Endoplasmic Reticulum Stress Signaling in Pancreatic β-Cells
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[1] H. P. Wagener,et al. DIABETES MELLITUS AND SIMPLE OPTIC ATROPHY AMONG SIBLINGS: REPORT OF FOUR CASES , 1938 .
[2] M. Rallison,et al. Infancy-onset diabetes mellitus and multiple epiphyseal dysplasia. , 1972, The Journal of pediatrics.
[3] A. Karasik,et al. Genetically Programmed Selective Islet β-Cell Loss in Diabetic Subjects With Wolfram's Syndrome , 1989, Diabetes Care.
[4] D. Ron,et al. CHOP, a novel developmentally regulated nuclear protein that dimerizes with transcription factors C/EBP and LAP and functions as a dominant-negative inhibitor of gene transcription. , 1992, Genes & development.
[5] J. Horton,et al. Wolfram syndrome , 1992, Neurology.
[6] P. Cohen,et al. Inhibition of glycogen synthase kinase-3 by insulin mediated by protein kinase B , 1995, Nature.
[7] A. Rosen,et al. DNA-dependent protein kinase is one of a subset of autoantigens specifically cleaved early during apoptosis , 1995, The Journal of experimental medicine.
[8] M. Matsumoto,et al. Purification and Characterization of a Novel Stress Protein, the 150-kDa Oxygen-regulated Protein (ORP150), from Cultured Rat Astrocytes and Its Expression in Ischemic Mouse Brain (*) , 1996, The Journal of Biological Chemistry.
[9] M. Matsumoto,et al. Cloning and expression of cDNA encoding the human 150 kDa oxygen-regulated protein, ORP150. , 1997, Biochemical and biophysical research communications.
[10] M. Muda,et al. Bcl-2 Undergoes Phosphorylation by c-Jun N-terminal Kinase/Stress-activated Protein Kinases in the Presence of the Constitutively Active GTP-binding Protein Rac1* , 1997, The Journal of Biological Chemistry.
[11] Masato Yoshioka,et al. A Novel Locus, Mody4, Distal to D7Mit189 on Chromosome 7 Determines Early-Onset NIDDM in Nonobese C57BL/6 (Akita) Mutant Mice , 1997, Diabetes.
[12] T. Barrett,et al. Wolfram (DIDMOAD) syndrome. , 1997, Journal of medical genetics.
[13] Identification and Characterization of Pancreatic Eukaryotic Initiation Factor 2 α-Subunit Kinase, PEK, Involved in Translational Control , 1998, Molecular and Cellular Biology.
[14] R. Kaufman,et al. A stress response pathway from the endoplasmic reticulum to the nucleus requires a novel bifunctional protein kinase/endoribonuclease (Ire1p) in mammalian cells. , 1998, Genes & development.
[15] T. Meitinger,et al. Diabetes insipidus, diabetes mellitus, optic atrophy and deafness (DIDMOAD) caused by mutations in a novel gene (wolframin) coding for a predicted transmembrane protein. , 1998, Human molecular genetics.
[16] A. Koizumi,et al. Mapping of murine diabetogenic gene mody on chromosome 7 at D7Mit258 and its involvement in pancreatic islet and beta cell development during the perinatal period. , 1998, The Journal of clinical investigation.
[17] Xiaozhong Wang,et al. CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum. , 1998, Genes & development.
[18] K. Miyazono,et al. ASK1 is essential for JNK/SAPK activation by TRAF2. , 1998, Molecular cell.
[19] P. Behn,et al. A gene encoding a transmembrane protein is mutated in patients with diabetes mellitus and optic atrophy (Wolfram syndrome) , 1998, Nature Genetics.
[20] Masahiko Kuroda,et al. Cloning of mammalian Ire1 reveals diversity in the ER stress responses , 1998, The EMBO journal.
