Single-cell RNA sequencing reveals the dynamics of hepatic non-parenchymal cells in autoprotection against acetaminophen-induced hepatotoxicity
暂无分享,去创建一个
Y. Zhan | Fanfan Zhou | Xiaohui Fan | Jingyang Qian | Xiaoyan Lu | Lingqi Yu | Anyao Li | Lidan Zhu | Jun Yan
[1] R. Vernal,et al. Neutrophil N1 and N2 Subsets and Their Possible Association with Periodontitis: A Scoping Review , 2022, International journal of molecular sciences.
[2] A. Bell,et al. Single-cell spatial transcriptomics reveals a dynamic control of metabolic zonation and liver regeneration by endothelial cell Wnt2 and Wnt9b , 2022, Cell reports. Medicine.
[3] Zuolin Wang,et al. A novel lineage of osteoprogenitor cells with dual epithelial and mesenchymal properties govern maxillofacial bone homeostasis and regeneration after MSFL , 2022, Cell Research.
[4] Aleksandra A. Kolodziejczyk,et al. The spatiotemporal program of zonal liver regeneration following acute injury. , 2022, Cell stem cell.
[5] P. Song,et al. Notch-Regulated c-Kit–Positive Liver Sinusoidal Endothelial Cells Contribute to Liver Zonation and Regeneration , 2022, Cellular and molecular gastroenterology and hepatology.
[6] Y. Tao,et al. Pharmacotherapies for Drug-Induced Liver Injury: A Current Literature Review , 2022, Frontiers in Pharmacology.
[7] E. Ballestar,et al. Tolerogenic Dendritic Cells in Autoimmunity and Inflammatory Diseases. , 2020, Trends in immunology.
[8] T. Horng,et al. Lipid Metabolism in Regulation of Macrophage Functions. , 2020, Trends in cell biology.
[9] Gaofeng Wang,et al. Neutrophil extracellular traps impair regeneration , 2020, bioRxiv.
[10] S. Möller,et al. An Attempt to Polarize Human Neutrophils Toward N1 and N2 Phenotypes in vitro , 2020, Frontiers in Immunology.
[11] W. Goessling,et al. Functional compensation precedes recovery of tissue mass following acute liver injury , 2020, Nature Communications.
[12] Jiandie D. Lin,et al. Landscape of Intercellular Crosstalk in Healthy and NASH Liver Revealed by Single-Cell Secretome Gene Analysis. , 2019, Molecular cell.
[13] R. Andrade,et al. Endoplasmic Reticulum Stress-induced Upregulation of STARD1 Promotes Acetaminophen-induced Acute Liver Failure. , 2019, Gastroenterology.
[14] D. Sancho,et al. Metabolic Control of Dendritic Cell Functions: Digesting Information , 2019, Front. Immunol..
[15] H. Gröne,et al. The Atypical Chemokine Receptor 2 Limits Progressive Fibrosis after Acute Ischemic Kidney Injury. , 2019, The American journal of pathology.
[16] Michael J. T. Stubbington,et al. Single-cell reconstruction of the early maternal–fetal interface in humans , 2018, Nature.
[17] T. Iwanaga,et al. Intestinal Lymphatic Endothelial Cells Produce R-Spondin3 , 2018, Scientific Reports.
[18] Ana E. Paiva,et al. Macrophage‐derived GPNMB accelerates skin healing , 2018, Experimental dermatology.
[19] F. Bazer,et al. C-C motif chemokine ligand 2 induces proliferation and prevents lipopolysaccharide-induced inflammatory responses in bovine mammary epithelial cells. , 2018, Journal of dairy science.
[20] H. Bi,et al. p53 attenuates acetaminophen-induced hepatotoxicity by regulating drug-metabolizing enzymes and transporter expression , 2018, Cell Death & Disease.
[21] T. Yoshimura. The chemokine MCP-1 (CCL2) in the host interaction with cancer: a foe or ally? , 2018, Cellular & Molecular Immunology.
[22] Radika Soysa,et al. Dendritic cells in hepatitis and liver transplantation , 2017, Liver transplantation : official publication of the American Association for the Study of Liver Diseases and the International Liver Transplantation Society.
[23] Michael J. T. Stubbington,et al. Single-cell transcriptomics to explore the immune system in health and disease , 2017, Science.
[24] T. Yoshimura. The production of monocyte chemoattractant protein-1 (MCP-1)/CCL2 in tumor microenvironments. , 2017, Cytokine.
[25] Hannah A. Pliner,et al. Reversed graph embedding resolves complex single-cell trajectories , 2017, Nature Methods.
