Juvenile dermatomyositis calcifications selectively displayed markers of bone formation.
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[1] L. Wedderburn,et al. Age-dependent inhibition of ectopic calcification: a possible role for fetuin-A and osteopontin in patients with juvenile dermatomyositis with calcinosis. , 2008, Rheumatology.
[2] V. Castronovo,et al. Small integrin-binding ligand N-linked glycoproteins (SIBLINGs): multifunctional proteins in cancer , 2008, Nature Reviews Cancer.
[3] A. Paller,et al. Persistent association of nailfold capillaroscopy changes and skin involvement over thirty-six months with duration of untreated disease in patients with juvenile dermatomyositis. , 2008, Arthritis and rheumatism.
[4] W. Kuis,et al. Calcinosis in juvenile dermatomyositis: a possible role for the vitamin K-dependent protein matrix Gla protein. , 2007, Rheumatology.
[5] M. McKee,et al. Mineral chaperones: a role for fetuin-A and osteopontin in the inhibition and regression of pathologic calcification , 2008, Journal of Molecular Medicine.
[6] R. D'Souza,et al. Dentin Matrix Protein 1 (DMP1): New and Important Roles for Biomineralization and Phosphate Homeostasis , 2007, Journal of dental research.
[7] Honglin Li,et al. Apoptosis in the skeletal muscle of untreated children with juvenile dermatomyositis: impact of duration of untreated disease. , 2007, Clinical immunology.
[8] L. Liaw,et al. Osteopontin: A Multifunctional Molecule Regulating Chronic Inflammation and Vascular Disease , 2007, Arteriosclerosis, thrombosis, and vascular biology.
[9] D. Sosnoski,et al. Evaluation of bone‐derived and marrow‐derived vascular endothelial cells by microarray analysis , 2007, Journal of cellular biochemistry.
[10] J. Gordon,et al. Bone sialoprotein expression enhances osteoblast differentiation and matrix mineralization in vitro. , 2007, Bone.
[11] M. Goldberg,et al. Dentonin, a MEPE Fragment, Initiates Pulp-healing Response to Injury , 2007, Journal of dental research.
[12] G. Schett,et al. Bone histomorphometry in arthritis models. , 2007, Methods in molecular medicine.
[13] A. Freemont,et al. Expression of osteonectin and matrix Gla protein in scleroderma patients with and without calcinosis. , 2006, Rheumatology.
[14] S. Stock,et al. Composition of calcifications in children with juvenile dermatomyositis: association with chronic cutaneous inflammation. , 2006, Arthritis and rheumatism.
[15] J. Lorenzo,et al. Cytokines regulating osteoclast formation and function , 2006, Current opinion in rheumatology.
[16] A. Dyer,et al. Duration of illness is an important variable for untreated children with juvenile dermatomyositis. , 2006, The Journal of pediatrics.
[17] J. Kawaguchi. Generation of osteoblasts and chondrocytes from embryonic stem cells. , 2006, Methods in molecular biology.
[18] Q. Tu,et al. BSP and RANKL Induce Osteoclastogenesis and Bone Resorption Synergistically , 2005, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[19] O. Wendler,et al. Expression and function of laminins in the embryonic and mature vasculature. , 2005, Physiological reviews.
[20] W. Edwards,et al. Calcified Rheumatic Valve Neoangiogenesis Is Associated With Vascular Endothelial Growth Factor Expression and Osteoblast-Like Bone Formation , 2005, Circulation.
[21] A. Dyer,et al. History of infection before the onset of juvenile dermatomyositis: results from the National Institute of Arthritis and Musculoskeletal and Skin Diseases Research Registry. , 2005, Arthritis and rheumatism.
[22] F. Cunha,et al. Macrophages and Mast Cells Control the Neutrophil Migration Induced by Dentin Proteins , 2005, Journal of dental research.
[23] O. Baba,et al. Post-translational modifications of sibling proteins and their roles in osteogenesis and dentinogenesis. , 2004, Critical reviews in oral biology and medicine : an official publication of the American Association of Oral Biologists.
