Human BRCA1-BARD1 Ubiquitin ligase activity counters chromatin barriers to DNA resection
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N. Keep | J. Beesley | R. Densham | Alexander J. Garvin | M. Daza-Martin | Sarah Blair-Reid | Helen R. Stone | R. Baldock | Joanna Strachan | Daza-Martin | Manuel Fletcher | Alice | Blair-Reed Sarah | Laurence H. Pearl | Robert Neely | Nicholas H Watts | Felicity Z | Morris | Joanna R | Robert A 6 Baldock | Alice Fletcher | Balraj | Johal | Felicity Z. Watts
[1] P. Cejka,et al. DNA End Resection: Nucleases Team Up with the Right Partners to Initiate Homologous Recombination* , 2015, The Journal of Biological Chemistry.
[2] R. Greenberg,et al. Deciphering the BRCA1 Tumor Suppressor Network* , 2015, The Journal of Biological Chemistry.
[3] D. Durocher,et al. MAD2L2 controls DNA repair at telomeres and DNA breaks by inhibiting 5′ end-resection , 2015, Nature.
[4] Peter Bouwman,et al. REV7 counteracts DNA double-strand break resection and affects PARP inhibition , 2015, Nature.
[5] David J. Chen,et al. PTIP associates with Artemis to dictate DNA repair pathway choice , 2014, Genes & development.
[6] P. Huertas,et al. BRCA1 accelerates CtIP-mediated DNA-end resection. , 2014, Cell reports.
[7] Stéphanie Panier,et al. Double-strand break repair: 53BP1 comes into focus , 2013, Nature Reviews Molecular Cell Biology.
[8] I. Hickson,et al. A role for BLM in double-strand break repair pathway choice: prevention of CtIP/Mre11-mediated alternative nonhomologous end-joining. , 2013, Cell reports.
[9] A. Shibata,et al. Co-operation of BRCA1 and POH1 relieves the barriers posed by 53BP1 and RAP80 to resection , 2013, Nucleic acids research.
[10] M. Hendzel,et al. A Small Molecule Inhibitor of Polycomb Repressive Complex 1 Inhibits Ubiquitin Signaling at DNA Double-strand Breaks* , 2013, The Journal of Biological Chemistry.
[11] Danny T. Huang,et al. Essentiality of a non-RING element in priming donor ubiquitin for catalysis by a monomeric E3 , 2013, Nature Structural &Molecular Biology.
[12] Dustin E. Schones,et al. The ATP-dependent Chromatin Remodeling Enzyme Fun30 Represses Transcription by Sliding Promoter-proximal Nucleosomes* , 2013, The Journal of Biological Chemistry.
[13] Adam P. Rosebrock,et al. A cell cycle-dependent regulatory circuit composed of 53BP1-RIF1 and BRCA1-CtIP controls DNA repair pathway choice. , 2013, Molecular cell.
[14] R. Aebersold,et al. A DNA-Damage Selective Role for BRCA1 E3 Ligase in Claspin Ubiquitylation, CHK1 Activation, and DNA Repair , 2012, Current Biology.
[15] James E. Haber,et al. The Saccharomyces cerevisiae Chromatin Remodeler Fun30 Regulates DNA End Resection and Checkpoint Deactivation , 2012, Molecular and Cellular Biology.
[16] S. Jackson,et al. BRCA1-associated exclusion of 53BP1 from DNA damage sites underlies temporal control of DNA repair , 2012, Journal of Cell Science.
[17] Agnès Thierry,et al. The yeast Fun30 and human SMARCAD1 chromatin remodelers promote DNA end resection , 2012, Nature.
[18] Hong Wu,et al. A distinct replication fork protection pathway connects Fanconi anemia tumor suppressors to RAD51-BRCA1/2. , 2012, Cancer cell.
[19] J. Walter,et al. A novel function for BRCA1 in crosslink repair. , 2012, Molecular cell.
