Enhanced Biliary Excretion of Lithocholate-3-Sulfate by Ursodeoxycholate-3,7-disulfate Infusion in Eisai Hyperbilirubinemic Rat (EHBR)
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[1] Y. Sugiyama,et al. Evidence that interference with binding to hepatic cytosol binders can inhibit bile acid excretion in rats , 1996, Hepatology.
[2] D. Keppler,et al. Transport of glutathione, glucuronate, and sulfate conjugates by the MRP gene-encoded conjugate export pump. , 1996, Cancer research.
[3] P. Bosma,et al. Congenital Jaundice in Rats with a Mutation in a Multidrug Resistance-Associated Protein Gene , 1996, Science.
[4] Y. Sugiyama,et al. Expression of a putative ATP-binding cassette region, homologous to that in multidrug resistance-associated protein (MRP), is hereditarily defective in Eisai hyperbilirubinemic rats (EHBR) , 1996 .
[5] D. Keppler,et al. Expression of the MRP gene-encoded conjugate export pump in liver and its selective absence from the canalicular membrane in transport- deficient mutant hepatocytes , 1995, The Journal of cell biology.
[6] Y. Sugiyama,et al. Different biliary excretion systems for glucuronide and sulfate of a model compound; study using Eisai hyperbilirubinemic rats. , 1995, The Journal of pharmacology and experimental therapeutics.
[7] H. Takikawa,et al. Mechanisms of biliary excretion of lithocholate-3-sulfate in Eisai hyperbilirubinemic rats (EHBR) , 1995, Digestive Diseases and Sciences.
[8] Y. Sugiyama,et al. Binding of lithocholate and its glucuronide and sulfate by human serum albumin. , 1995, Biochimica et biophysica acta.
[9] H. Takikawa,et al. Effects of organic anions and bile acids on hepatocellular uptake of lithocholate-3-sulfate , 1995 .
[10] Y. Sugiyama,et al. Multiple systems for the biliary excretion of organic anions in rats: liquiritigenin conjugates as model compounds. , 1994, The Journal of pharmacology and experimental therapeutics.
[11] H. Takikawa,et al. Lithocholate-3-O-glucuronide-induced cholestasis , 1993, Digestive Diseases and Sciences.
[12] Y. Sugiyama,et al. Kinetic analysis of hepatobiliary transport of organic anions in Eisai hyperbilirubinemic mutant rats. , 1993, The Journal of pharmacology and experimental therapeutics.
[13] H. Takikawa,et al. Effects of ursodeoxycholate, its glucuronide and disulfate and beta-muricholate on biliary bicarbonate concentration and biliary lipid excretion. , 1992, Journal of hepatology.
[14] T. Mikami,et al. A new rat mutant with chronic conjugated hyperbilirubinemia and renal glomerular lesions. , 1992, Laboratory animal science.
[15] N. Kaplowitz,et al. Canalicular transport of reduced glutathione in normal and mutant Eisai hyperbilirubinemic rats. , 1992, The Journal of biological chemistry.
[16] Toshio Yamamoto,et al. Bilirubin diglucuronide transport by rat liver canalicular membrane vesicles: Stimulation by bicarbonate ion , 1991, Hepatology.
[17] T. Mikami,et al. Biliary excretion of bile acid conjugates in a hyperbilirubinemic mutant sprague‐dawley rat , 1991, Hepatology.
[18] K. Hayashi,et al. ATP-dependent transport for glucuronides in canalicular plasma membrane vesicles. , 1991, Biochemical and biophysical research communications.
[19] T. Ishikawa,et al. ATP-dependent primary active transport of cysteinyl leukotrienes across liver canalicular membrane. Role of the ATP-dependent transport system for glutathione S-conjugates. , 1990, The Journal of biological chemistry.
[20] K. Hayashi,et al. Mechanism of glutathione S-conjugate transport in canalicular and basolateral rat liver plasma membranes. , 1990, The Journal of biological chemistry.
[21] H. Takikawa,et al. Comparison of biliary excretion and metabolism of lithocholic acid and its sulfate and glucuronide conjugates in rats. , 1989, Biochimica et biophysica acta.
[22] M. Enserink,et al. Separate transport systems for biliary secretion of sulfated and unsulfated bile acids in the rat. , 1988, The Journal of clinical investigation.
[23] Y. Sugiyama,et al. Binding of bile acids by glutathione S-transferases from rat liver. , 1986, Journal of lipid research.
[24] M. Vore,et al. Taurocholate and steroid glucuronides: mutual protection against cholestasis in the isolated perfused rat liver. , 1986, The Journal of pharmacology and experimental therapeutics.
[25] W. Lamers,et al. Hereditary chronic conjugated hyperbilirubinemia in mutant rats caused by defective hepatic anion transport , 1985, Hepatology.
[26] D. Dhumeaux,et al. Biliary transport of cholephilic dyes: evidence for two different pathways. , 1977, The American journal of physiology.
[27] C. Kao,et al. Shared and separate pathways for biliary excretion of bilirubin and BSP in rats. , 1973, The American journal of physiology.
[28] Y. Sugiyama,et al. Multiple transport systems for organic anions across the bile canalicular membrane. , 1994, The Journal of pharmacology and experimental therapeutics.
[29] H. Takikawa,et al. Effects of organic anions and bile acids on biliary lipid excretion in hyperbilirubinemic mutant Sprague-Dawley rats. , 1993, Journal of hepatology.
[30] H. Takikawa,et al. Estradiol-17 beta-glucuronide-induced cholestasis. Effects of ursodeoxycholate-3-O-glucuronide and 3,7-disulfate. , 1993, Journal of hepatology.
[31] T. Uesugi,et al. Organic anion transport study in mutant rats with autosomal recessive conjugated hyperbilirubinemia. , 1991, Life sciences.
[32] 北村 庸雄. Defective ATP-dependent bile canalicular transport of organic anions in mutant (TR[-]) rats with conjugated hyperbilirubinemia , 1990 .