Recent advances in the molecular pathophysiology of atrial fibrillation.
暂无分享,去创建一个
Niels Voigt | Stanley Nattel | Dobromir Dobrev | Reza Wakili | Stefan Kääb | S. Nattel | D. Dobrev | S. Kääb | R. Wakili | N. Voigt
[1] R Fischmeister,et al. Nitric oxide regulates cardiac Ca2+ current. Involvement of cGMP-inhibited and cGMP-stimulated phosphodiesterases through guanylyl cyclase activation. , 1993, The Journal of biological chemistry.
[2] C. Antzelevitch,et al. Atrial fibrillation and Brugada syndrome. , 2008, Journal of the American College of Cardiology.
[3] Y. Shibamoto,et al. The ZFHX3 (ATBF1) transcription factor induces PDGFRB, which activates ATM in the cytoplasm to protect cerebellar neurons from oxidative stress , 2010, Disease Models & Mechanisms.
[4] A. Zima,et al. Endothelin-1–Induced Arrhythmogenic Ca2+ Signaling Is Abolished in Atrial Myocytes of Inositol-1,4,5-Trisphosphate(IP3)–Receptor Type 2–Deficient Mice , 2005, Circulation research.
[5] Eric E. Smith,et al. Variants conferring risk of atrial fibrillation on chromosome 4q25 , 2007, Nature.
[6] Stanley Nattel,et al. Characterization of a hyperpolarization‐activated time‐dependent potassium current in canine cardiomyocytes from pulmonary vein myocardial sleeves and left atrium , 2004, The Journal of physiology.
[7] Delon Wu,et al. Intracellular calcium dynamics and acetylcholine-induced triggered activity in the pulmonary veins of dogs with pacing-induced heart failure. , 2008, Heart rhythm.
[8] Da-Zhi Wang,et al. The Emerging Role of MicroRNAs as a Therapeutic Target for Cardiovascular Disease , 2010, BioDrugs.
[9] M. Chung,et al. Atrial Glutathione Content, Calcium Current, and Contractility* , 2007, Journal of Biological Chemistry.
[10] M. Rienstra,et al. Monogenic atrial fibrillation as pathophysiological paradigms. , 2011, Cardiovascular research.
[11] C. McGann,et al. MRI of the left atrium: predicting clinical outcomes in patients with atrial fibrillation , 2011, Expert review of cardiovascular therapy.
[12] Ning Wang,et al. MicroRNA-328 Contributes to Adverse Electrical Remodeling in Atrial Fibrillation , 2010, Circulation.
[13] G. Breithardt,et al. Atrial Arrhythmias in Long‐QT Syndrome under Daily Life Conditions: A Nested Case Control Study , 2009, Journal of cardiovascular electrophysiology.
[14] Nolwenn Le Meur,et al. Gene expression profile associated with chronic atrial fibrillation and underlying valvular heart disease in man. , 2006, Journal of molecular and cellular cardiology.
[15] Stanley Nattel,et al. Atrial Remodeling and Atrial Fibrillation: Mechanisms and Implications , 2008, Circulation. Arrhythmia and electrophysiology.
[16] Ping Zhang,et al. Inositol-1,4,5-Trisphosphate and Ryanodine-Dependent Ca2+ Signaling in a Chronic Dog Model of Atrial Fibrillation , 2006, Cardiology.
[17] X. Wehrens,et al. Intracellular calcium leak due to FKBP12.6 deficiency in mice facilitates the inducibility of atrial fibrillation. , 2008, Heart rhythm.
[18] J. Auer,et al. Subclinical hyperthyroidism as a risk factor for atrial fibrillation. , 2001, American heart journal.
[19] S. Nattel,et al. Abstract 1343: Atrial Cardiomyocyte Tachycardia Alters Cardiac Fibroblast Function: A Novel Consideration in Atrial Remodeling , 2007 .
