Assessment of Myocardial Viability with Positron Emission Tomography

[1]  M. Pfeffer,et al.  Ventricular Remodeling After Myocardial Infarction: Experimental Observations and Clinical Implications , 1990, Circulation.

[2]  R. Gropler,et al.  Comparison of carbon-11-acetate with fluorine-18-fluorodeoxyglucose for delineating viable myocardium by positron emission tomography. , 1993, Journal of the American College of Cardiology.

[3]  E. Erdmann,et al.  Predictive value of low dose dobutamine transesophageal echocardiography and fluorine-18 fluorodeoxyglucose positron emission tomography for recovery of regional left ventricular function after successful revascularization. , 1996, Journal of the American College of Cardiology.

[4]  T. Spinks,et al.  Increased uptake of 18F-fluorodeoxyglucose in postischemic myocardium of patients with exercise-induced angina. , 1986, Circulation.

[5]  M. Schwaiger,et al.  Clinical outcome of patients with advanced coronary artery disease after viability studies with positron emission tomography. , 1992, Journal of the American College of Cardiology.

[6]  F. Fazio,et al.  Assessing hibernating myocardium: an emerging cost-effectiveness issue , 1997, European Journal of Nuclear Medicine.

[7]  Diao Mf.  Predicting improved function after myocardial revascularization. , 1998 .

[8]  A. Buecker,et al.  Validation of an evaluation routine for left ventricular volumes, ejection fraction and wall motion from gated cardiac FDG PET: a comparison with cardiac magnetic resonance imaging , 2003, European Journal of Nuclear Medicine and Molecular Imaging.

[9]  Y. Yonekura,et al.  Metabolic activity in the areas of new fill-in after thallium-201 reinjection: comparison with positron emission tomography using fluorine-18-deoxyglucose. , 1991, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[10]  M. Phelps,et al.  Value of metabolic imaging with positron emission tomography for evaluating prognosis in patients with coronary artery disease and left ventricular dysfunction. , 1994, The American journal of cardiology.

[11]  A. Bol,et al.  Mechanisms of Chronic Regional Postischemic Dysfunction in Humans New Insights From the Study of Noninfarcted Collateral‐Dependent Myocardium , 1993, Circulation.

[12]  Jeroen J. Bax,et al.  Cardiac computed tomography: indications, applications, limitations, and training requirements: report of a Writing Group deployed by the Working Group Nuclear Cardiology and Cardiac CT of the European Society of Cardiology and the European Council of Nuclear Cardiology. , 2008, European heart journal.

[13]  M. Schwaiger,et al.  Relation of regional function, perfusion, and metabolism in patients with advanced coronary artery disease undergoing surgical revascularization. , 1994, Circulation.

[14]  H. Adachi,et al.  Left ventricular volume predicts postoperative course in patients with ischemic cardiomyopathy. , 1998, The Annals of thoracic surgery.

[15]  Jeroen J. Bax,et al.  Prediction of functional recovery after revascularization in patients with coronary artery disease and left ventricular dysfunction by gated FDG-PET. , 2006, Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology.

[16]  E. Fallen,et al.  Positron emission tomography and recovery following revascularization (PARR-1): the importance of scar and the development of a prediction rule for the degree of recovery of left ventricular function. , 2002, Journal of the American College of Cardiology.

[17]  M Grover-McKay,et al.  Detection of coronary artery disease with positron emission tomography and rubidium 82. , 1992, American heart journal.

[18]  Jeroen J. Bax,et al.  The extent of perfusion-F18-fluorodeoxyglucose positron emission tomography mismatch determines mortality in medically treated patients with chronic ischemic left ventricular dysfunction. , 2005, Journal of the American College of Cardiology.

[19]  W. Schaper,et al.  Hibernating myocardium: an incomplete adaptation to ischemia. , 1997, Circulation.

[20]  F. Sheehan,et al.  Myocyte degeneration and cell death in hibernating human myocardium. , 1996, Journal of the American College of Cardiology.

[21]  James W. Sayre,et al.  Long-Term Survival of Patients with Viable and Nonviable Aneurysms Assessed by 99mTc-MIBI SPECT and 18F-FDG PET: A Comparative Study of Medical and Surgical Treatment , 2008, Journal of Nuclear Medicine.

