Nitric oxide‐mediated inhibition of caspase‐dependent T lymphocyte proliferation
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[1] R. Budd. Death receptors couple to both cell proliferation and apoptosis. , 2002, The Journal of clinical investigation.
[2] H. Ford,et al. Nitric oxide mediates dendritic cell apoptosis by downregulating inhibitors of apoptosis proteins and upregulating effector caspase activity. , 2001, Surgery.
[3] Y. Wan,et al. Transgenic expression of the coxsackie/adenovirus receptor enables adenoviral-mediated gene delivery in naive T cells. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[4] Y. Vodovotz,et al. Inducible Nitric Oxide Synthase and Inflammatory Diseases , 2000, Molecular medicine.
[5] T. Billiar,et al. Cellular Non-heme Iron Content Is a Determinant of Nitric Oxide-mediated Apoptosis, Necrosis, and Caspase Inhibition* , 2000, The Journal of Biological Chemistry.
[6] D. Wink,et al. Mechanisms of Cell Death Governed by the Balance between Nitrosative and Oxidative Stress , 2000, Annals of the New York Academy of Sciences.
[7] R. Sékaly,et al. Early Activation of Caspases during T Lymphocyte Stimulation Results in Selective Substrate Cleavage in Nonapoptotic Cells , 1999, The Journal of experimental medicine.
[8] J. Tschopp,et al. Caspase Activation Is Required for T Cell Proliferation , 1999, The Journal of experimental medicine.
[9] A. Allione,et al. Nitric oxide suppresses human T lymphocyte proliferation through IFN-gamma-dependent and IFN-gamma-independent induction of apoptosis. , 1999, Journal of immunology.
[10] M. Zeng,et al. Fas-induced caspase denitrosylation. , 1999, Science.
[11] T. Billiar,et al. Nitric Oxide Prevents IL-1β and IFN-γ-Inducing Factor (IL-18) Release from Macrophages by Inhibiting Caspase-1 (IL-1β-Converting Enzyme) , 1998, The Journal of Immunology.
[12] P. Holt,et al. Macrophage-derived nitric oxide regulates T cell activation via reversible disruption of the Jak3/STAT5 signaling pathway. , 1998, Journal of immunology.
[13] A. Zeiher,et al. Nitric oxide inhibits APO-1/Fas-mediated cell death. , 1998, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.
[14] John Calvin Reed,et al. Granzyme Release and Caspase Activation in Activated Human T-Lymphocytes* , 1998, The Journal of Biological Chemistry.
[15] G. Häcker,et al. Activation of caspase‐3‐like enzymes in non‐apoptotic T cells , 1998, European journal of immunology.
[16] T. Billiar,et al. Nitric Oxide Inhibits Apoptosis by Preventing Increases in Caspase-3-like Activity via Two Distinct Mechanisms* , 1997, The Journal of Biological Chemistry.
[17] T. Billiar,et al. Nitric oxide reversibly inhibits seven members of the caspase family via S-nitrosylation. , 1997, Biochemical and biophysical research communications.
[18] C. Martínez-A,et al. Nitric oxide regulates clonal expansion and activation-induced cell death triggered by staphylococcal enterotoxin B , 1997, Infection and immunity.
[19] J. Stamler,et al. Nitric Oxide Inhibits Fas-induced Apoptosis* , 1997, The Journal of Biological Chemistry.
[20] F. Fassy,et al. Evidence for CPP32 Activation in the Absence of Apoptosis during T Lymphocyte Stimulation* , 1997, The Journal of Biological Chemistry.
[21] M. Nehls,et al. Suppression of Apoptosis by Nitric Oxide via Inhibition of Interleukin-1β–converting Enzyme (ICE)-like and Cysteine Protease Protein (CPP)-32–like Proteases , 1997, The Journal of experimental medicine.
[22] H. Vapaatalo,et al. Nitric oxide in inflammation and immune response. , 1995, Annals of medicine.
[23] T. M. Walker,et al. Induction of macrophage suppressor activity by fibrosarcoma‐derived transforming growth factor‐β1: contrasting effects on resting and activated macrophages , 1995, Journal of leukocyte biology.
[24] C. Hunter,et al. Roles of gamma interferon and other cytokines in suppression of the spleen cell proliferative response to concanavalin A and toxoplasma antigen during acute toxoplasmosis , 1995, Infection and immunity.
[25] P. Lagadec,et al. Nitric oxide involvement in tumor-induced immunosuppression. , 1994, Journal of immunology.
[26] K. Rockett,et al. Possible role of nitric oxide in malarial immunosuppression , 1994, Parasite immunology.
[27] R. Simmons,et al. Reactive nitrogen intermediates suppress the primary immunologic response to Listeria. , 1993, Journal of immunology.
[28] T. Billiar,et al. EVIDENCE THAT NITRIC OXIDE PRODUCTION BY IN VIVO ALLOSENSITIZED CELLS INHIBITS THE DEVELOPMENT OF ALLOSPECIFIC CTL , 1992, Transplantation.
[29] T. Billiar,et al. Alloantigen-induced activation of rat splenocytes is regulated by the oxidative metabolism of L-arginine. , 1990, Journal of immunology.
[30] R. Siegel,et al. Mature T lymphocyte apoptosis--immune regulation in a dynamic and unpredictable antigenic environment. , 1999, Annual review of immunology.
[31] M. Lutter,et al. Biochemical pathways of caspase activation during apoptosis. , 1999, Annual review of cell and developmental biology.
[32] T. Billiar,et al. Nitric oxide prevents IL-1beta and IFN-gamma-inducing factor (IL-18) release from macrophages by inhibiting caspase-1 (IL-1beta-converting enzyme). , 1998, Journal of immunology.
[33] C. Nathan,et al. Nitric oxide and macrophage function. , 1997, Annual review of immunology.