[20] E. Niggli,et al. Increased Ca2+ leak and spatiotemporal coherence of Ca2+ release in cardiomyocytes during β‐adrenergic stimulation , 2010, The Journal of physiology.
[21] R. Duisters,et al. MIRNA-133 AND MIRNA-30 REGULATE CONNECTIVE TISSUE GROWTH FACTOR: IMPLICATIONS FOR A ROLE OF MIRNAS IN MYOCARDIAL MATRIX REMODELING , 2013 .
[22] Stanley Nattel,et al. Atrial Tachycardia Remodeling of Pulmonary Vein Cardiomyocytes: Comparison With Left Atrium and Potential Relation to Arrhythmogenesis , 2005, Circulation.
[23] Richard P. Harvey,et al. Molecular Pathway for the Localized Formation of the Sinoatrial Node , 2007, Circulation research.
[24] A. Yndestad,et al. Connective tissue growth factor--a novel mediator of angiotensin II-stimulated cardiac fibroblast activation in heart failure in rats. , 2004, Journal of molecular and cellular cardiology.
[25] W. Park,et al. Down‐regulation of ATBF1 is a major inactivating mechanism in hepatocellular carcinoma , 2008, Histopathology.
[26] B. Lüderitz,et al. Conduction disturbances and increased atrial vulnerability in Connexin40-deficient mice analyzed by transesophageal stimulation. , 1999, Circulation.
[27] R. Hauer,et al. Association of Human Connexin40 Gene Polymorphisms With Atrial Vulnerability as a Risk Factor for Idiopathic Atrial Fibrillation , 2004, Circulation research.
[28] Stephen S. Cha,et al. Secular Trends in Incidence of Atrial Fibrillation in Olmsted County, Minnesota, 1980 to 2000, and Implications on the Projections for Future Prevalence , 2006, Circulation.
[29] A. Gramolini,et al. Ankyrin-B mutation causes type 4 long-QT cardiac arrhythmia and sudden cardiac death , 2003, Nature.
[30] Douglas L. Jones,et al. Somatic mutations in the connexin 40 gene (GJA5) in atrial fibrillation. , 2006, The New England journal of medicine.
[31] Peng-Sheng Chen,et al. New concepts in atrial fibrillation: neural mechanisms and calcium dynamics. , 2009, Cardiology clinics.
[32] Ramon Brugada,et al. Short QT Syndrome and Atrial Fibrillation Caused by Mutation in KCNH2 , 2005, Journal of cardiovascular electrophysiology.
[33] S. Nattel,et al. Atrial Ischemia Promotes Atrial Fibrillation in Dogs , 2003, Circulation.
[34] S. Nattel,et al. Kir3-Based Inward Rectifier Potassium Current: Potential Role in Atrial Tachycardia Remodeling Effects on Atrial Repolarization and Arrhythmias , 2006, Circulation.
[35] Levi A. Gheber,et al. Molecular Basis for Zinc Transporter 1 Action as an Endogenous Inhibitor of L-type Calcium Channels* , 2009, The Journal of Biological Chemistry.
[36] D. Clapham,et al. Evaluation of the role of I(KACh) in atrial fibrillation using a mouse knockout model. , 2001, Journal of the American College of Cardiology.
[37] F A Mathewson,et al. The natural history of atrial fibrillation: incidence, risk factors, and prognosis in the Manitoba Follow-Up Study. , 1995, The American journal of medicine.
[38] U. Schotten,et al. Calmodulin kinase II-mediated sarcoplasmic reticulum Ca2+ leak promotes atrial fibrillation in mice. , 2009, The Journal of clinical investigation.
[39] Sander Verheule,et al. Increased Vulnerability to Atrial Fibrillation in Transgenic Mice With Selective Atrial Fibrosis Caused by Overexpression of TGF-β1 , 2004, Circulation research.
[40] D. Roden,et al. Mutations in Sodium Channel β1- and β2-Subunits Associated With Atrial Fibrillation , 2009, Circulation. Arrhythmia and electrophysiology.
