabeled immunoperoxidase method. , 1983, Laboratory investigation; a journal of technical methods and pathology.

[37]  Keiichi Higuchi,et al.  Spontaneous age-associated amyloidosis in senescence-accelerated mouse (SAM) , 1982, Mechanisms of Ageing and Development.

[38]  M. Hosokawa,et al.  A novel amyloid fibril protein isolated from senescence-accelerated mice. , 1982, Laboratory investigation; a journal of technical methods and pathology.

[39]  Keiichi Higuchi,et al.  A new murine model of accelerated senescence , 1981, Mechanisms of Ageing and Development.

[40]  W. Brown,et al.  Hereditary premature sensescence of the rabbit. I. Chronic form; general features. , 1960, The Journal of experimental medicine.

[41]  W. Brown,et al.  Hereditary premature senescence of the rabbit. II. Acute form; general features. , 1960 .

[42]  M. Hosokawa,et al.  Accelerated aging of dermal fibroblast-like cells from the senescence-accelerated mouse (SAM): acceleration of changes in DNA ploidy associated with in vitro cellular aging. , 1998, The journals of gerontology. Series A, Biological sciences and medical sciences.

[43]  M. Hosokawa,et al.  Senescence-accelerated Mouse (SAM): With Special Reference to Development and Pathological Phenotypes. , 1997, ILAR journal.

[44]  Ulrich Mohr,et al.  Pathobiology of the aging mouse , 1996 .

[45]  H. Kimura,et al.  Acidic fibroblast growth factor protects memory and immunoreactivity impairment in senescence accelerated mice. , 1995, Neurobiology.

[46]  K. Higuchi,et al.  Molecular genetic characterization of the senescence-accelerated mouse (SAM) strains. , 1994, Journal of gerontology.

[47]  T. Takeda.  Senescence-Accelerated Mouse (SAM). A novel murine model of aging , 1994 .

[48]  E. Masoro.  Animal Models in Aging Research , 1990 .

[49]  A. Awaya,et al.  Effects of repeated administrations of facteur thymique sérique (FTS) on biochemical changes related to aging in senescence-accelerated mouse (SAM). , 1990, Japanese journal of pharmacology.

[50]  K. Yagi,et al.  Lipid Peroxide Levels in the Skin of the Senescence-Accelerated Mouse , 1988 .

[51]  松下 睦.  Age-related changes in bone mass in the senescence-accelerated mouse (SAM) : SAM-R/3 and SAM-P/6 as new murine models for senile osteoporosis , 1987 .

[52]  M. Hosokawa,et al.  Purification and Characterization of a Senile Amyloid-related Antigenic Substance (apoSASsAM) from Mouse Serum , 1986 .

[53]  Popp Dm.  Use of congenic mice to study the genetic basis of degenerative disease. , 1978 .