Amino acids in rat neostriatum: Alteration by kainic acid lesion

[1]  N. M. Gelder.  Taurine, the compartmentalized metabolism of glutamic acid, and the epilepsies. , 1978 .

[2]  W. Nicklas,et al.  AMINO ACID METABOLISM IN GLIAL CELLS: HOMEOSTATIC REGULATION OF INTRA‐ and EXTRACELLULAR MILIEU BY C‐6 GLIOMA CELLS , 1978, Journal of neurochemistry.

[3]  J. Olney,et al.  The fate of synaptic receptors in the kainate-lesioned striatum , 1978, Brain Research.

[4]  P. Mcgeer,et al.  Kainate-induced degeneration of neostriatal neurons: dependency upon corticostriatal tract , 1978, Brain Research.

[5]  R. Hassler,et al.  Effect of frontal cortex ablation on striatal glutamic acid level in rat , 1977, Brain Research.

[6]  D. Rassin,et al.  Taurine in development. , 1977, Life sciences.

[7]  P. Mcgeer,et al.  A glutamatergic corticostriatal path? , 1977, Brain Research.

[8]  J. Coyle,et al.  Striatal lesions with kainic acid: neurochemical characteristics , 1977, Brain Research.

[9]  M. Norenberg,et al.  Glutamine synthetase: glial localization in brain , 1977, Science.

[10]  I. Divac,et al.  High affinity uptake of glutamate in terminals of corticostriatal axons , 1977, Nature.

[11]  P. Campochiaro,et al.  Clinical, neuropathologic and pharmacologic aspects of Huntington's disease: correlates with a new animal model. , 1977, Progress in neuro-psychopharmacology.

[12]  P. Mcgeer,et al.  Duplication of biochemical changes of Huntington's chorea by intrastriatal injections of glutamic and kainic acids , 1976, Nature.

[13]  J. Coyle,et al.  Lesion of striatal neurons with kainic acid provides a model for Huntington's chorea , 1976, Nature.

[14]  Hugh J. Spencer.  Antagonism of cortical excitation of striatal neurons by glutamic acid diethyl ester: Evidence for glutamic acid as an excitatory transmitter in the rat striatum , 1976, Brain Research.

[15]  F. Henn.  Neurotransmission and glial cells: A functional relationship? , 1976, Journal of neuroscience research.

[16]  J. Olney,et al.  CYSTEINE‐S‐SULFATE: BRAIN DAMAGING METABOLITE IN SULFITE OXIDASE DEFICIENCY1 , 1975, Journal of neuropathology and experimental neurology.

[17]  S. Berl,et al.  Metabolic compartmentation and neurotransmission : relation to brain structure and function , 1975 .

[18]  J. Olney,et al.  Kainic acid: a powerful neurotoxic analogue of glutamate. , 1974, Brain research.

[19]  J. H. Quastel,et al.  FATE OF l‐GLUTAMATE IN THE BRAIN , 1974, Journal of neurochemistry.

[20]  N. A. Buchwald,et al.  Caudate intracellular response to thalamic and cortical inputs. , 1973, Experimental neurology.

[21]  G. Johnston,et al.  HIGH AFFINITY UPTAKE OF TRANSMITTERS: STUDIES ON THE UPTAKE OF l‐ASPARTATE, GABA, l‐GLUTAMATE AND GLYCINE IN CAT SPINAL CORD , 1973, Journal of neurochemistry.

[22]  W. Nicklas,et al.  EFFECT OF VINBLASTINE AND COLCHICINE ON UPTAKE AND RELEASE OF PUTATIVE TRANSMITTERS BY SYNAPTOSOMES AND ON BRAIN ACTOMYOSIN‐LIKE PROTEIN , 1973, Journal of neurochemistry.

[23]  A. Patel,et al.  Metabolic Compartments in the Brain: Their Properties and Relation to Morphological Structures , 1973 .

[24]  W. Nicklas,et al.  COMPARTMENTATION OF CITRIC ACID CYCLE METABOLISM IN BRAIN: EFFECT OF AMINOOXYACETIC ACID, OUABAIN AND Ca2+ ON THE LABELLING OF GLUTAMATE, GLUTAMINE, ASPARTATE AND GABA BY [1‐14C]ACETATE, [U‐14C]GLUTAMATE AND [U‐14C] ASPARTATE 1 , 1970, Journal of neurochemistry.

[25]  D Garfinkel,et al.  A simulation study of the metabolism and compartmentation in brain of glutamate, aspartate, the Krebs cycle, and related metabolites. , 1966, The Journal of biological chemistry.

[26]  O. H. Lowry,et al.  Protein measurement with the Folin phenol reagent. , 1951, The Journal of biological chemistry.