Heterogeneous nuclear ribonucleoprotein C1/C2, MeCP1, and SWI/SNF form a chromatin remodeling complex at the beta-globin locus control region.
暂无分享,去创建一个
Milind C Mahajan | M. Mahajan | S. Weissman | R. Kingston | G. Narlikar | Robert E Kingston | Sherman M Weissman | Geeta J Narlikar | Gokul Boyapaty | Gokul Boyapaty
[1] B. Emerson,et al. A SWI/SNF–Related Chromatin Remodeling Complex, E-RC1, Is Required for Tissue-Specific Transcriptional Regulation by EKLF In Vitro , 1998, Cell.
[2] H. Erdjument-Bromage,et al. Tissue-specific and developmental stage-specific DNA binding by a mammalian SWI/SNF complex associated with human fetal-to-adult globin gene switching. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[3] Jeroen Krijgsveld,et al. GATA‐1 forms distinct activating and repressive complexes in erythroid cells , 2005, The EMBO journal.
[4] F. Grosveld,et al. The beta‐globin dominant control region: hypersensitive site 2. , 1990, The EMBO journal.
[5] J. Strouboulis,et al. Heterochromatin Effects on the Frequency and Duration of LCR-Mediated Gene Transcription , 1996, Cell.
[6] Félix Recillas-Targa,et al. Position-effect protection and enhancer blocking by the chicken β-globin insulator are separable activities , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[7] R. Aebersold,et al. Dynamic changes in transcription factor complexes during erythroid differentiation revealed by quantitative proteomics , 2004, Nature Structural &Molecular Biology.
[8] J. McDowell,et al. Essential role of NF-E2 in remodeling of chromatin structure and transcriptional activation of the epsilon-globin gene in vivo by 5' hypersensitive site 2 of the beta-globin locus control region , 1996, Molecular and cellular biology.
[9] M. Groudine,et al. The Locus Control Region Is Necessary for Gene Expression in the Human β-Globin Locus but Not the Maintenance of an Open Chromatin Structure in Erythroid Cells , 1998, Molecular and Cellular Biology.
[10] G. Stamatoyannopoulos,et al. Hypersensitive site 5 of the human beta locus control region functions as a chromatin insulator. , 1994, Blood.
[11] J. D. Engel,et al. Human β-Globin Locus Control Region HS5Contains CTCF- and Developmental Stage-Dependent Enhancer-BlockingActivity in ErythroidCells , 2003, Molecular and Cellular Biology.
[12] W. C. Forrester,et al. A deletion of the human beta-globin locus activation region causes a major alteration in chromatin structure and replication across the entire beta-globin locus. , 1990, Genes & development.
[13] T. Ley,et al. Targeted deletion of 5'HS2 of the murine beta-globin LCR reveals that it is not essential for proper regulation of the beta-globin locus. , 1995, Genes & development.
[14] J. Sharpe,et al. A single beta-globin locus control region element (5' hypersensitive site 2) is sufficient for developmental regulation of human globin genes in transgenic mice , 1992, Molecular and cellular biology.
[15] W Miller,et al. Locus control regions of mammalian beta-globin gene clusters: combining phylogenetic analyses and experimental results to gain functional insights. , 1997, Gene.
[16] T. Kimbrough,et al. Effect of deletion of 5'HS3 or 5'HS2 of the human beta-globin locus control region on the developmental regulation of globin gene expression in beta-globin locus yeast artificial chromosome transgenic mice. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[17] J. Stamatoyannopoulos,et al. NF‐E2 and GATA binding motifs are required for the formation of DNase I hypersensitive site 4 of the human beta‐globin locus control region. , 1995, The EMBO journal.
[18] H. Erdjument-Bromage,et al. An Ikaros-Containing Chromatin-Remodeling Complex in Adult-Type Erythroid Cells , 2000, Molecular and Cellular Biology.
[19] S. Orkin,et al. Purification of the human NF-E2 complex: cDNA cloning of the hematopoietic cell-specific subunit and evidence for an associated partner , 1993, Molecular and cellular biology.
