Orthodontic cell stress modifies proinflammatory cytokine expression in human PDL cells and induces immunomodulatory effects via TLR-4 signaling in vitro
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A. Jäger | I. Knaup | S. Lossdörfer | R. Craveiro | M. Wolf | J. Marciniak | A. Bastian
[1] P. Proff,et al. Valid gene expression normalization by RT-qPCR in studies on hPDL fibroblasts with focus on orthodontic tooth movement and periodontitis , 2017, Scientific Reports.
[2] P. Proff,et al. Regular nicotine intake increased tooth movement velocity, osteoclastogenesis and orthodontically induced dental root resorptions in a rat model , 2017, International Journal of Oral Science.
[3] P. Proff,et al. Interactive effects of periodontitis and orthodontic tooth movement on dental root resorption, tooth movement velocity and alveolar bone loss in a rat model. , 2017, Annals of anatomy = Anatomischer Anzeiger : official organ of the Anatomische Gesellschaft.
[4] J. Jacobs,et al. Cobalt Alloy Implant Debris Induces Inflammation and Bone Loss Primarily through Danger Signaling, Not TLR4 Activation: Implications for DAMP-ening Implant Related Inflammation , 2016, PloS one.
[5] J. Deschner,et al. Short-term heat pre-treatment modulates the release of HMGB1 and pro-inflammatory cytokines in hPDL cells following mechanical loading and affects monocyte behavior , 2016, Clinical Oral Investigations.
[6] S. Murakami,et al. PLAP-1/Asporin Regulates TLR2- and TLR4-induced Inflammatory Responses , 2015, Journal of dental research.
[7] A. Jäger,et al. Regulation of high mobility group box protein 1 expression following mechanical loading by orthodontic forces in vitro and in vivo. , 2014, European journal of orthodontics.
[8] J. Deschner,et al. Anabolic Properties of High Mobility Group Box Protein-1 in Human Periodontal Ligament Cells In Vitro , 2014, Mediators of inflammation.
[9] A. Jäger,et al. High-mobility group box protein-1 released by human-periodontal ligament cells modulates macrophage migration and activity in vitro , 2014, Innate immunity.
[10] J. Cirelli,et al. Orthodontic force increases interleukin-1β and tumor necrosis factor-α expression and alveolar bone loss in periodontitis. , 2013, Journal of periodontology.
[11] E-C Kim,et al. Mechanical stress‐activated immune response genes via Sirtuin 1 expression in human periodontal ligament cells , 2012, Clinical and experimental immunology.
[12] J. Deschner,et al. Regulatory role of periodontal ligament fibroblasts for innate immune cell function and differentiation , 2012, Innate immunity.
[13] J. Deschner,et al. Human β‐defensins differently affect proliferation, differentiation, and mineralization of osteoblast‐like MG63 cells , 2012, Journal of cellular physiology.
[14] S. Kook,et al. Involvement of JNK-AP-1 and ERK-NF-κB signaling in tension-stimulated expression of type I collagen and MMP-1 in human periodontal ligament fibroblasts. , 2011, Journal of applied physiology.
[15] W. Götz,et al. PTH(1-34)-induced changes in RANKL and OPG expression by human PDL cells modify osteoclast biology in a co-culture model with RAW 264.7 cells , 2011, Clinical Oral Investigations.
[16] Xuedong Zhou,et al. Expression of TRAF6 and pro-inflammatory cytokines through activation of TLR2, TLR4, NOD1, and NOD2 in human periodontal ligament fibroblasts. , 2011, Archives of oral biology.
[17] Hong Wang,et al. TLR-signaling Networks , 2011, Journal of dental research.
[18] H. Kajiya,et al. Hyperocclusion Stimulates Osteoclastogenesis via CCL2 Expression , 2011, Journal of dental research.
[19] Grace Y Chen,et al. Sterile inflammation: sensing and reacting to damage , 2010, Nature Reviews Immunology.
[20] Suk-Keun Lee,et al. SIRT1 modulates high‐mobility group box 1‐induced osteoclastogenic cytokines in human periodontal ligament cells , 2010, Journal of cellular biochemistry.
[21] S. Frede,et al. Acute Hypoxia Induces HIF-Independent Monocyte Adhesion to Endothelial Cells through Increased Intercellular Adhesion Molecule-1 Expression: The Role of Hypoxic Inhibition of Prolyl Hydroxylase Activity for the Induction of NF-κB , 2010, The Journal of Immunology.
[22] S. Akira,et al. Pattern Recognition Receptors and Inflammation , 2010, Cell.
