Deficiency in TIMP-3 increases cardiac rupture and mortality post-myocardial infarction via EGFR signaling: beneficial effects of cetuximab
暂无分享,去创建一个
Ming Lei | Lamis Hammoud | Xiangru Lu | Q. Feng | Lamis Hammoud | M. Lei | Qingping Feng | Xiangru Lu | Q. Feng
[1] Stefan Frantz,et al. Transforming growth factor beta inhibition increases mortality and left ventricular dilatation after myocardial infarction , 2008, Basic Research in Cardiology.
[2] J. Fischer,et al. Reduced MMP-2 activity contributes to cardiac fibrosis in experimental diabetic cardiomyopathy , 2008, Basic Research in Cardiology.
[3] H. Kodama,et al. Effects of epidermal growth factor on osteoblastic cellsin vitro , 1983, Calcified Tissue International.
[4] Douglas L. Jones,et al. Mortality After Myocardial Infarction in Mice , 2022 .
[5] A. Leask. TGFβ, cardiac fibroblasts, and the fibrotic response , 2007 .
[6] C. Long,et al. The cardiac fibroblast: therapeutic target in myocardial remodeling and failure. , 2005, Annual review of pharmacology and toxicology.
[7] F. Harrell,et al. Cardiac rupture, mortality and the timing of thrombolytic therapy: a meta-analysis. , 1990, Journal of the American College of Cardiology.
[8] Vijay G Divakaran,et al. Transforming growth factor-β receptor antagonism attenuates myocardial fibrosis in mice with cardiac-restricted overexpression of tumor necrosis factor , 2007, Basic Research in Cardiology.
[9] B. Strauss,et al. Matrix remodeling in experimental and human heart failure: a possible regulatory role for TIMP-3. , 2003, American journal of physiology. Heart and circulatory physiology.
[10] A. Maseri,et al. Age-Related Increase in Mortality among Patients with First Myocardial Infarctions Treated with Thrombolysis , 1993 .
[11] K. Brew,et al. Tissue inhibitors of metalloproteinases: evolution, structure and function. , 2000, Biochimica et biophysica acta.
[12] J. Simons,et al. Direct comparison of GAPDH, beta-actin, cyclophilin, and 28S rRNA as internal standards for quantifying RNA levels under hypoxia. , 1999, Biochemical and biophysical research communications.
[13] A. Maseri,et al. Age-related increase in mortality among patients with first myocardial infarctions treated with thrombolysis. The Investigators of the Gruppo Italiano per lo Studio della Sopravvivenza nell'Infarto Miocardico (GISSI-2). , 1993, The New England journal of medicine.
[14] Andrew Leask,et al. TGF‐β signaling and the fibrotic response , 2004, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[15] A. Takeshita,et al. Targeted deletion of MMP-2 attenuates early LV rupture and late remodeling after experimental myocardial infarction. , 2003, American journal of physiology. Heart and circulatory physiology.
[16] G. Cloutier,et al. Anti-inflammatory effect of oxytocin in rat myocardial infarction , 2010, Basic Research in Cardiology.
[17] T. Mak,et al. Spontaneous air space enlargement in the lungs of mice lacking tissue inhibitor of metalloproteinases-3 (TIMP-3). , 2001, The Journal of clinical investigation.
[18] A. Dart,et al. Mouse model of post-infarct ventricular rupture: time course, strain- and gender-dependency, tensile strength, and histopathology. , 2005, Cardiovascular research.
[19] R. Khokha,et al. TIMP-3 Deficiency Leads to Dilated Cardiomyopathy , 2004, Circulation.
[20] H. Kodama,et al. Effects of epidermal growth factor on osteoblastic cells in vitro. , 1983, Calcified tissue international.
[21] J. Skurnick,et al. Cardiac Rupture in Acute Myocardial Infarction: A Reassessment , 2002, The American journal of forensic medicine and pathology.
[22] T. Peng,et al. Pivotal Role of gp91phox-Containing NADH Oxidase in Lipopolysaccharide-Induced Tumor Necrosis Factor-α Expression and Myocardial Depression , 2005, Circulation.
[23] U. Walter,et al. Increased Spreading, Rac/p21-activated Kinase (PAK) Activity, and Compromised Cell Motility in Cells Deficient in Vasodilator-stimulated Phosphoprotein (VASP)* , 2002, The Journal of Biological Chemistry.
[24] S. Kurata,et al. Epidermal growth factor inhibits transcription of type I collagen genes and production of type I collagen in cultured human skin fibroblasts in the presence and absence of L-ascorbic acid 2-phosphate, a long-acting vitamin C derivative. , 1991, The Journal of biological chemistry.