[20] J. D. Engel,et al. Hypersensitive Site 2 Specifies a Unique Function within the Human β-Globin Locus Control Region To Stimulate Globin Gene Transcription , 1999, Molecular and Cellular Biology.
[21] A. Dean,et al. A Human Globin Enhancer Causes both Discrete and Widespread Alterations in Chromatin Structure , 2003, Molecular and Cellular Biology.
[22] W. Hörz,et al. ATP-dependent nucleosome remodeling. , 2002, Annual review of biochemistry.
[23] M. Groudine,et al. The β-Globin LCR Is Not Necessary for an Open Chromatin Structure or Developmentally Regulated Transcription of the Native Mouse β-Globin Locus , 1998 .
[24] Tony Kouzarides,et al. Histone H3 Lysine 4 Methylation Disrupts Binding of Nucleosome Remodeling and Deacetylase (NuRD) Repressor Complex* , 2002, The Journal of Biological Chemistry.
[25] Paul Tempst,et al. Erythroid transcription factor NF-E2 is a haematopoietic-specific basic–leucine zipper protein , 1993, Nature.
[26] Q. Feng,et al. The NuRD complex: linking histone modification to nucleosome remodeling. , 2003, Current topics in microbiology and immunology.
[27] A. West,et al. Recruitment of histone modifications by USF proteins at a vertebrate barrier element. , 2004, Molecular cell.
[28] P. Fraser,et al. Intergenic transcription and developmental remodeling of chromatin subdomains in the human beta-globin locus. , 2000, Molecular cell.
[29] B. Emerson,et al. NF-E2 disrupts chromatin structure at human beta-globin locus control region hypersensitive site 2 in vitro , 1996, Molecular and cellular biology.
[30] N. Proudfoot,et al. Definition of transcriptional promoters in the human beta globin locus control region. , 2002, Journal of molecular biology.
[31] M. Gerstein,et al. GATA-1 binding sites mapped in the β-globin locus by using mammalian chIp-chip analysis , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[32] A. Nienhuis,et al. Tandem AP-1-binding sites within the human beta-globin dominant control region function as an inducible enhancer in erythroid cells. , 1990, Genes & development.
[33] R. Rosli,et al. Requiem: a novel zinc finger gene essential for apoptosis in myeloid cells. , 1994, The Journal of biological chemistry.
[34] M. Groudine,et al. β-globin Gene Switching and DNase I Sensitivity of the Endogenous β-globin Locus in Mice Do Not Require the Locus Control Region , 2000 .
[35] R. Kiyama,et al. Enhancer activity of HS2 of the human beta-LCR is modulated by distance from the key nucleosome. , 2001, Nucleic acids research.
[36] Xiangdong Fang,et al. Locus control regions. , 2002, Blood.
[37] D. Tuan,et al. Transcription of the HS2 enhancer toward a cis-linked gene is independent of the orientation, position, and distance of the enhancer relative to the gene , 1997, Molecular and cellular biology.
[38] A. West,et al. Conserved CTCF Insulator Elements Flank the Mouse and Human β-Globin Loci , 2002, Molecular and Cellular Biology.
[39] J. Bieker,et al. Role of Erythroid Kruppel-like Factor in Human - to -Globin Gene Switching (*) , 1995, The Journal of Biological Chemistry.
[40] G. Blobel,et al. FOG‐1 recruits the NuRD repressor complex to mediate transcriptional repression by GATA‐1 , 2005, The EMBO journal.
[41] D. Tuan,et al. Transcription of the hypersensitive site HS2 enhancer in erythroid cells. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[42] M. Mahajan,et al. DNA-dependent adenosine triphosphatase (helicaselike transcription factor) activates beta-globin transcription in K562 cells. , 2002, Blood.
[43] P. Levings,et al. The human β‐globin locus control region , 2002 .
[44] G. Stamatoyannopoulos,et al. Genomic footprinting and sequencing of human beta-globin locus. Tissue specificity and cell line artifact. , 1994, The Journal of biological chemistry.