Tumor necrosis factor-enhanced leukotriene B4 generation and chemotaxis in human neutrophils.
暂无分享,去创建一个
K. Tracey | S. Lowry | Y. Fong | D. Hesse | J. Meyer | R. Yurt | R. Duhaney | M. Verma | G. T. Shires | P. Dineen | J. D. Meyer | D. G. Hesse
[1] M. Vadas,et al. Recombinant human tumor necrosis factor-alpha. Regulation of N-formylmethionylleucylphenylalanine receptor affinity and function on human neutrophils. , 1988, The Journal of clinical investigation.
[2] T. Ulich,et al. Kinetics and mechanisms of recombinant human interleukin 1 and tumor necrosis factor-alpha-induced changes in circulating numbers of neutrophils and lymphocytes. , 1987, Journal of immunology.
[3] M. Klempner,et al. Human recombinant interleukin 1 beta has no effect on intracellular calcium or on functional responses of human neutrophils. , 1987, Journal of immunology.
[4] A. Mantovani,et al. Tumor necrosis factor is chemotactic for monocytes and polymorphonuclear leukocytes. , 1987, Journal of immunology.
[5] J. D. Albert,et al. Shock and tissue injury induced by recombinant human cachectin. , 1986, Science.
[6] J. Rosenbaum,et al. Lipopolysaccharide modulates receptors for leukotriene B4, C5a, and formyl-methionyl-leucyl-phenylalanine on rabbit polymorphonuclear leukocytes. , 1986, Journal of immunology.
[7] G. Weissmann,et al. Tumor necrosis factor provokes superoxide anion generation from neutrophils. , 1986, Biochemical and biophysical research communications.
[8] J. Gamble,et al. Stimulation of neutrophils by tumor necrosis factor. , 1986, Journal of immunology.
[9] B. Beutler,et al. Cachectin and tumour necrosis factor as two sides of the same biological coin , 1986, Nature.
[10] J. Vilček,et al. Fibroblast growth enhancing activity of tumor necrosis factor and its relationship to other polypeptide growth factors , 1986, The Journal of experimental medicine.
[11] J. Rosenbaum,et al. Endotoxin-induced selective dysfunction of rabbit polymorphonuclear leukocytes in response to endogenous chemotactic factors , 1985, Infection and immunity.
[12] B. Aggarwal,et al. Activation of human polymorphonuclear neutrophil functions by interferon-gamma and tumor necrosis factors. , 1985, Journal of immunology.
[13] B. Beutler,et al. Passive immunization against cachectin/tumor necrosis factor protects mice from lethal effect of endotoxin. , 1985, Science.
[14] B. Beutler,et al. Identity of tumour necrosis factor and the macrophage-secreted factor cachectin , 1985, Nature.
[15] B. Beutler,et al. Purification of cachectin, a lipoprotein lipase-suppressing hormone secreted by endotoxin-induced RAW 264.7 cells , 1985, The Journal of experimental medicine.
[16] C. Haslett,et al. Modulation of multiple neutrophil functions by preparative methods or trace concentrations of bacterial lipopolysaccharide. , 1985, The American journal of pathology.
[17] L. Mcphail,et al. Priming of neutrophils for enhanced release of oxygen metabolites by bacterial lipopolysaccharide. Evidence for increased activity of the superoxide-producing enzyme , 1984, The Journal of experimental medicine.
[18] J. Rosenbaum,et al. Antiinflammatory effects of endotoxin. Inhibition of rabbit polymorphonuclear leukocyte responses to complement (C5)-derived peptides in vivo and in vitro. , 1983, The American journal of pathology.
[19] C. Galanos,et al. Granulocyte activation by endotoxin. I. Correlation between adherence and other granulocyte functions, and role of endotoxin structure on biologic activity. , 1983, Journal of immunology.
[20] R. Proctor. Endotoxin in vitro interactions with human neutrophils: depression of chemiluminescence, oxygen consumption, superoxide production, and killing , 1979, Infection and immunity.
[21] K. Brigham,et al. Increased Sheep Lung Vascular Permeability Caused by Escherichia coli Endotoxin , 1979, Circulation research.
[22] B. Samuelsson,et al. Arachidonic acid metabolism in polymorphonuclear leukocytes: effects of ionophore A23187. , 1979, Proceedings of the National Academy of Sciences of the United States of America.
[23] L. Kline,et al. Activation of the classical and properdin pathways of complement by bacterial lipopolysaccharides (LPS). , 1977, Journal of immunology.
[24] A. Mauer,et al. Leukokinetic studies. IV. The total blood, circulating and marginal granulocyte pools and the granulocyte turnover rate in normal subjects. , 1961, The Journal of clinical investigation.
[25] P. Pekala,et al. Model for cachexia in chronic disease: secretory products of endotoxin-stimulated macrophages induce a catabolic state in 3T3-L1 adipocytes. , 1984, Transactions of the Association of American Physicians.