[25] M. Daemen,et al. Collagen remodeling after myocardial infarction in the rat heart. , 1995, The American journal of pathology.
[26] J. Frampton. Cetuximab , 2010, Drugs.
[27] R. M. Rubison,et al. A composite view of cardiac rupture in the United States National Registry of Myocardial Infarction. , 1996, Journal of the American College of Cardiology.
[28] Vijay G Divakaran,et al. Transforming growth factor-beta receptor antagonism attenuates myocardial fibrosis in mice with cardiac-restricted overexpression of tumor necrosis factor. , 2008, Basic research in cardiology.
[29] Stephen C. Jones,et al. Regulation of collagen synthesis by inhibitory Smad7 in cardiac myofibroblasts. , 2007, American journal of physiology. Heart and circulatory physiology.
[30] J. F. Woessner,et al. The determination of hydroxyproline in tissue and protein samples containing small proportions of this imino acid. , 1961, Archives of biochemistry and biophysics.
[31] J. Mlczoch,et al. Frequency of left ventricular free wall rupture complicating acute myocardial infarction since the advent of thrombolysis. , 1994, The American journal of cardiology.
[32] Hai Tian,et al. TIMP-3 deficiency accelerates cardiac remodeling after myocardial infarction. , 2007, Journal of molecular and cellular cardiology.
[33] N. Dhalla,et al. Cardiac Remodeling and Failure , 2003, Progress in Experimental Cardiology.
[34] S. Verma,et al. Cardiac remodeling and failure From molecules to man (Part II). , 2005, Cardiovascular pathology : the official journal of the Society for Cardiovascular Pathology.
[35] F. Gao,et al. Heterogeneous effects of tissue inhibitors of matrix metalloproteinases on cardiac fibroblasts. , 2005, American journal of physiology. Heart and circulatory physiology.
[36] T. Peng,et al. Development of Heart Failure and Congenital Septal Defects in Mice Lacking Endothelial Nitric Oxide Synthase , 2002, Circulation.
[37] J. Cosgaya,et al. Ras- and Raf-mediated regulation of transforming growth factor beta 1 gene expression by ligands of tyrosine kinase receptors in PC12 cells. , 1996, Oncogene.
[38] S. Hawkes,et al. Role of the 21-kDa protein TIMP-3 in oncogenic transformation of cultured chicken embryo fibroblasts. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[39] J. Zarzyńska,et al. Effects of hormones and growth factors on TGF-β1 expression in bovine mammary epithelial cells , 2005, Journal of Dairy Research.
[40] V. A. Flørenes,et al. Different Expression and Clinical Role of S100A4 in Serous Ovarian Carcinoma at Different Anatomic Sites , 2009, Tumor Biology.
[41] S. Hunyor,et al. Regional cardiac dysfunction is associated with specific alterations in inflammatory cytokines and matrix metalloproteinases after acute myocardial infarction in sheep , 2006, Basic Research in Cardiology.
[42] R. Becker,et al. Cardiac Rupture After Myocardial Infarction: New Insights From Murine Models , 2009, Cardiology in review.
[43] Douglas L. Jones,et al. Role of tumor necrosis factor-α in myocardial dysfunction and apoptosis during hindlimb ischemia and reperfusion , 2006 .
[44] Douglas L. Jones,et al. Role of tumor necrosis factor-alpha in myocardial dysfunction and apoptosis during hindlimb ischemia and reperfusion. , 2006, Critical care medicine.
[45] J. J. Creely,et al. Effects of epidermal growth factor on collagen synthesis by an epithelioid cell line derived from normal rat kidney. , 1990, The American journal of pathology.
[46] Arnold E. Postlethwaite,et al. Infarct scar as living tissue , 2002, Basic Research in Cardiology.
[47] Xiangru Lu,et al. Tissue inhibitor of metalloproteinase-3 inhibits neonatal mouse cardiomyocyte proliferation via EGFR/JNK/SP-1 signaling. , 2009, American journal of physiology. Cell physiology.
[48] J. Fischer,et al. Transcriptional and posttranscriptional regulators of biglycan in cardiac fibroblasts , 2009, Basic Research in Cardiology.
[49] T. Peng,et al. Endothelial Nitric-oxide Synthase Enhances Lipopolysaccharide-stimulated Tumor Necrosis Factor-α Expression via cAMP-mediated p38 MAPK Pathway in Cardiomyocytes* , 2003, The Journal of Biological Chemistry.
[50] J. García-Foncillas,et al. Improving disease control in advanced colorectal cancer: Panitumumab and cetuximab. , 2010, Critical reviews in oncology/hematology.