[41] E. Benjamin,et al. Genome-wide association studies of atrial fibrillation: past, present, and future. , 2011, Cardiovascular research.
[42] Martin Bienengraeber,et al. Kv1.5 channelopathy due to KCNA5 loss-of-function mutation causes human atrial fibrillation. , 2006, Human molecular genetics.
[43] Markus Grube,et al. Rac1-induced connective tissue growth factor regulates connexin 43 and N-cadherin expression in atrial fibrillation. , 2010, Journal of the American College of Cardiology.
[44] J. Struck,et al. Circulating cardiovascular biomarkers in recurrent atrial fibrillation: data from the GISSI‐Atrial Fibrillation Trial , 2011, Journal of internal medicine.
[45] A. Goette,et al. Changes in microRNA-1 expression and IK1 up-regulation in human atrial fibrillation. , 2009, Heart rhythm.
[46] S. Nattel. New ideas about atrial fibrillation 50 years on , 2002, Nature.
[47] S. Bendahhou,et al. Identification of a KCNE2 gain-of-function mutation in patients with familial atrial fibrillation. , 2004, American journal of human genetics.
[48] José Jalife,et al. Ionic determinants of functional reentry in a 2-D model of human atrial cells during simulated chronic atrial fibrillation. , 2005, Biophysical journal.
[49] U Ravens,et al. Molecular Basis of Downregulation of G-Protein–Coupled Inward Rectifying K+ Current (IK,ACh) in Chronic Human Atrial Fibrillation: Decrease in GIRK4 mRNA Correlates With Reduced IK,ACh and Muscarinic Receptor–Mediated Shortening of Action Potentials , 2001, Circulation.
[50] Hongmei Li,et al. Neural Mechanisms of Paroxysmal Atrial Fibrillation and Paroxysmal Atrial Tachycardia in Ambulatory Canines , 2008, Circulation.
[51] Michael J Ackerman,et al. Prevalence of early-onset atrial fibrillation in congenital long QT syndrome. , 2008, Heart rhythm.
[52] S. Nattel,et al. Animal models for atrial fibrillation: clinical insights and scientific opportunities. , 2010, Europace : European pacing, arrhythmias, and cardiac electrophysiology : journal of the working groups on cardiac pacing, arrhythmias, and cardiac cellular electrophysiology of the European Society of Cardiology.
[53] H. Yoshida,et al. Genetic factors for lone atrial fibrillation. , 2007, International journal of molecular medicine.
[54] K. Steinmeyer,et al. Molecular mechanisms of early electrical remodeling: transcriptional downregulation of ion channel subunits reduces I(Ca,L) and I(to) in rapid atrial pacing in rabbits. , 2003, Journal of the American College of Cardiology.
[55] S. Nattel,et al. New antiarrhythmic drugs for treatment of atrial fibrillation , 2010, The Lancet.
[56] W. Rottbauer,et al. MicroRNA-21 contributes to myocardial disease by stimulating MAP kinase signalling in fibroblasts , 2008, Nature.
[57] Shuichi Kaneko,et al. AGEs‐RAGE System Mediates Atrial Structural Remodeling in the Diabetic Rat , 2008, Journal of cardiovascular electrophysiology.
[58] S. Hohnloser,et al. Drug therapy , 2022 .
[59] Jun Hu,et al. Ryanodine Receptor-Mediated Ca2+ Events in Atrial Myocytes of Patients with Atrial Fibrillation , 2008, Cardiology.
[60] J. Ruskin,et al. Cardiac sodium channel mutation in atrial fibrillation. , 2008, Heart rhythm.
[61] Fikret Er,et al. Angiotensin II-induced changes of calcium sparks and ionic currents in human atrial myocytes: potential role for early remodeling in atrial fibrillation. , 2006, Cell calcium.
[62] W. Shen,et al. Epidemiology of atrial fibrillation: a current perspective. , 2007, Heart rhythm.