[21] Kazuhito Yamamoto,et al. BCL-2 Is Phosphorylated and Inactivated by an ASK1/Jun N-Terminal Protein Kinase Pathway Normally Activated at G2/M , 1999, Molecular and Cellular Biology.
[22] Danhong Lu,et al. A mutation in the insulin 2 gene induces diabetes with severe pancreatic beta-cell dysfunction in the Mody mouse. , 1999, The Journal of clinical investigation.
[23] K. Mori,et al. Mammalian transcription factor ATF6 is synthesized as a transmembrane protein and activated by proteolysis in response to endoplasmic reticulum stress. , 1999, Molecular biology of the cell.
[24] Zhengbin Yao,et al. Mediation of TNF receptor-associated factor effector functions by apoptosis signal-regulating kinase-1 (ASK1) , 1999, Oncogene.
[25] D. Ron,et al. Protein translation and folding are coupled by an endoplasmic-reticulum-resident kinase , 1999, Nature.
[26] D. Ron,et al. Perk is essential for translational regulation and cell survival during the unfolded protein response. , 2000, Molecular cell.
[27] F. Urano,et al. Coupling of stress in the ER to activation of JNK protein kinases by transmembrane protein kinase IRE1. , 2000, Science.
[28] Cheryl Brantley-Finley,et al. Vinblastine-induced Phosphorylation of Bcl-2 and Bcl-XL Is Mediated by JNK and Occurs in Parallel with Inactivation of the Raf-1/MEK/ERK Cascade* , 2000, The Journal of Biological Chemistry.
[29] K. Mori. Tripartite Management of Unfolded Proteins in the Endoplasmic Reticulum , 2000, Cell.
[30] F. Urano,et al. IRE1 and efferent signaling from the endoplasmic reticulum. , 2000, Journal of cell science.
[31] S. Orkin,et al. An essential role in liver development for transcription factor XBP-1. , 2000, Genes & development.
[32] M. Schapira,et al. Regulated translation initiation controls stress-induced gene expression in mammalian cells. , 2000, Molecular cell.
[33] G. Lathrop,et al. EIF2AK3, encoding translation initiation factor 2-α kinase 3, is mutated in patients with Wolcott-Rallison syndrome , 2000, Nature Genetics.
[34] Zhi-Min Yuan,et al. Translocation of SAPK/JNK to Mitochondria and Interaction with Bcl-xL in Response to DNA Damage* , 2000, The Journal of Biological Chemistry.
[35] Junying Yuan,et al. Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-β , 2000, Nature.
[36] Christophe Benoist,et al. β-Cell death during progression to diabetes , 2001, Nature.
[37] Neal N. Iwakoshi,et al. Plasma cell differentiation requires the transcription factor XBP-1 , 2001, Nature.
[38] D. Ron,et al. Diabetes mellitus and exocrine pancreatic dysfunction in perk-/- mice reveals a role for translational control in secretory cell survival. , 2001, Molecular cell.
[39] Masataka Mori,et al. Nitric oxide-induced apoptosis in pancreatic β cells is mediated by the endoplasmic reticulum stress pathway , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[40] D. Schorderet,et al. Cell-Permeable Peptide Inhibitors of JNK: Novel Blockers of β-Cell Death , 2001 .
[41] M. Tohyama,et al. Activation of Caspase-12, an Endoplastic Reticulum (ER) Resident Caspase, through Tumor Necrosis Factor Receptor-associated Factor 2-dependent Mechanism in Response to the ER Stress* , 2001, The Journal of Biological Chemistry.
[42] E McEwen,et al. Translational control is required for the unfolded protein response and in vivo glucose homeostasis. , 2001, Molecular cell.
[43] Judy H. Cho,et al. Increased sensitivity to dextran sodium sulfate colitis in IRE1beta-deficient mice. , 2001, The Journal of clinical investigation.