[26] Cheng Huang,et al. Macrophage Phenotype in Liver Injury and Repair , 2017, Scandinavian journal of immunology.
[27] E. Morrisey,et al. Neutrophils promote alveolar epithelial regeneration by enhancing type II pneumocyte proliferation in a model of acid-induced acute lung injury. , 2016, American journal of physiology. Lung cellular and molecular physiology.
[28] G. Sutter,et al. CCL2 expression is mediated by type I IFN receptor and recruits NK and T cells to the lung during MVA infection , 2016, Journal of leukocyte biology.
[29] Courtney A Cates,et al. Temporal neutrophil polarization following myocardial infarction. , 2016, Cardiovascular research.
[30] Yujing Chi,et al. Gut‐derived lymphocyte recruitment to liver and induce liver injury in non‐alcoholic fatty liver disease mouse model , 2016, Journal of gastroenterology and hepatology.
[31] J. Connolly,et al. Metabolism Is Central to Tolerogenic Dendritic Cell Function , 2016, Mediators of inflammation.
[32] H. Augustin,et al. Endothelial RSPO3 Controls Vascular Stability and Pruning through Non-canonical WNT/Ca(2+)/NFAT Signaling. , 2016, Developmental cell.
[33] E. Pearce,et al. Immunometabolism governs dendritic cell and macrophage function , 2016, The Journal of experimental medicine.
[34] H. Okamoto,et al. The Angiocrine Factor Rspondin3 Is a Key Determinant of Liver Zonation. , 2015, Cell reports.
[35] Guozhong Tao,et al. Augmented Wnt signaling as a therapeutic tool to prevent ischemia/reperfusion injury in liver: Preclinical studies in a mouse model , 2015, Liver transplantation : official publication of the American Association for the Study of Liver Diseases and the International Liver Transplantation Society.
[36] N. Kitteringham,et al. Adaptation to acetaminophen exposure elicits major changes in expression and distribution of the hepatic proteome , 2015, Scientific Reports.
[37] Xiang Gao,et al. Natural Killer Cells-Produced IFN-γ Improves Bone Marrow-Derived Hepatocytes Regeneration in Murine Liver Failure Model , 2015, Scientific Reports.
[38] Michael D. Schneider,et al. Monocyte/Macrophage-derived IGF-1 Orchestrates Murine Skeletal Muscle Regeneration and Modulates Autocrine Polarization. , 2015, Molecular therapy : the journal of the American Society of Gene Therapy.
[39] S. Tian,et al. Macrophage polarization in kidney diseases. , 2015, Macrophage.
[40] R. Sun,et al. TIGIT safeguards liver regeneration through regulating natural killer cell‐hepatocyte crosstalk , 2014, Hepatology.
[41] Ronald N Hines,et al. Tolerance to acetaminophen hepatotoxicity in the mouse model of autoprotection is associated with induction of flavin-containing monooxygenase-3 (FMO3) in hepatocytes. , 2014, Toxicological sciences : an official journal of the Society of Toxicology.
[42] Y. Cho,et al. Characteristics of liver injury in drug-induced systemic hypersensitivity reactions. , 2013, Journal of the American Academy of Dermatology.
[43] L. DeLeve,et al. Liver sinusoidal endothelial cells and liver regeneration. , 2013, The Journal of clinical investigation.
[44] G. Graham,et al. Regulation of the immune and inflammatory responses by the 'atypical' chemokine receptor D6 , 2013, The Journal of pathology.
[45] Christopher D. Conrady,et al. IFN-α-driven CCL2 Production Recruits Inflammatory Monocytes to Infection Site in Mice , 2012, Mucosal Immunology.
[46] Guangchuang Yu,et al. clusterProfiler: an R package for comparing biological themes among gene clusters. , 2012, Omics : a journal of integrative biology.
[47] A. Malhotra,et al. Dendritic cell depletion exacerbates acetaminophen hepatotoxicity , 2011, Hepatology.
[48] Jinghang Zhang,et al. CCL2 recruits inflammatory monocytes to facilitate breast tumor metastasis , 2011, Nature.
[49] N. Rooijen,et al. NK cells suppress experimental cholestatic liver injury by an interleukin-6-mediated, Kupffer cell-dependent mechanism. , 2011, Journal of hepatology.
[50] B. Cieply,et al. Beta-catenin activation promotes liver regeneration after acetaminophen-induced injury. , 2009, The American journal of pathology.
[51] M. L. Ruiz,et al. Effect of repeated administration with subtoxic doses of acetaminophen to rats on enterohepatic recirculation of a subsequent toxic dose. , 2009, Biochemical Pharmacology.