[63] C. Guiraudon,et al. Characterization of Sustained Atrial Tachycardia in Dogs with Rapid Ventricular Pacing‐Induced Heart Failure , 2003, Journal of cardiovascular electrophysiology.
[64] Michael R. Rosen,et al. Pathophysiology and Prevention of Atrial Fibrillation , 2001, Circulation.
[65] D. Dobrev,et al. The G Protein–Gated Potassium Current IK,ACh Is Constitutively Active in Patients With Chronic Atrial Fibrillation , 2005, Circulation.
[66] E. Behr,et al. The variation of the sarcolipin gene (SLN) in atrial fibrillation, long QT syndrome and sudden arrhythmic death syndrome. , 2007, Clinica chimica acta; international journal of clinical chemistry.
[67] A. Gori,et al. Lone and secondary nonvalvular atrial fibrillation: role of a genetic susceptibility. , 2007, International journal of cardiology.
[68] Eric Boerwinkle,et al. Variants in ZFHX3 are associated with atrial fibrillation in individuals of European ancestry , 2009, Nature Genetics.
[69] R F Bosch,et al. Ionic mechanisms of electrical remodeling in human atrial fibrillation. , 1999, Cardiovascular research.
[70] Toru Yamashita,et al. Effects of a Highly Selective Acetylcholine-Activated K+ Channel Blocker on Experimental Atrial Fibrillation , 2011, Circulation. Arrhythmia and electrophysiology.
[71] D. Roden,et al. Novel KCNA5 mutation implicates tyrosine kinase signaling in human atrial fibrillation. , 2010, Heart rhythm.
[72] V. Novack,et al. Selective Molecular Potassium Channel Blockade Prevents Atrial Fibrillation , 2010, Circulation.
[73] Jeffrey E. Thatcher,et al. Dysregulation of microRNAs after myocardial infarction reveals a role of miR-29 in cardiac fibrosis , 2008, Proceedings of the National Academy of Sciences.
[74] Takeshi Imamura,et al. Arkadia amplifies TGF‐β superfamily signalling through degradation of Smad7 , 2003 .
[75] G. Breithardt,et al. Knock-in gain-of-function sodium channel mutation prolongs atrial action potentials and alters atrial vulnerability. , 2010, Heart rhythm.
[76] Niels Voigt,et al. Changes in I K, ACh single-channel activity with atrial tachycardia remodelling in canine atrial cardiomyocytes. , 2008, Cardiovascular research.
[77] Yusong He,et al. A Kir2.1 gain-of-function mutation underlies familial atrial fibrillation. , 2005, Biochemical and biophysical research communications.
[78] L. Lai,et al. Tachycardia of atrial myocytes induces collagen expression in atrial fibroblasts through transforming growth factor β1. , 2011, Cardiovascular research.
[79] M. Rosen,et al. Defective Cardiac Ryanodine Receptor Regulation During Atrial Fibrillation , 2005, Circulation.
[80] A. Camm,et al. Upstream therapies for management of atrial fibrillation: review of clinical evidence and implications for European Society of Cardiology guidelines. Part I: primary prevention. , 2011, Europace : European pacing, arrhythmias, and cardiac electrophysiology : journal of the working groups on cardiac pacing, arrhythmias, and cardiac cellular electrophysiology of the European Society of Cardiology.
[81] J Clémenty,et al. Spontaneous initiation of atrial fibrillation by ectopic beats originating in the pulmonary veins. , 1998, The New England journal of medicine.
[82] G. Hasenfuss,et al. CaMKII-Dependent Diastolic SR Ca2+ Leak and Elevated Diastolic Ca2+ Levels in Right Atrial Myocardium of Patients With Atrial Fibrillation , 2010, Circulation research.
[83] Keiko Tsuji,et al. A novel SCN5A gain-of-function mutation M1875T associated with familial atrial fibrillation. , 2008, Journal of the American College of Cardiology.
[84] G. Lip,et al. Lone atrial fibrillation: what do we know? , 2009, Heart.
