Metanephric organ culture.
暂无分享,去创建一个
[1] B. Magenheimer,et al. A mutation affecting polycystin-1 mediated heterotrimeric G-protein signaling causes PKD , 2018, Human molecular genetics.
[2] E. Baloglu,et al. Anticystogenic activity of a small molecule PAK4 inhibitor may be a novel treatment for autosomal dominant polycystic kidney disease. , 2017, Kidney international.
[3] B. Magenheimer,et al. Phosphodiesterase Isoform Regulation of Cell Proliferation and Fluid Secretion in Autosomal Dominant Polycystic Kidney Disease. , 2016, Journal of the American Society of Nephrology : JASN.
[4] B. Magenheimer,et al. Ouabain Regulates CFTR-Mediated Anion Secretion and Na,K-ATPase Transport in ADPKD Cells , 2015, The Journal of Membrane Biology.
[5] D. Beier,et al. Downregulating hedgehog signaling reduces renal cystogenic potential of mouse models. , 2014, Journal of the American Society of Nephrology : JASN.
[6] G. Schley,et al. Anoctamin 1 induces calcium-activated chloride secretion and proliferation of renal cyst-forming epithelial cells. , 2014, Kidney international.
[7] G. Schley,et al. Hypoxia-inducible factor-1α causes renal cyst expansion through calcium-activated chloride secretion. , 2014, Journal of the American Society of Nephrology : JASN.
[8] G. Germino,et al. Impaired glomerulogenesis and endothelial cell migration in Pkd1-deficient renal organ cultures , 2014, Biochemical and biophysical research communications.
[9] B. Magenheimer,et al. Endogenous concentrations of ouabain act as a cofactor to stimulate fluid secretion and cyst growth of in vitro ADPKD models via cAMP and EGFR-Src-MEK pathways. , 2012, American journal of physiology. Renal physiology.
[10] Baoxue Yang,et al. Ginkgolide B inhibits renal cyst development in in vitro and in vivo cyst models , 2012, American journal of physiology. Renal physiology.
[11] S. Somlo,et al. Activating AMP-activated protein kinase (AMPK) slows renal cystogenesis , 2011, Proceedings of the National Academy of Sciences.
[12] Jing Zhou,et al. Failure to ubiquitinate c-Met leads to hyperactivation of mTOR signaling in a mouse model of autosomal dominant polycystic kidney disease. , 2010, The Journal of clinical investigation.
[13] A. Woolf,et al. Corticosteroid-induced kidney dysmorphogenesis is associated with deregulated expression of known cystogenic molecules, as well as Indian hedgehog. , 2010, American journal of physiology. Renal physiology.
[14] B. Magenheimer,et al. A tumor necrosis factor-α–mediated pathway promoting autosomal dominant polycystic kidney disease , 2008, Nature Medicine.
[15] A S Verkman,et al. Small-molecule CFTR inhibitors slow cyst growth in polycystic kidney disease. , 2008, Journal of the American Society of Nephrology : JASN.
[16] B. Magenheimer,et al. Early embryonic renal tubules of wild-type and polycystic kidney disease kidneys respond to cAMP stimulation with cystic fibrosis transmembrane conductance regulator/Na(+),K(+),2Cl(-) Co-transporter-dependent cystic dilation. , 2006, Journal of the American Society of Nephrology : JASN.
[17] S. Nigam,et al. Organic anion and cation transporter expression and function during embryonic kidney development and in organ culture models. , 2006, Kidney international.
[18] P. Wilson,et al. Disruption of polycystin-1 function interferes with branching morphogenesis of the ureteric bud in developing mouse kidneys. , 2005, Developmental biology.
[19] E. Avner,et al. Sodium-potassium ATPase activity mediates cyst formation in metanephric organ culture. , 1985, Kidney international.
[20] E. Avner,et al. Cyst formation in metanephric organ culture induced by cis-dichlorodiammineplatinum (II) , 1983, Experientia.
[21] E. Avner,et al. Metanephric development in serum-free organ culture , 1982, In Vitro - Plant.
[22] V. Jha,et al. Calcium ameliorates renal cyst growth in metanephric organ culture: a morphological study. , 2012, Journal of environmental pathology, toxicology and oncology : official organ of the International Society for Environmental Toxicology and Cancer.
[23] Laurie A. Smith,et al. Pkd1 and Nek8 mutations affect cell-cell adhesion and cilia in cysts formed in kidney organ cultures. , 2008, American journal of physiology. Renal physiology.
[24] J. Adelsberg. Peptides from the PKD repeats of polycystin, the PKD1 gene product, modulate pattern formation in the developing kidney. , 1999 .