[52] H. Augustin,et al. The Wnt signaling regulator R-spondin 3 promotes angioblast and vascular development , 2008, Development.
[53] A. Ben-Baruch,et al. The inflammatory chemokines CCL2 and CCL5 in breast cancer. , 2008, Cancer letters.
[54] J. Manautou,et al. Acquired resistance to acetaminophen hepatotoxicity is associated with induction of multidrug resistance-associated protein 4 (Mrp4) in proliferating hepatocytes. , 2008, Toxicological sciences : an official journal of the Society of Toxicology.
[55] S. Nagata,et al. Identification of Tim4 as a phosphatidylserine receptor , 2007, Nature.
[56] A. Dolganiuc,et al. Innate immune response and hepatic inflammation. , 2007, Seminars in liver disease.
[57] W. Jeong,et al. Activation of natural killer cells inhibits liver fibrosis: a novel strategy to treat liver fibrosis , 2007, Expert review of gastroenterology & hepatology.
[58] S. Sozzani,et al. Dendritic cell-endothelial cell cross-talk in angiogenesis. , 2007, Trends in immunology.
[59] A. P. Holt,et al. Attenuated liver fibrosis in the absence of B cells , 2006, Hepatology.
[60] M. Miyazaki,et al. Vascular endothelial growth factor and angiopoietins regulate sinusoidal regeneration and remodeling after partial hepatectomy in rats. , 2005, World journal of gastroenterology.
[61] S. Thorgeirsson,et al. Progenitor cells in liver regeneration: molecular responses controlling their activation and expansion , 2005, APMIS : acta pathologica, microbiologica, et immunologica Scandinavica.
[62] R. Sun,et al. Negative regulation of liver regeneration by innate immunity (natural killer cells/interferon-gamma). , 2004, Gastroenterology.
[63] R. DeMatteo,et al. Liver Dendritic Cells Are Less Immunogenic Than Spleen Dendritic Cells because of Differences in Subtype Composition1 , 2004, The Journal of Immunology.
[64] H. Young,et al. Expression of IFN-γ Upon Triggering of Activating Ly49D NK Receptors In Vitro and In Vivo: Costimulation with IL-12 or IL-18 Overrides Inhibitory Receptors 1 , 2003, The Journal of Immunology.
[65] T. Sato,et al. Sinusoidal endothelial cell proliferation and expression of angiopoietin/Tie family in regenerating rat liver. , 2001, Journal of hepatology.
[66] N. Tygstrup,et al. Autoprotection in acetaminophen intoxication in rats: the role of liver regeneration. , 2001, Pharmacology & toxicology.
[67] S. Govindarajan,et al. NK Cells Cause Liver Injury and Facilitate the Induction of T Cell-Mediated Immunity to a Viral Liver Infection1 , 2000, The Journal of Immunology.
[68] J. Waltenberger,et al. Regulation der Endothelfunktion und der Angiogenese durch den Vaskulären Endothelialen Wachstumsfaktor-A (VEGF-A) , 2000, Zeitschrift für Kardiologie.
[69] H. Okamura,et al. Regulation of interferon-γ production by IL-12 and IL-18 , 1998 .
[70] H. Mehendale,et al. Hepatocellular regeneration: key to thioacetamide autoprotection. , 1995, Pharmacology & toxicology.
[71] Harihara M. Mehendale,et al. Role of Hepatocellular Regeneration in CCl4 Autoprotection , 1991, Toxicologic pathology.
[72] W. Anderson,et al. Modification of ribosomal proteins during liver regeneration. , 1975, Biochemical and biophysical research communications.
[73] T. E. Webb,et al. Regulation of ribosome formation in regenerating rat liver. , 1972, European journal of biochemistry.
[74] Ahmed Enayetallah,et al. Analysis of changes in hepatic gene expression in a murine model of tolerance to acetaminophen hepatotoxicity (autoprotection). , 2014, Toxicology and applied pharmacology.
[75] H. Mehendale,et al. 2-Butoxyethanol autoprotection is due to resiliance of newly formed erythrocytes to hemolysis , 2009, Archives of Toxicology.
[76] D. Brenner,et al. NK and NKT cells in liver injury and fibrosis. , 2009, Clinical immunology.
[77] A. Larson. Acetaminophen hepatotoxicity. , 2007, Clinics in liver disease.
[78] William M. Lee,et al. LIVER FAILURE AND LIVER DISEASE LIVER FAILURE Acetaminophen-Induced Acute Liver Failure: Results of a United States Multicenter, Prospective Study , 2005 .
[79] H. Wild. [Conflict-centered group discussion in the industrial outpatient clinic]. , 1973, Zeitschrift fur arztliche Fortbildung.