[85] A. Zima,et al. Inositol‐1,4,5‐trisphosphate‐dependent Ca2+ signalling in cat atrial excitation–contraction coupling and arrhythmias , 2004, The Journal of physiology.
[86] Michel Haïssaguerre,et al. Loss-of-Function Mutations in the Cardiac Calcium Channel Underlie a New Clinical Entity Characterized by ST-Segment Elevation, Short QT Intervals, and Sudden Cardiac Death , 2007, Circulation.
[87] S. Nattel. From guidelines to bench: implications of unresolved clinical issues for basic investigations of atrial fibrillation mechanisms. , 2011, The Canadian journal of cardiology.
[88] A. Marks,et al. The ryanodine receptor in cardiac physiology and disease. , 2010, Advances in pharmacology.
[89] Stefan Wagner,et al. Altered Na(+) currents in atrial fibrillation effects of ranolazine on arrhythmias and contractility in human atrial myocardium. , 2010, Journal of the American College of Cardiology.
[90] Robert Lemery,et al. Gain-of-function mutation of Nav1.5 in atrial fibrillation enhances cellular excitability and lowers the threshold for action potential firing. , 2009, Biochemical and biophysical research communications.
[91] H. Nakauchi,et al. Cardiac mast cells cause atrial fibrillation through PDGF-A-mediated fibrosis in pressure-overloaded mouse hearts. , 2010, The Journal of clinical investigation.
[92] Thomas Meitinger,et al. Common Variants in KCNN3 are Associated with Lone Atrial Fibrillation , 2010, Nature Genetics.
[93] Wei Huang,et al. KCNQ1 Gain-of-Function Mutation in Familial Atrial Fibrillation , 2003, Science.
[94] D. Zipes,et al. NO modulates autonomic effects on sinus discharge rate and AV nodal conduction in open-chest dogs. , 1997, The American journal of physiology.
[95] Nolwenn Le Meur,et al. Human Atrial Ion Channel and Transporter Subunit Gene-Expression Remodeling Associated With Valvular Heart Disease and Atrial Fibrillation , 2005, Circulation.
[96] S. Hohnloser,et al. Atrial Fibrillation and Congestive Heart Failure: Specific Considerations at the Intersection of Two Common and Important Cardiac Disease Sets , 2002, Journal of cardiovascular electrophysiology.
[97] Zhao Zhang,et al. Ablation of a Ca2+‐activated K+ channel (SK2 channel) results in action potential prolongation in atrial myocytes and atrial fibrillation , 2009, The Journal of physiology.
[98] S. Nattel,et al. Mechanisms of Atrial Tachyarrhythmias Associated With Coronary Artery Occlusion in a Chronic Canine Model , 2011, Circulation.
[99] Shenming Wang,et al. Atrial Fibrillation Induces Myocardial Fibrosis Through Angiotensin II Type 1 Receptor–Specific Arkadia-Mediated Downregulation of Smad7 , 2011, Circulation research.
[100] Masanori Hirose,et al. Early electrical remodeling in rabbit pulmonary vein results from trafficking of intracellular SK2 channels to membrane sites. , 2007, Cardiovascular research.
[101] S. Nattel,et al. Atrial fibrosis: mechanisms and clinical relevance in atrial fibrillation. , 2008, Journal of the American College of Cardiology.
[102] M. Allessie,et al. Atrial fibrillation begets atrial fibrillation. A study in awake chronically instrumented goats. , 1995, Circulation.
[103] T. Olson,et al. A Common Polymorphism in SCN5A is Associated with Lone Atrial Fibrillation , 2007, Clinical pharmacology and therapeutics.
[104] S. Nattel,et al. Differential Behaviors of Atrial Versus Ventricular Fibroblasts: A Potential Role for Platelet-Derived Growth Factor in Atrial-Ventricular Remodeling Differences , 2008, Circulation.
[105] E. Benjamin,et al. Current perceptions of the epidemiology of atrial fibrillation. , 2009, Cardiology clinics.