[22]  B. Nowak,et al.  Cardiac resynchronization therapyhomogenizes myocardial glucosemetabolism and perfusion in dilatedcardiomyopathy and left bundle branch block , 2003 .

[23]  G. Schulz,et al.  Prolonged myocardial hibernation exacerbates cardiomyocyte degeneration and impairs recovery of function after revascularization. , 1998, Journal of the American College of Cardiology.

[24]  R Härkönen,et al.  Myocardial viability: fluorine-18-deoxyglucose positron emission tomography in prediction of wall motion recovery after revascularization. , 1994, American heart journal.

[25]  A. Soricelli,et al.  Dobutamine echocardiography predicts improvement of hypoperfused dysfunctional myocardium after revascularization in patients with coronary artery disease. , 1995, Circulation.

[26]  U. Ruotsalainen,et al.  Euglycemic hyperinsulinemic clamp and oral glucose load in stimulating myocardial glucose utilization during positron emission tomography. , 1992, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[27]  M. Phelps,et al.  Quantitative relation between myocardial viability and improvement in heart failure symptoms after revascularization in patients with ischemic cardiomyopathy. , 1995, Circulation.

[28]  Jeroen J. Bax,et al.  PET myocardial glucose metabolism and perfusion imaging: Part 1-Guidelines for data acquisition and patient preparation. , 2003, Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology.

[29]  R M Whitlock,et al.  Left ventricular end-systolic volume as the major determinant of survival after recovery from myocardial infarction. , 1987, Circulation.

[30]  H. Adachi,et al.  Left ventricular end-systolic volume index in patients with ischemic cardiomyopathy predicts postoperative ventricular function. , 1995, The Annals of thoracic surgery.

[31]  W. J. Maclntyre,et al.  Metabolic Responses of Hibernating and Infarcted Myocardium to Revascularization: A Follow‐up Study of Regional Perfusion, Function, and Metabolism , 1992, Circulation.

[32]  M Schwaiger,et al.  Preoperative positron emission tomographic viability assessment and perioperative and postoperative risk in patients with advanced ischemic heart disease. , 1997, Journal of the American College of Cardiology.

[33]  B J Messmer,et al.  Recovery of regional left ventricular dysfunction after coronary revascularization. Impact of myocardial viability assessed by nuclear imaging and vessel patency at follow-up angiography. , 1996, Journal of the American College of Cardiology.

[34]  M Schwaiger,et al.  Reversibility of cardiac wall-motion abnormalities predicted by positron tomography. , 1986, The New England journal of medicine.

[35]  S L Bacharach,et al.  13N-ammonia myocardial blood flow and uptake: relation to functional outcome of asynergic regions after revascularization. , 1999, Journal of the American College of Cardiology.

[36]  J J Bax,et al.  Positron emission tomography using(18)F-fluoro-deoxyglucose and euglycaemic hyperinsulinaemic glucose clamp: optimal criteria for the prediction of recovery of post-ischaemic left ventricular dysfunction. Results from the European Community Concerted Action Multicenter study on use of(18)F-fluoro-de , 2001, European heart journal.

[37]  W. J. Maclntyre,et al.  Prognosis of Patients With Left Ventricular Dysfunction, With and Without Viable Myocardium After Myocardial Infarction: Relative Efficacy of Medical Therapy and Revascularization , 1994, Circulation.

[38]  O. Simonetti,et al.  The use of contrast-enhanced magnetic resonance imaging to identify reversible myocardial dysfunction. , 2000, The New England journal of medicine.

[39]  Y. Yonekura,et al.  Positron emission tomography using fluorine-18 deoxyglucose in evaluation of coronary artery bypass grafting. , 1989, The American journal of cardiology.

[40]  R. Russell,et al.  Cellular and molecular regulation of cardiac glucose transport , 2000, Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology.

[41]  S. Kaul,et al.  Quantitative Planar Rest‐Redistribution 201 Imaging in Detection of Myocardial Viability and Prediction of Improvement in Left Ventricular Function After Coronary Bypass Surgery in Patients With Severely Depressed Left Ventricular Function , 1993, Circulation.

[42]  N. Tamaki,et al.  Noninvasive assessment of myocardial viability by positron emission tomography with 11C acetate in patients with old myocardial infarction. Usefulness of low-dose dobutamine infusion. , 1996, Circulation.

