Sec22b Regulates Phagosomal Maturation and Antigen Crosspresentation by Dendritic Cells
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J. Enninga | I. Cebrian | S. Amigorena | M. Jouve | A. Savina | L. Moita | Géraldine Visentin | N. Blanchard | C. Moita | Alexandre Bobard | Catarina F Moita
[1] R. Tampé,et al. The transporter associated with antigen processing (TAP) is active in a post-ER compartment , 2010, Journal of Cell Science.
[2] C. Roy,et al. Legionella pneumophila Promotes Functional Interactions between Plasma Membrane Syntaxins and Sec22b , 2010, Traffic.
[3] P. Sansonetti,et al. Tracking the dynamic interplay between bacterial and host factors during pathogen‐induced vacuole rupture in real time , 2010, Cellular microbiology.
[4] S. Amigorena,et al. Intracellular mechanisms of antigen cross presentation in dendritic cells. , 2010, Current opinion in immunology.
[5] T. Elliott,et al. Calreticulin‐dependent recycling in the early secretory pathway mediates optimal peptide loading of MHC class I molecules , 2009, The EMBO journal.
[6] I. Wada,et al. Sec22b is a negative regulator of phagocytosis in macrophages. , 2009, Molecular biology of the cell.
[7] P. van Endert,et al. IRAP Identifies an Endosomal Compartment Required for MHC Class I Cross-Presentation , 2009, Science.
[8] N. Hacohen,et al. The small GTPase Rac2 controls phagosomal alkalinization and antigen crosspresentation selectively in CD8(+) dendritic cells. , 2009, Immunity.
[9] I. Coppens,et al. Host ER–parasitophorous vacuole interaction provides a route of entry for antigen cross-presentation in Toxoplasma gondii–infected dendritic cells , 2009, The Journal of experimental medicine.
[10] T. Südhof,et al. Membrane Fusion: Grappling with SNARE and SM Proteins , 2009, Science.
[11] G. Hämmerling,et al. A novel CD11c.DTR transgenic mouse for depletion of dendritic cells reveals their requirement for homeostatic proliferation of natural killer cells , 2008, European journal of immunology.
[12] N. Shastri,et al. Immunodominant, protective response to the parasite Toxoplasma gondii requires antigen processing in the endoplasmic reticulum , 2008, Nature Immunology.
[13] Li Wu,et al. Selective suicide of cross-presenting CD8+ dendritic cells by cytochrome c injection shows functional heterogeneity within this subset , 2008, Proceedings of the National Academy of Sciences.
[14] J. Slot,et al. Cryosectioning and immunolabeling , 2007, Nature Protocols.
[15] S. Amigorena,et al. Phagocytosis and antigen presentation in dendritic cells , 2007, Immunological reviews.
[16] P. Cresswell,et al. Selective loading of high-affinity peptides onto major histocompatibility complex class I molecules by the tapasin-ERp57 heterodimer , 2007, Nature Immunology.
[17] G. Raposo,et al. Rab27a regulates phagosomal pH and NADPH oxidase recruitment to dendritic cell phagosomes , 2007, Nature Cell Biology.
[18] P. Cresswell,et al. A role for the endoplasmic reticulum protein retrotranslocation machinery during crosspresentation by dendritic cells. , 2006, Immunity.
[19] I. Wada,et al. Involvement of syntaxin 18, an endoplasmic reticulum (ER)-localized SNARE protein, in ER-mediated phagocytosis. , 2006, Molecular biology of the cell.
[20] G. Raposo,et al. NOX2 Controls Phagosomal pH to Regulate Antigen Processing during Crosspresentation by Dendritic Cells , 2006, Cell.
[21] R. Isberg,et al. Targeting of host Rab GTPase function by the intravacuolar pathogen Legionella pneumophila. , 2006, Developmental cell.
[22] H. Hauri,et al. The ER-Golgi intermediate compartment (ERGIC): in search of its identity and function , 2006, Journal of Cell Science.
[23] Anne E Carpenter,et al. A Lentiviral RNAi Library for Human and Mouse Genes Applied to an Arrayed Viral High-Content Screen , 2006, Cell.
[24] N. Katunuma,et al. Processing of human cathepsin D is independent of its catalytic function and auto-activation: involvement of cathepsins L and B. , 2006, Journal of biochemistry.