[44] M. Kruhøffer,et al. A Comprehensive Analysis of Cytokine-induced and Nuclear Factor-κB-dependent Genes in Primary Rat Pancreatic β-Cells* , 2001, The Journal of Biological Chemistry.
[45] Xiaohua Shen,et al. The unfolded protein response in nutrient sensing and differentiation , 2002, Nature Reviews Molecular Cell Biology.
[46] Masataka Mori,et al. Targeted disruption of the Chop gene delays endoplasmic reticulum stress-mediated diabetes. , 2002, The Journal of clinical investigation.
[47] Kiyoshi Inoue,et al. ASK1 is essential for endoplasmic reticulum stress-induced neuronal cell death triggered by expanded polyglutamine repeats. , 2002, Genes & development.
[48] M. Katze,et al. Control of PERK eIF2α kinase activity by the endoplasmic reticulum stress-induced molecular chaperone P58IPK , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[49] Stevan R. Hubbard,et al. IRE1 couples endoplasmic reticulum load to secretory capacity by processing the XBP-1 mRNA , 2002, Nature.
[50] F. Urano,et al. Transcriptional and translational control in the Mammalian unfolded protein response. , 2002, Annual review of cell and developmental biology.
[51] Richard A. Flavell,et al. The Bax Subfamily of Bcl2-Related Proteins Is Essential for Apoptotic Signal Transduction by c-Jun NH2-Terminal Kinase , 2002, Molecular and Cellular Biology.
[52] R. Paules,et al. An integrated stress response regulates amino acid metabolism and resistance to oxidative stress. , 2003, Molecular cell.
[53] K. Rajewsky,et al. Plasma cell differentiation and the unfolded protein response intersect at the transcription factor XBP-1 , 2003, Nature Immunology.
[54] L. Glimcher,et al. The X‐box binding protein‐1 transcription factor is required for plasma cell differentiation and the unfolded protein response , 2003, Immunological reviews.
[55] Hiderou Yoshida,et al. A time-dependent phase shift in the mammalian unfolded protein response. , 2003, Developmental cell.
[56] Robert A. Rizza,et al. β-Cell Deficit and Increased β-Cell Apoptosis in Humans With Type 2 Diabetes , 2003, Diabetes.
[57] L. Glimcher,et al. XBP-1 Regulates a Subset of Endoplasmic Reticulum Resident Chaperone Genes in the Unfolded Protein Response , 2003, Molecular and Cellular Biology.
[58] M. Permutt,et al. Wolframin Expression Induces Novel Ion Channel Activity in Endoplasmic Reticulum Membranes and Increases Intracellular Calcium* , 2003, Journal of Biological Chemistry.
[59] N. Welsh,et al. The harmony of the spheres: inducible nitric oxide synthase and related genes in pancreatic beta cells , 1996, Diabetologia.
[60] M. Cnop,et al. Free Fatty Acids and Cytokines Induce Pancreatic β-Cell Apoptosis by Different Mechanisms: Role of Nuclear Factor-κB and Endoplasmic Reticulum Stress , 2004 .
[61] Marc Prentki,et al. Role for Activating Transcription Factor 3 in Stress-Induced β-Cell Apoptosis , 2004, Molecular and Cellular Biology.
[62] K. Mori,et al. XBP1: a link between the unfolded protein response, lipid biosynthesis, and biogenesis of the endoplasmic reticulum. , 2004, The Journal of cell biology.
[63] L. Glimcher,et al. Endoplasmic Reticulum Stress Links Obesity, Insulin Action, and Type 2 Diabetes , 2004, Science.
[64] H. Katagiri,et al. Disruption of the WFS1 gene in mice causes progressive beta-cell loss and impaired stimulus-secretion coupling in insulin secretion. , 2004, Human molecular genetics.
[65] L. Staudt,et al. XBP1, downstream of Blimp-1, expands the secretory apparatus and other organelles, and increases protein synthesis in plasma cell differentiation. , 2004, Immunity.
