Effects of potassium channel blockers on synaptic plasticity in the corticostriatal pathway
暂无分享,去创建一个
[1] J. Wickens,et al. Dopamine reverses the depression of rat corticostriatal synapses which normally follows high-frequency stimulation of cortex In vitro , 1996, Neuroscience.
[2] P. Calabresi,et al. Transmitter Release Associated with Long‐term Synaptic Depression in Rat Corticostriatal Slices , 1995, The European journal of neuroscience.
[3] P. Calabresi,et al. Post-receptor mechanisms underlying striatal long-term depression , 1994, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[4] D. Lovinger,et al. Short- and long-term synaptic depression in rat neostriatum. , 1993, Journal of neurophysiology.
[5] J. Wickens. A Theory of the Striatum , 1993 .
[6] J. Walsh. Depression of excitatory synaptic input in rat striatal neurons , 1993, Brain Research.
[7] Yy Huang,et al. Examination of TEA-induced synaptic enhancement in area CA1 of the hippocampus: the role of voltage-dependent Ca2+ channels in the induction of LTP , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[8] F. H. Lopes da Silva,et al. Synaptic Plasticity in an In Vitro Slice Preparation of the Rat Nucleus Accumbens , 1993, The European journal of neuroscience.
[9] T. Bliss,et al. A synaptic model of memory: long-term potentiation in the hippocampus , 1993, Nature.
[10] P. Calabresi,et al. Long-term synaptic depression in the striatum: physiological and pharmacological characterization , 1992, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[11] P. Calabresi,et al. Long‐term Potentiation in the Striatum is Unmasked by Removing the Voltage‐dependent Magnesium Block of NMDA Receptor Channels , 1992, The European journal of neuroscience.
[12] V. Olivier,et al. Differential Effects of Potassium Channel Blockers on Dopamine Release from Rat Striatal Slices , 1991, The Journal of pharmacy and pharmacology.
[13] Y. Ben-Ari,et al. Novel form of long-term potentiation produced by a K+channel blocker in the hippocampus , 1991, Nature.
[14] Y. Ben-Ari,et al. NMDA-independent form of long-term potentiation produced by tetraethylammonium in the hippocampal CA1 region. , 1990, European journal of pharmacology.
[15] C. Wilson,et al. Intracellular recording of identified neostriatal patch and matrix spiny cells in a slice preparation preserving cortical inputs. , 1989, Journal of neurophysiology.
[16] E. Cherubini,et al. An inward calcium current underlying regenerative calcium potentials in rat striatal neurons in vitro enhanced by BAY K 8644 , 1987, Neuroscience.
[17] P. Calabresi,et al. Intrinsic membrane properties of neostriatal neurons can account for their low level of spontaneous activity , 1987, Neuroscience.
[18] R. Beninger. The role of dopamine in locomotor activity and learning , 1983, Brain Research Reviews.
[19] M. Sugimori,et al. Response properties and electrical constants of caudate nucleus neurons in the cat. , 1978, Journal of neurophysiology.
[20] R. Werman,et al. Tetraethylammonium Ions: Effect of Presynaptic Injection on Synaptic Transmission , 1967, Science.
[21] J. Wickens,et al. Cellular models of reinforcement. , 1995 .
[22] M. Mauk,et al. Distinct LTP induction mechanisms: contribution of NMDA receptors and voltage-dependent calcium channels. , 1995, Journal of neurophysiology.
[23] Joel L. Davis,et al. Adaptive Critics and the Basal Ganglia , 1995 .
[24] N. White. A functional hypothesis concerning the striatal matrix and patches: mediation of S-R memory and reward. , 1989, Life sciences.
[25] P. Stanfield. Tetraethylammonium ions and the potassium permeability of excitable cells. , 1983, Reviews of physiology, biochemistry and pharmacology.