[24] S. Stock,et al. Pathological Calcification in Juvenile Dermatomyositis (JDM): MicroCT and Synchrotron X-Ray Diffraction Reveal Hydroxyapatite with Varied Microstructures , 2004, Connective tissue research.
[25] G. Garlet,et al. Dentin Sialoprotein and Phosphoprotein Induce Neutrophil Recruitment: A Mechanism Dependent on IL-1β, TNF-α, and CXC Chemokines , 2004, Calcified Tissue International.
[26] G. Uzel,et al. Cytokines in juvenile dermatomyositis pathophysiology: potential and challenge , 2003, Current opinion in rheumatology.
[27] M. Cho,et al. Dual Functional Roles of Dentin Matrix Protein 1 , 2003, The Journal of Biological Chemistry.
[28] R. Bode,et al. Disease activity score for children with juvenile dermatomyositis: reliability and validity evidence. , 2003, Arthritis and rheumatism.
[29] J. Stock,et al. Targeted Disruption of the Osteoblast/Osteocyte Factor 45 Gene (OF45) Results in Increased Bone Formation and Bone Mass* , 2003, The Journal of Biological Chemistry.
[30] N. Fedarko,et al. Six Genes Expressed in Bones and Teeth Encode the Current Members of the SIBLING Family of Proteins , 2003, Connective tissue research.
[31] H. Cantor,et al. Phosphorylation‐dependent interaction of osteopontin with its receptors regulates macrophage migration and activation , 2002, Journal of leukocyte biology.
[32] L. Pachman,et al. Expression of TNFα by muscle fibers in biopsies from children with untreated juvenile dermatomyositis : Association with the TNFα-308A allele , 2001 .
[33] S. Shi,et al. Antigen Retrieval Technique for Immunohistochemistry: Principles, Protocols, and Further Development , 2001 .
[34] R. Brekken,et al. SPARC, a matricellular protein: at the crossroads of cell-matrix communication. , 2001, Matrix biology : journal of the International Society for Matrix Biology.
[35] P. Ducy. CBFA1: A molecular switch in osteoblast biology , 2000, Developmental dynamics : an official publication of the American Association of Anatomists.
[36] A. Boskey,et al. Fourier transform infrared microspectroscopic analysis of bones of osteocalcin-deficient mice provides insight into the function of osteocalcin. , 1998, Bone.
[37] R. Behringer,et al. Spontaneous calcification of arteries and cartilage in mice lacking matrix GLA protein , 1997, Nature.
[38] Allan Bradley,et al. Increased bone formation in osteocalcin-deficient mice , 1996, Nature.
[39] A. Peña,et al. Differences in the intrinsic capacity of peripheral blood mononuclear cells to produce tumor necrosis factor alpha and beta in patients with inflammatory bowel disease and healthy controls. , 1995, Scandinavian journal of gastroenterology.
[40] A. Gown,et al. Distribution of SPARC in normal and neoplastic human tissue. , 1995, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.
[41] D. Denhardt,et al. Osteopontin: a protein with diverse functions , 1993, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[42] O. Olerup,et al. HLA-DR typing by PCR amplification with sequence-specific primers (PCR-SSP) in 2 hours: an alternative to serological DR typing in clinical practice including donor-recipient matching in cadaveric transplantation. , 1992, Tissue antigens.
[43] J. Lian,et al. Gamma-carboxyglutamate excretion and calcinosis in juvenile dermatomyositis. , 1982, Arthritis and rheumatism.
[44] J. Lian,et al. The presence of γ-carboxyglutamic acid in the proteins associated with ectopic calcification , 1976 .
[45] A. Bohan,et al. Polymyositis and dermatomyositis (second of two parts). , 1975 .
[46] M. Victor,et al. DERMATOMYOSITIS (SYSTEMIC ANGIOPATHY) OF CHILDHOOD , 1966, Medicine.