[20] K. Khanna,et al. Exo1 plays a major role in DNA end resection in humans and influences double-strand break repair and damage signaling decisions. , 2012, DNA repair.
[21] Peter Bouwman,et al. BRCA1 RING function is essential for tumor suppression but dispensable for therapy resistance. , 2011, Cancer cell.
[22] F. Gage,et al. BRCA1 tumor suppression occurs via heterochromatin mediated silencing , 2011, Nature.
[23] J. Naismith,et al. Mechanism of ubiquitylation by dimeric RING ligase RNF4 , 2011, Nature Structural &Molecular Biology.
[24] T. Sixma,et al. Symmetry and asymmetry of the RING-RING dimer of Rad18. , 2011, Journal of molecular biology.
[25] D. Oxley,et al. Maintenance of silent chromatin through replication requires SWI/SNF-like chromatin remodeler SMARCAD1. , 2011, Molecular cell.
[26] Olivia Barton,et al. Role of ATM and the Damage Response Mediator Proteins 53BP1 and MDC1 in the Maintenance of G2/M Checkpoint Arrest , 2010, Molecular and Cellular Biology.
[27] Jeremy M. Stark,et al. 53BP1 Inhibits Homologous Recombination in Brca1-Deficient Cells by Blocking Resection of DNA Breaks , 2010, Cell.
[28] T. Owen-Hughes,et al. The Snf2 Homolog Fun30 Acts as a Homodimeric ATP-dependent Chromatin-remodeling Enzyme* , 2010, The Journal of Biological Chemistry.
[29] J. Miller,et al. The SNF2-Family Member Fun30 Promotes Gene Silencing in Heterochromatic Loci , 2009, PloS one.
[30] T. Ludwig,et al. E3 ligase activity of BRCA1 is not essential for mammalian cell viability or homology-directed repair of double-strand DNA breaks , 2008, Proceedings of the National Academy of Sciences.
[31] Oliver Weichenrieder,et al. Structure and E3‐ligase activity of the Ring–Ring complex of Polycomb proteins Bmi1 and Ring1b , 2006, The EMBO journal.
[32] T. Katagiri,et al. Genetic analysis of BRCA1 ubiquitin ligase activity and its relationship to breast cancer susceptibility. , 2006, Human molecular genetics.
[33] N. Curtin,et al. Identification and Characterization of a Novel and Specific Inhibitor of the Ataxia-Telangiectasia Mutated Kinase ATM , 2004, Cancer Research.
[34] T. Ohta,et al. Binding and recognition in the assembly of an active BRCA1/BARD1 ubiquitin-ligase complex , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[35] J. Hurley,et al. A ubiquitin‐binding motif required for intramolecular monoubiquitylation, the CUE domain , 2003, The EMBO journal.
[36] J. Bartek,et al. Distinct spatiotemporal dynamics of mammalian checkpoint regulators induced by DNA damage , 2003, Nature Cell Biology.
[37] C. Vandenberg,et al. Activation of the E3 ligase function of the BRCA1/BARD1 complex by polyubiquitin chains , 2002, The EMBO journal.
[38] N. Keep,et al. Identification of Residues Required for the Interaction of BARD1 with BRCA1* , 2002, The Journal of Biological Chemistry.
[39] C. Boutell,et al. Herpes Simplex Virus Type 1 Immediate-Early Protein ICP0 and Its Isolated RING Finger Domain Act as Ubiquitin E3 Ligases In Vitro , 2002, Journal of Virology.
[40] Rachel E. Klevit,et al. Structure of a BRCA1–BARD1 heterodimeric RING–RING complex , 2001, Nature Structural Biology.
[41] J. Pruneda,et al. RING-type E3 ligases: master manipulators of E2 ubiquitin-conjugating enzymes and ubiquitination. , 2014, Biochimica et biophysica acta.
[42] D. Mallery,et al. University of Dundee BRCA 1 is a histone-H 2 A-specific ubiquitin ligase , 2014 .