[43]  R. deKemp,et al.  Delay in revascularization is associated with increased mortality rate in patients with severe left ventricular dysfunction and viable myocardium on fluorine 18-fluorodeoxyglucose positron emission tomography imaging. , 1998, Circulation.

[44]  M. Lauer,et al.  Functional status and quality of life in patients with heart failure undergoing coronary bypass surgery after assessment of myocardial viability. , 1999, Journal of the American College of Cardiology.

[45]  A. Lammertsma,et al.  Preoperative Prediction of the Outcome of Coronary Revascularization Using Positron Emission Tomography , 1992, Circulation.

[46]  O. Muzik,et al.  Myocardial blood flow, function, and metabolism in repetitive stunning. , 2000, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[47]  E. Voth,et al.  F-18-FDG uptake is a reliable predictory of functional recovery of akinetic but viable infarct regions as defined by magnetic resonance imaging before and after revascularization. , 2004, Magnetic resonance imaging.

[48]  A. Liedtke.  Alterations of carbohydrate and lipid metabolism in the acutely ischemic heart. , 1981, Progress in cardiovascular diseases.

[49]  U. Ruotsalainen,et al.  The value of quantitative analysis of glucose utilization in detection of myocardial viability by PET. , 1993, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[50]  Jeroen J. Bax,et al.  Prediction of functional recovery after revascularization in patients with chronic ischaemic left ventricular dysfunction: head-to-head comparison between 99mTc-sestamibi/18F-FDG DISA SPECT and 13N-ammonia/18F-FDG PET , 2006, European Journal of Nuclear Medicine and Molecular Imaging.

[51]  K. Gould,et al.  Identifying and Measuring Severity of Coronary Artery Stenosis: Quantitative Coronary Arteriography and Positron Emission Tomography , 1988, Circulation.

[52]  A. Lammertsma,et al.  A New Strategy for the Assessment of Viable Myocardium and Regional Myocardial Blood Flow Using 15O‐Water and Dynamic Positron Emission Tomography , 1992, Circulation.

[53]  A. Buecker,et al.  Assessment of reversible myocardial dysfunction in chronic ischaemic heart disease: comparison of contrast-enhanced cardiovascular magnetic resonance and a combined positron emission tomography-single photon emission computed tomography imaging protocol. , 2006, European heart journal.

[54]  Jeroen J. Bax,et al.  Hibernating myocardium: diagnosis and patient outcomes. , 2007, Current problems in cardiology.

[55]  J. Knuuti,et al.  EANM/ESC procedural guidelines for myocardial perfusion imaging in nuclear cardiology , 2005, European Journal of Nuclear Medicine and Molecular Imaging.

[56]  A. Bol,et al.  Delineation of myocardial viability with PET. , 1995, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[57]  H. Schelbert,et al.  Blood flow-metabolism imaging with positron emission tomography in patients with diabetes mellitus for the assessment of reversible left ventricular contractile dysfunction. , 1999, Journal of the American College of Cardiology.

[58]  M E Phelps,et al.  Compartment model for measuring myocardial oxygen consumption using [1-11C]acetate. , 1997, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[59]  E. Hoffman,et al.  Investigation of [18F]2-fluoro-2-deoxyglucose for the measure of myocardial glucose metabolism. , 1978, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[60]  B. Gerber,et al.  Time course of functional recovery after coronary artery bypass graft surgery in patients with chronic left ventricular ischemic dysfunction. , 2000, The American journal of cardiology.

[61]  H R Schelbert,et al.  Validation of [1-11C]acetate as a tracer for noninvasive assessment of oxidative metabolism with positron emission tomography in normal, ischemic, postischemic, and hyperemic canine myocardium. , 1990, Circulation.

[62]  U. Ruotsalainen,et al.  Enhancement of myocardial [fluorine-18]fluorodeoxyglucose uptake by a nicotinic acid derivative. , 1994, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[63]  Jeroen J. Bax,et al.  No difference in cardiac event-free survival between positron emission tomography-guided and single-photon emission computed tomography-guided patient management: a prospective, randomized comparison of patients with suspicion of jeopardized myocardium. , 2001, Journal of the American College of Cardiology.

[64]  D. Duboc,et al.  Myocardial viability assessment in ischemic cardiomyopathy: benefits of coronary revascularization. , 1994, The Annals of thoracic surgery.

