Altered effects of potassium channel modulation in the coronary circulation in experimental hypercholesterolemia.

[1]  L. Lerman,et al.  In vivo renal vascular and tubular function in experimental hypercholesterolemia. , 1999, Hypertension.

[2]  A. Lerman,et al.  New insight into coronary endothelial dysfunction: role of endothelin. , 1998, The Journal of laboratory and clinical medicine.

[3]  R. Bache,et al.  ATP-sensitive K+ channels, adenosine, and nitric oxide-mediated mechanisms account for coronary vasodilation during exercise. , 1998, Circulation research.

[4]  A. Terzic,et al.  Operative condition-dependent response of cardiac ATP-sensitive K+ channels toward sulfonylureas. , 1998, Circulation research.

[5]  R. Schwartz,et al.  Enhanced endothelin-mediated coronary vasoconstriction and attenuated basal nitric oxide activity in experimental hypercholesterolemia. , 1997, Circulation.

[6]  R. Schwartz,et al.  The effect of basic fibroblast growth factor on coronary vascular tone in experimental hypercholesterolemia in vivo and in vitro , 1997, Coronary artery disease.

[7]  A. Lerman,et al.  The role of endothelin in coronary atherosclerosis. , 1996, Mayo Clinic proceedings.

[8]  F. Behrendt,et al.  Pinacidil relaxes porcine and human coronary arteries by activating ATP-dependent potassium channels in smooth muscle cells. , 1995, The Journal of pharmacology and experimental therapeutics.

[9]  F. Sellke,et al.  Adenosine triphosphate-sensitive K+ channels mediate postcardioplegia coronary hyperemia. , 1995, The Journal of thoracic and cardiovascular surgery.

[10]  A. Terzic,et al.  Cardiac ATP-sensitive K+ channels: regulation by intracellular nucleotides and K+ channel-opening drugs. , 1995, The American journal of physiology.

[11]  L. Kuo,et al.  Adenosine potentiates flow-induced dilation of coronary arterioles by activating KATP channels in endothelium. , 1995, The American journal of physiology.

[12]  J. Dubois-Randé,et al.  Comparisons of the effects of nicorandil, pinacidil, nicardipine and nitroglycerin on coronary vessels in the conscious dog: role of the endothelium , 1995, British journal of pharmacology.

[13]  D. S. Weiss,et al.  Inhibition of adenosine-induced coronary vasodilation by block of large-conductance Ca(2+)-activated K+ channels. , 1994, The American journal of physiology.

[14]  R. Cohen,et al.  Nitric oxide directly activates calcium-dependent potassium channels in vascular smooth muscle , 1994, Nature.

[15]  D. Heistad,et al.  Impaired Relaxation of the Carotid Artery During Activation of ATP‐Sensitive Potassium Channels in Atherosclerotic Monkeys , 1994, Stroke.

[16]  T. M. Griffith,et al.  Modulation of vasodilatation to levcromakalim by hypoxia and EDRF in the rabbit isolated ear: a comparison with pinacidil, sodium nitroprusside and verapamil , 1993, British journal of pharmacology.

[17]  M. Kern,et al.  Coronary flow velocity dynamics in normal and diseased arteries. , 1993, The American journal of cardiology.

[18]  T. Takishima,et al.  The role of ATP-sensitive potassium channels in regulating coronary microcirculation. , 1993, Biorheology.

[19]  H. M. Payne,et al.  Validation of A Doppler Guide Wire for Intravascular Measurement of Coronary Artery Flow Velocity , 1992, Circulation.

[20]  T. Griffith,et al.  Collateral Perfusion: The Role of Endothelium‐Derived Relaxing Factor and Effects of Vasodilators , 1992, Journal of cardiovascular pharmacology.

[21]  Y. Nakaya,et al.  Endothelin blocks ATP-sensitive K+ channels and depolarizes smooth muscle cells of porcine coronary artery. , 1992, Japanese journal of pharmacology.

[22]  E. Shibata,et al.  A voltage‐dependent potassium current in rabbit coronary artery smooth muscle cells. , 1991, The Journal of physiology.

[23]  J. Daut,et al.  Hypoxic dilation of coronary arteries is mediated by ATP-sensitive potassium channels. , 1990, Science.

[24]  D. Holmes,et al.  Estimation of the effects of angioplasty on coronary stenosis using quantitative video angiography. , 1985, Catheterization and cardiovascular diagnosis.