[66] J. Weissman,et al. Oxidative protein folding in eukaryotes , 2004, The Journal of cell biology.
[67] Kathryn L. Lipson,et al. High ER stress in beta-cells stimulates intracellular degradation of misfolded insulin. , 2004, Biochemical and biophysical research communications.
[68] M. Permutt,et al. Endoplasmic reticulum stress-induced apoptosis is partly mediated by reduced insulin signaling through phosphatidylinositol 3-kinase/Akt and increased glycogen synthase kinase-3beta in mouse insulinoma cells. , 2005, Diabetes.
[69] Kathryn L. Lipson,et al. WFS1 Is a Novel Component of the Unfolded Protein Response and Maintains Homeostasis of the Endoplasmic Reticulum in Pancreatic β-Cells* , 2005, Journal of Biological Chemistry.
[70] A. Wanaka,et al. OASIS, a CREB/ATF-family member, modulates UPR signalling in astrocytes , 2005, Nature Cell Biology.
[71] F. Ortis,et al. Cytokines downregulate the sarcoendoplasmic reticulum pump Ca2+ ATPase 2b and deplete endoplasmic reticulum Ca2+, leading to induction of endoplasmic reticulum stress in pancreatic beta-cells. , 2005, Diabetes.
[72] Junying Yuan,et al. A Selective Inhibitor of eIF2α Dephosphorylation Protects Cells from ER Stress , 2005, Science.
[73] M. Katze,et al. Pancreatic (cid:1) -Cell Failure and Diabetes in Mice With a Deletion Mutation of the Endoplasmic Reticulum Molecular Chaperone Gene P58 IPK , 2022 .
[74] R. Kaufman,et al. The mammalian unfolded protein response. , 2003, Annual review of biochemistry.
[75] D. Scheuner,et al. Control of mRNA translation preserves endoplasmic reticulum function in beta cells and maintains glucose homeostasis , 2005, Nature Medicine.
[76] Nils Welsh,et al. Mechanisms of pancreatic beta-cell death in type 1 and type 2 diabetes: many differences, few similarities. , 2005, Diabetes.
[77] H. Kaneto,et al. Involvement of Endoplasmic Reticulum Stress in Insulin Resistance and Diabetes* , 2005, Journal of Biological Chemistry.
[78] Kathryn L. Lipson,et al. Regulation of insulin biosynthesis in pancreatic beta cells by an endoplasmic reticulum-resident protein kinase IRE1. , 2006, Cell metabolism.
[79] B. Yusta,et al. GLP-1 receptor activation improves β cell function and survival following induction of endoplasmic reticulum stress , 2006 .
[80] Barbara C. McGrath,et al. PERK EIF2AK3 control of pancreatic beta cell differentiation and proliferation is required for postnatal glucose homeostasis. , 2006, Cell metabolism.
[81] F. Urano,et al. Endoplasmic reticulum stress-induced apoptosis and auto-immunity in diabetes. , 2006, Current molecular medicine.
[82] Randal J. Kaufman,et al. Endoplasmic Reticulum Stress Activates Cleavage of CREBH to Induce a Systemic Inflammatory Response , 2006, Cell.
[83] A. Volchuk,et al. Chronic palmitate but not oleate exposure induces endoplasmic reticulum stress, which may contribute to INS-1 pancreatic beta-cell apoptosis. , 2006, Endocrinology.
[84] E. Yilmaz,et al. Chemical Chaperones Reduce ER Stress and Restore Glucose Homeostasis in a Mouse Model of Type 2 Diabetes , 2006, Science.
[85] Junying Yuan,et al. Selective Inhibition of Eukaryotic Translation Initiation Factor 2α Dephosphorylation Potentiates Fatty Acid-induced Endoplasmic Reticulum Stress and Causes Pancreatic β-Cell Dysfunction and Apoptosis* , 2006, Journal of Biological Chemistry.