[65]  B J Messmer,et al.  Effect of myocardial viability assessed by technetium-99m-sestamibi SPECT and fluorine-18-FDG PET on clinical outcome in coronary artery disease. , 1997, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[66]  T. Marwick,et al.  Prediction by postexercise fluoro-18 deoxyglucose positron emission tomography of improvement in exercise capacity after revascularization. , 1992, The American journal of cardiology.

[67]  T. Ruddy,et al.  F-18-fluorodeoxyglucose PET imaging alters clinical decision making in patients with impaired ventricular function. , 1997, The American journal of cardiology.

[68]  Y. Yonekura,et al.  Prediction of reversible ischemia after revascularization. Perfusion and metabolic studies with positron emission tomography. , 1995, Circulation.

[69]  P. Camici,et al.  Pathophysiology of chronic left ventricular dysfunction. New insights from the measurement of absolute myocardial blood flow and glucose utilization. , 1996, Circulation.

[70]  R. Gropler,et al.  Functional recovery after coronary revascularization for chronic coronary artery disease is dependent on maintenance of oxidative metabolism. , 1992, Journal of the American College of Cardiology.

[71]  R. Gropler,et al.  Superiority of C-11 acetate compared with F-18 fluorodeoxyglucose in predicting myocardial functional recovery by positron emission tomography in patients with acute myocardial infarction. , 1996, The American journal of cardiology.

[72]  Jeroen J. Bax,et al.  Sensitivity, specificity, and predictive accuracies of various noninvasive techniques for detecting hibernating myocardium. , 2001, Current problems in cardiology.

[73]  R. Gropler,et al.  Flow and metabolic determinants of myocardial viability assessed by positron-emission tomography. , 1993, Coronary artery disease.

[74]  Jeroen J. Bax,et al.  Imaging techniques for the assessment of myocardial hibernation. Report of a Study Group of the European Society of Cardiology. , 2004 .

[75]  S. Bergmann,et al.  Utility of positron emission tomography in predicting cardiac events and survival in patients with coronary artery disease and severe left ventricular dysfunction. , 2001, The American journal of cardiology.

[76]  Jeroen J. Bax,et al.  Optimal metabolic conditions during fluorine-18 fluorodeoxyglucose imaging; a comparative study using different protocols , 2005, European Journal of Nuclear Medicine.

[77]  J. Holden,et al.  Effect of nicotinic acid on exogenous myocardial glucose utilization. , 1995, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[78]  J. van den Hoff,et al.  Assessment of myocardial viability by use of 11C-acetate and positron emission tomography. Threshold criteria of reversible dysfunction. , 1997, Circulation.

[79]  M. Pasic,et al.  Improvement of severely reduced left ventricular function after surgical revascularization in patients with preoperative myocardial infarction. , 1992, European journal of cardio-thoracic surgery : official journal of the European Association for Cardio-thoracic Surgery.

[80]  André Lamy,et al.  F-18-fluorodeoxyglucose positron emission tomography imaging-assisted management of patients with severe left ventricular dysfunction and suspected coronary disease: a randomized, controlled trial (PARR-2). , 2007, Journal of the American College of Cardiology.

[81]  L. Shaw,et al.  Myocardial viability testing and impact of revascularization on prognosis in patients with coronary artery disease and left ventricular dysfunction: a meta-analysis. , 2002, Journal of the American College of Cardiology.

[82]  D. Berman,et al.  The art and science of predicting postrevascularization improvement in left ventricular (LV) function in patients with severely depressed LV function. , 2002, Journal of the American College of Cardiology.

[83]  F. Van de Werf,et al.  Assessment of myocardial viability in chronic coronary artery disease using technetium-99m sestamibi SPECT. Correlation with histologic and positron emission tomographic studies and functional follow-up. , 1997, Journal of the American College of Cardiology.

[84]  Jeroen J. Bax,et al.  Prevalence of myocardial viability assessed by single photon emission computed tomography in patients with chronic ischaemic left ventricular dysfunction , 2002, Heart.

[85]  V. Dilsizian,et al.  [18F]fluorodeoxyglucose single photon emission computed tomography: can it replace PET and thallium SPECT for the assessment of myocardial viability? , 1998, Circulation.