[106] I. V. Van Gelder,et al. New risk factors for atrial fibrillation: causes of 'not-so-lone atrial fibrillation'. , 2008, Europace : European pacing, arrhythmias, and cardiac electrophysiology : journal of the working groups on cardiac pacing, arrhythmias, and cardiac cellular electrophysiology of the European Society of Cardiology.
[107] S. Nattel,et al. Cellular Signaling Underlying Atrial Tachycardia Remodeling of L-type Calcium Current , 2008, Circulation research.
[108] J. Nerbonne,et al. Atrial L-type Ca2+ currents and human atrial fibrillation. , 1999, Circulation research.
[109] S. Nattel,et al. Rate-dependence of atrial tachycardia effects on atrial refractoriness and atrial fibrillation maintenance. , 2009, Cardiovascular research.
[110] R. Bosch,et al. Atrial L-type Ca2+-channel, β-adrenoreceptor, and 5-hydroxytryptamine type 4 receptor mRNAs in human atrial fibrillation , 2001, Basic Research in Cardiology.
[111] Niels Voigt,et al. The ryanodine receptor channel as a molecular motif in atrial fibrillation: pathophysiological and therapeutic implications. , 2011, Cardiovascular research.
[112] L. Hunyady,et al. Pleiotropic AT1 receptor signaling pathways mediating physiological and pathogenic actions of angiotensin II. , 2006, Molecular endocrinology.
[113] Maurits A. Allessie,et al. Reverse Structural and Gap-Junctional Remodeling After Prolonged Atrial Fibrillation in the Goat , 2003, Circulation.
[114] Jan D'hooge,et al. Ultrastructural and Functional Remodeling of the Coupling Between Ca2+ Influx and Sarcoplasmic Reticulum Ca2+ Release in Right Atrial Myocytes From Experimental Persistent Atrial Fibrillation , 2009, Circulation research.
[115] Shien-Fong Lin,et al. Intrinsic Cardiac Nerve Activity and Paroxysmal Atrial Tachyarrhythmia in Ambulatory Dogs , 2010, Circulation.
[116] C. Vahl,et al. Human Cardiac Inwardly-Rectifying K+ Channel Kir2.1b Is Inhibited by Direct Protein Kinase C-Dependent Regulation in Human Isolated Cardiomyocytes and in an Expression System , 2002, Circulation.
[117] V. Shusterman,et al. Atrial contractile dysfunction, fibrosis, and arrhythmias in a mouse model of cardiomyopathy secondary to cardiac-specific overexpression of tumor necrosis factor-{alpha}. , 2005, American journal of physiology. Heart and circulatory physiology.
[118] D. Levy,et al. Prevention of Atrial Fibrillation: Report From a National Heart, Lung, and Blood Institute Workshop , 2009, Circulation.
[119] U Ravens,et al. Atrial Fibrillation Is Associated With Increased Activity of , 2022 .
[120] Leif Hove-Madsen,et al. Atrial Fibrillation Is Associated With Increased Spontaneous Calcium Release From the Sarcoplasmic Reticulum in Human Atrial Myocytes , 2004, Circulation.
[121] L. J. Leon,et al. Cholinergic Atrial Fibrillation in a Computer Model of a Two-Dimensional Sheet of Canine Atrial Cells With Realistic Ionic Properties , 2002, Circulation research.
[122] Thomas Jespersen,et al. Inhibition of Small-Conductance Ca2+-Activated K+ Channels Terminates and Protects Against Atrial Fibrillation , 2010, Circulation. Arrhythmia and electrophysiology.
[123] S. Nattel,et al. Changes in Connexin Expression and the Atrial Fibrillation Substrate in Congestive Heart Failure , 2009, Circulation research.
[124] D. Dobrev,et al. Differential phosphorylation-dependent regulation of constitutively active and muscarinic receptor-activated IK,ACh channels in patients with chronic atrial fibrillation. , 2007, Cardiovascular research.
