Factors causing malnutrition in patients with chronic uremia.

[1]  J. Walls,et al.  Role of an improvement in acid-base status and nutrition in CAPD patients. , 1997, Kidney international.

[2]  J. Wernerman,et al.  Correction of acidosis in dialysis patients increases branched-chain and total essential amino acid levels in muscle. , 1997, Clinical nephrology.

[3]  A. Levey,et al.  Effect of dietary protein restriction on nutritional status in the Modification of Diet in Renal Disease Study. , 1997, Kidney international.

[4]  A. Manatunga,et al.  Mechanisms permitting nephrotic patients to achieve nitrogen equilibrium with a protein-restricted diet. , 1997, The Journal of clinical investigation.

[5]  W. Mitch,et al.  Role of nutrition in prevention of the progression of renal disease. , 1997, Annual review of nutrition.

[6]  A. Goldberg,et al.  Mechanisms of muscle wasting. The role of the ubiquitin-proteasome pathway. , 1996, The New England journal of medicine.

[7]  P. Södersten,et al.  Middle-sized molecule fractions isolated from uremic ultrafiltrate and normal urine inhibit ingestive behavior in the rat. , 1996, Journal of the American Society of Nephrology : JASN.

[8]  W. Mitch,et al.  The acidosis of chronic renal failure activates muscle proteolysis in rats by augmenting transcription of genes encoding proteins of the ATP-dependent ubiquitin-proteasome pathway. , 1996, The Journal of clinical investigation.

[9]  J. Bergström.  Nutrition and mortality in hemodialysis. , 1995, Journal of the American Society of Nephrology : JASN.

[10]  M. McNurlan,et al.  Chronic metabolic acidosis decreases albumin synthesis and induces negative nitrogen balance in humans. , 1995, The Journal of clinical investigation.

[11]  W. Mitch,et al.  Metabolic acidosis and uremic toxicity: protein and amino acid metabolism. , 1994, Seminars in nephrology.

[12]  C. Scrimgeour,et al.  Correction of acidosis in humans with CRF decreases protein degradation and amino acid oxidation. , 1993, The American journal of physiology.

[13]  J. Walls,et al.  Metabolic acidosis and skeletal muscle adaptation to low protein diets in chronic uremia. , 1991, Kidney international.

[14]  E G Lowrie,et al.  Death risk in hemodialysis patients: the predictive value of commonly measured variables and an evaluation of death rate differences between facilities. , 1990, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[15]  W. Mitch,et al.  Acidosis, not azotemia, stimulates branched-chain, amino acid catabolism in uremic rats. , 1987, Kidney international.

[16]  J. Kopple,et al.  Energy expenditure in patients with chronic renal failure. , 1986, Kidney international.

[17]  G. Kaysen,et al.  Effect of dietary protein intake on albumin homeostasis in nephrotic patients. , 1986, Kidney international.

[18]  W. Mitch,et al.  A method for estimating nitrogen intake of patients with chronic renal failure. , 1985, Kidney international.

[19]  J. Kopple,et al.  Metabolic balance studies and dietary protein requirements in patients undergoing continuous ambulatory peritoneal dialysis. , 1982, Kidney international.

[20]  F. Gotch,et al.  Nitrogen balance during intermittent dialysis therapy of uremia. , 1978, Kidney international.

[21]  J. Kopple,et al.  METABOLIC STUDIES OF LOW PROTEIN DIETS IN UREMIA: I. NlTEOGEN AND POTASSIUM , 1973, Medicine.