[23] A. Kuijpers-Jagtman,et al. Matrix metalloproteinases and tissue inhibitors of metalloproteinases in gingival crevicular fluid during orthodontic tooth movement. , 2009, European journal of orthodontics.
[24] K. Beggs,et al. Cultured human periodontal ligament cells constitutively express multiple osteotropic cytokines and growth factors, several of which are responsive to mechanical deformation. , 2008, Journal of periodontal research.
[25] Ying Sun,et al. Toll-like receptor 4 signaling plays a role in triggering periodontal infection. , 2008, FEMS immunology and medical microbiology.
[26] A. Vissink,et al. Cytokines in crevicular fluid and orthodontic tooth movement. , 2008, European journal of oral sciences.
[27] Katherine A. Fitzgerald,et al. Tyrosine Phosphorylation of MyD88 Adapter-like (Mal) Is Critical for Signal Transduction and Blocked in Endotoxin Tolerance* , 2008, Journal of Biological Chemistry.
[28] Xiangrui Wang,et al. Role of Rel A and IkappaB of nuclear factor κB in the release of interleukin‐8 by cyclic mechanical strain in human alveolar type II epithelial cells A549 , 2007, Respirology.
[29] R. Medzhitov. Recognition of microorganisms and activation of the immune response , 2007, Nature.
[30] Zhihe Zhao,et al. NF‐κB responds to mechanical strains in osteoblast‐like cells, and lighter strains create an NF‐κB response more readily , 2007 .
[31] T. Goto,et al. Intermittent Force Induces High RANKL Expression in Human Periodontal Ligament Cells , 2007, Journal of dental research.
[32] Lutz Claes,et al. Signal transduction pathways involved in mechanotransduction in bone cells. , 2006, Biochemical and biophysical research communications.
[33] Ashutosh Kumar Singh,et al. Gingival epithelial cells heterozygous for Toll-like receptor 4 polymorphisms Asp299Gly and Thr399Ile are hypo-responsive to Porphyromonas gingivalis , 2006, Genes and Immunity.
[34] J. Tanaka,et al. DNA microarray analysis of human gingival fibroblasts from healthy and inflammatory gingival tissues. , 2003, Biochemical and biophysical research communications.
[35] J. Hatakeyama,et al. Contrasting responses of human gingival and periodontal ligament fibroblasts to bacterial cell-surface components through the CD14/Toll-like receptor system. , 2003, Oral microbiology and immunology.
[36] Siamon Gordon,et al. Pattern Recognition Receptors Doubling Up for the Innate Immune Response , 2002, Cell.
[37] D. Golenbock,et al. Dysregulation of LPS-Induced Toll-Like Receptor 4-MyD88 Complex Formation and IL-1 Receptor-Associated Kinase 1 Activation in Endotoxin-Tolerant Cells1 , 2002, The Journal of Immunology.
[38] K. Naruse,et al. Uni‐axial cyclic stretch induces the activation of transcription factor nuclear factor κB in human fibroblast cells , 2002, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[39] Hideo Mitani,et al. Periodontal Ligament Cells Under Mechanical Stress Induce Osteoclastogenesis by Receptor Activator of Nuclear Factor κB Ligand Up‐Regulation via Prostaglandin E2 Synthesis , 2002, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[40] S. Akira,et al. Cutting edge: Toll-like receptor 4 (TLR4)-deficient mice are hyporesponsive to lipopolysaccharide: evidence for TLR4 as the Lps gene product. , 1999, Journal of immunology.
[41] R. Davis,et al. Transcription factor AP-1 regulation by mitogen-activated protein kinase signal transduction pathways , 1996, Journal of Molecular Medicine.
[42] R. Radlanski,et al. Demonstration of cells of the mononuclear phagocyte lineage in the periodontium following experimental tooth movement in the rat , 1993, Histochemistry.
[43] C. Bourauel,et al. Regulation of visfatin by microbial and biomechanical signals in PDL cells , 2013, Clinical Oral Investigations.
[44] P. Römer,et al. Cellular response to orthodontically-induced short-term hypoxia in dental pulp cells , 2013, Cell and Tissue Research.
[45] S. Biswas,et al. Myeloid differentiation factor 88-independent Toll-like receptor pathway: Sustaining inflammation or promoting tolerance? , 2007, The international journal of biochemistry & cell biology.
[46] Zhihe Zhao,et al. NF-kappaB responds to mechanical strains in osteoblast-like cells, and lighter strains create an NF-kappaB response more readily. , 2007, Cell biology international.