[125] S Nattel,et al. Molecular mechanisms underlying ionic remodeling in a dog model of atrial fibrillation. , 1999, Circulation research.
[126] Mark I McCarthy,et al. Genome-wide association studies: past, present and future. , 2008, Human molecular genetics.
[127] B. Stambler,et al. Mapping of Atrial Activation During Sustained Atrial Fibrillation in Dogs with Rapid Ventricular Pacing Induced Heart Failure: Evidence for a Role of Driver Regions , 2005, Journal of cardiovascular electrophysiology.
[128] Jeffrey L. Anderson,et al. Sodium channel mutations and susceptibility to heart failure and atrial fibrillation. , 2005, JAMA.
[129] Ursula Ravens,et al. Molecular Determinants of Altered Ca2+ Handling in Human Chronic Atrial Fibrillation , 2006, Circulation.
[130] Richard P Harvey,et al. Pitx2c and Nkx2-5 Are Required for the Formation and Identity of the Pulmonary Myocardium , 2007, Circulation research.
[131] Yanjie Lu,et al. MicroRNAs and atrial fibrillation: new fundamentals. , 2011, Cardiovascular research.
[132] S. Kauppinen,et al. Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice. , 2010, The Journal of clinical investigation.
[133] S. Nattel,et al. Atrial fibrillation-associated minK38G/S polymorphism modulates delayed rectifier current and membrane localization. , 2005, Cardiovascular research.
[134] S Nattel,et al. Ionic remodeling underlying action potential changes in a canine model of atrial fibrillation. , 1997, Circulation research.
[135] F. Mohr,et al. Improving cardiac gap junction communication as a new antiarrhythmic mechanism: the action of antiarrhythmic peptides , 2010, Naunyn-Schmiedeberg's Archives of Pharmacology.
[136] Stanley Nattel,et al. Calcium-Handling Abnormalities Underlying Atrial Arrhythmogenesis and Contractile Dysfunction in Dogs With Congestive Heart Failure , 2008, Circulation. Arrhythmia and electrophysiology.
[137] J. Svendsen,et al. Mutations in sodium channel β-subunit SCN3B are associated with early-onset lone atrial fibrillation. , 2011, Cardiovascular research.
[138] K. Pietras,et al. Platelet-Derived Growth Factor D Induces Cardiac Fibrosis and Proliferation of Vascular Smooth Muscle Cells in Heart-Specific Transgenic Mice , 2005, Circulation research.
[139] D. Maclennan,et al. Store overload‐induced Ca2+ release as a triggering mechanism for CPVT and MH episodes caused by mutations in RYR and CASQ genes , 2009, The Journal of physiology.
[140] J. Tolstrup,et al. Alcohol Consumption and Risk of Atrial Fibrillation in Men and Women: The Copenhagen City Heart Study , 2005, Circulation.
[141] José Jalife,et al. Frequency-Dependent Breakdown of Wave Propagation Into Fibrillatory Conduction Across the Pectinate Muscle Network in the Isolated Sheep Right Atrium , 2002, Circulation research.
[142] Nolwenn Le Meur,et al. Contrasting Gene Expression Profiles in Two Canine Models of Atrial Fibrillation , 2007, Circulation research.
[143] M. Allessie,et al. Distinct contractile and molecular differences between two goat models of atrial dysfunction: AV block-induced atrial dilatation and atrial fibrillation. , 2009, Journal of molecular and cellular cardiology.
[144] S Nattel,et al. Promotion of atrial fibrillation by heart failure in dogs: atrial remodeling of a different sort. , 1999, Circulation.
[145] F. Sofi,et al. Analysis of minK and eNOS genes as candidate loci for predisposition to non-valvular atrial fibrillation. , 2006, European heart journal.
[146] A. Toker,et al. Lacrimal gland PKC isoforms are differentially involved in agonist-induced protein secretion. , 1997, The American journal of physiology.