Chromatin repression by COUP-TFII and HDAC dominates activation by NF-kappaB in regulating major histocompatibility complex class I transcription in adenovirus tumorigenic cells.
暂无分享,去创建一个
[1] R. Ricciardi,et al. In Adenovirus Type 12 Tumorigenic Cells, Major Histocompatibility Complex Class I Transcription Shutoff Is Overcome by Induction of NF-κB and Relief of COUP-TFII Repression , 2002, Journal of Virology.
[2] Xiaohong Liu,et al. Coup-TFII is up-regulated in adenovirus type 12 tumorigenic cells and is a repressor of MHC class I transcription. , 2001, Virology.
[3] S. Berger,et al. The Histone Modification Circus , 2001, Science.
[4] R. Ricciardi,et al. Association of histone deacetylase with COUP-TF in tumorigenic Ad12-transformed cells and its potential role in shut-off of MHC class I transcription. , 2000, Virology.
[5] C. Glass,et al. Transcriptional Activation by NF-κB Requires Multiple Coactivators , 1999, Molecular and Cellular Biology.
[6] E. Lees,et al. Mammalian Srb/Mediator complex is targeted by adenovirus E1A protein , 1999, Nature.
[7] S. Ghosh,et al. Phosphorylation of NF-kappa B p65 by PKA stimulates transcriptional activity by promoting a novel bivalent interaction with the coactivator CBP/p300. , 1998, Molecular cell.
[8] J. Mazzarelli,et al. The transactivation domain of adenovirus E1A interacts with the C terminus of human TAF(II)135 , 1997, Journal of virology.
[9] M. Gerritsen,et al. CREB-binding protein/p300 are transcriptional coactivators of p65. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[10] M. Tsai,et al. Chick ovalbumin upstream promoter-transcription factors (COUP-TFs): coming of age. , 1997, Endocrine reviews.
[11] D. Kushner,et al. The first exon of Ad12 E1A excluding the transactivation domain mediates differential binding of COUP-TF and NF-kappa B to the MHC class I enhancer in transformed cells. , 1996, Oncogene.
[12] R. Ricciardi,et al. Evidence for the involvement of a nuclear NF-kappa B inhibitor in global down-regulation of the major histocompatibility complex class I enhancer in adenovirus type 12-transformed cells , 1996, Molecular and cellular biology.
[13] J. V. Geisberg,et al. Subregions of the adenovirus E1A transactivation domain target multiple components of the TFIID complex , 1995, Molecular and cellular biology.
[14] R. Ricciardi,et al. E1A oncogene of adenovirus-12 mediates trans-repression of MHC class I transcription in Ad5/Ad12 somatic hybrid transformed cells. , 1994, Virology.
[15] A. Cooney,et al. Negative regulation by the R2 element of the MHC class I enhancer in adenovirus-12 transformed cells correlates with high levels of COUP-TF binding. , 1994, Oncogene.
[16] F. Graham,et al. Tumorigenicity of adenovirus-transformed rodent cells is influenced by at least two regions of adenovirus type 12 early region 1A , 1994, Journal of virology.
[17] P. Yaciuk,et al. The E1A products of oncogenic adenovirus serotype 12 include amino-terminally modified forms able to bind the retinoblastoma protein but not p300 , 1993, Journal of virology.
[18] R. Weinmann,et al. Negative regulation of the major histocompatibility complex class I enhancer in adenovirus type 12-transformed cells via a retinoic acid response element , 1992, Journal of virology.
[19] R. Weinmann,et al. Down-regulation of the major histocompatibility complex class I enhancer in adenovirus type 12-transformed cells is accompanied by an increase in factor binding , 1992, Journal of virology.
[20] Ackrill Am,et al. Regulation of major histocompatibility class I gene expression at the level of transcription in highly oncogenic adenovirus transformed rat cells. , 1988 .
[21] R. Ricciardi,et al. Adenovirus type 12 E1A gene represses accumulation of MHC class I mRNAs at the level of transcription. , 1988, Virology.
[22] R. Ricciardi,et al. Adenovirus type 12 early region 1A proteins repress class I HLA expression in transformed human cells. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[23] A. Israël,et al. Detailed analysis of the mouse H-2Kb promoter: Enhancer-like sequences and their role in the regulation of class I gene expression , 1986, Cell.
[24] E. Appella,et al. Expression of histocompatibility antigens H-2K, -D, and -L is reduced in adenovirus-12-transformed mouse cells and is restored by interferon gamma. , 1985, Proceedings of the National Academy of Sciences of the United States of America.
[25] M. O. Scott,et al. Production of a monospecific antiserum against the early region 1A proteins of adenovirus 12 and adenovirus 5 by an adenovirus 12 early region 1A-beta-galactosidase fusion protein antigen expressed in bacteria , 1984, Journal of virology.
[26] R. Ricciardi,et al. E1A-based determinants of oncogenicity in human adenovirus groups A and C. , 2004, Current topics in microbiology and immunology.
[27] C. Glass,et al. Transcriptional activation by NF-kappaB requires multiple coactivators. , 1999, Molecular and cellular biology.
[28] G. Blair,et al. Regulation of major histocompatibility class I gene expression at the level of transcription in highly oncogenic adenovirus transformed rat cells. , 1988, Oncogene.
[29] B. Oostra,et al. Expression of Class I Major Histocompatibility Antigens in Adenovirus-Transformed Cells , 1986 .
[30] C. Melief,et al. Tumorigenicity of cells transformed by adenovirus type 12 by evasion of T-cell immunity , 1983, Nature.
[31] A. Eb,et al. Expression of class I major histocompatibility antigens switched off by highly oncogenic adenovirus 12 in transformed rat cells , 1983, Nature.