[25] S. Grinstein,et al. Quantitative and Dynamic Assessment of the Contribution of the ER to Phagosome Formation , 2005, Cell.
[26] J. Rothman,et al. Differential use of endoplasmic reticulum membrane for phagocytosis in J774 macrophages. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[27] Henry C. Chang,et al. Differential Lysosomal Proteolysis in Antigen-Presenting Cells Determines Antigen Fate , 2005, Science.
[28] N. Shastri,et al. Class I Major Histocompatibility Complex Presentation of Antigens That Escape from the Parasitophorous Vacuole of Toxoplasma gondii , 2005, Infection and Immunity.
[29] R. Tampé,et al. Early phagosomes in dendritic cells form a cellular compartment sufficient for cross presentation of exogenous antigens , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[30] J. Davoust,et al. ER–phagosome fusion defines an MHC class I cross-presentation compartment in dendritic cells , 2003, Nature.
[31] Etienne Gagnon,et al. Phagosomes are competent organelles for antigen cross-presentation , 2003, Nature.
[32] J. Celli,et al. Brucella Evades Macrophage Killing via VirB-dependent Sustained Interactions with the Endoplasmic Reticulum , 2003, The Journal of experimental medicine.
[33] I. Mellman,et al. Activation of Lysosomal Function During Dendritic Cell Maturation , 2003, Science.
[34] Haruki Hasegawa,et al. Mammalian ykt6 is a neuronal SNARE targeted to a specialized compartment by its profilin-like amino terminal domain. , 2003, Molecular biology of the cell.
[35] Etienne Gagnon,et al. Endoplasmic Reticulum-Mediated Phagocytosis Is a Mechanism of Entry into Macrophages , 2002, Cell.
[36] Tao Zhang,et al. Ykt6 Forms a SNARE Complex with Syntaxin 5, GS28, and Bet1 and Participates in a Late Stage in Endoplasmic Reticulum-Golgi Transport* , 2001, The Journal of Biological Chemistry.
[37] J. Hay,et al. Subunit Structure of a Mammalian ER/Golgi SNARE Complex* , 2000, The Journal of Biological Chemistry.
[38] J. Rothman,et al. Compartmental specificity of cellular membrane fusion encoded in SNARE proteins , 2000, Nature.
[39] R. Scheller,et al. Syntaxin 18, a SNAP Receptor That Functions in the Endoplasmic Reticulum, Intermediate Compartment, and cis-Golgi Vesicle Trafficking* , 2000, The Journal of Biological Chemistry.
[40] W. Wickner,et al. Defining the functions of trans-SNARE pairs , 1998, Nature.
[41] T. Galli,et al. A novel tetanus neurotoxin-insensitive vesicle-associated membrane protein in SNARE complexes of the apical plasma membrane of epithelial cells. , 1998, Molecular biology of the cell.
[42] W. de Souza,et al. Relationship between the host cell endoplasmic reticulum and the parasitophorous vacuole containing Toxoplasma gondii. , 1997, Cell structure and function.
[43] Francesca Granucci,et al. Maturation Stages of Mouse Dendritic Cells in Growth Factor–dependent Long-Term Cultures , 1997, The Journal of experimental medicine.
[44] F. Gage,et al. In Vivo Gene Delivery and Stable Transduction of Nondividing Cells by a Lentiviral Vector , 1996, Science.
[45] S. Parmley,et al. Regulated secretion of multi-lamellar vesicles leads to formation of a tubulo-vesicular network in host-cell vacuoles occupied by Toxoplasma gondii. , 1995, Journal of cell science.
[46] M. Kovacsovics-Bankowski,et al. A phagosome-to-cytosol pathway for exogenous antigens presented on MHC class I molecules , 1995, Science.
[47] K Kumagai,et al. A simple method to eliminate the antigenicity of surface class I MHC molecules from the membrane of viable cells by acid treatment at pH 3. , 1987, Journal of immunological methods.
[48] P. Guermonprez,et al. Measuring pH, ROS production, maturation, and degradation in dendritic cell phagosomes using cytofluorometry-based assays. , 2010, Methods in molecular biology.
[49] T. Murata,et al. Analysis of Rab1 recruitment to vacuoles containing Legionella pneumophila. , 2005, Methods in enzymology.