sympathetic activity in hypertension Relation between QT interval variability and cardiac
暂无分享,去创建一个
P. Sanders | M. Baumert | E. Nalivaiko | D. Kaye | G. Lambert | E. Lambert | M. Schlaich | M. D. Elser | C. Sari
[1] M. Esler,et al. Sympathetic Nervous System Activity Is Associated With Obesity-Induced Subclinical Organ Damage in Young Adults , 2010, Hypertension.
[2] Ronald D Berger,et al. Intracardiac QT variability in patients with structural heart disease on class III antiarrhythmic drugs. , 2009, Journal of electrocardiology.
[3] Guido Grassi,et al. Assessment of Sympathetic Cardiovascular Drive in Human Hypertension: Achievements and Perspectives , 2009, Hypertension.
[4] B. Joung,et al. Autonomic nervous system activity measured directly and QT interval variability in normal and pacing-induced tachycardia heart failure dogs. , 2009, Journal of the American College of Cardiology.
[5] Mathias Baumert,et al. Short-term heart rate variability and cardiac norepinephrine spillover in patients with depression and panic disorder. , 2009, American journal of physiology. Heart and circulatory physiology.
[6] A. Sugiyama,et al. Beat-to-beat variability of repolarization differentiates the extent of torsadogenic potential of multi ion channel-blockers bepridil and amiodarone. , 2008, European journal of pharmacology.
[7] M. Baumert,et al. QT interval variability and cardiac norepinephrine spillover in patients with depression and panic disorder. , 2008, American journal of physiology. Heart and circulatory physiology.
[8] T. Masuyama,et al. Beat‐to‐Beat QT Interval Variability Is Primarily Affected by the Autonomic Nervous System , 2008, Annals of noninvasive electrocardiology : the official journal of the International Society for Holter and Noninvasive Electrocardiology, Inc.
[9] Derek Abbott,et al. Variability of QT interval duration in obstructive sleep apnea: an indicator of disease severity. , 2008, Sleep.
[10] A Varró,et al. Combined pharmacological block of IKr and IKs increases short‐term QT interval variability and provokes torsades de pointes , 2007, British journal of pharmacology.
[11] G. Melina,et al. Selective cardiac neuroadrenergic abnormalities in hypertensive patients with left ventricular hypertrophy. , 2007, Archives of medical research.
[12] R. Quaglione,et al. QT variability strongly predicts sudden cardiac death in asymptomatic subjects with mild or moderate left ventricular systolic dysfunction: a prospective study. , 2007, European heart journal.
[13] T. Opthof,et al. Ventricular repolarization: an overview of (patho)physiology, sympathetic effects and genetic aspects. , 2006, Progress in biophysics and molecular biology.
[14] P. Volders,et al. Beat-to-Beat variability of repolarization determines proarrhythmic outcome in dogs susceptible to drug-induced torsades de pointes. , 2005, Journal of the American College of Cardiology.
[15] A. Moss,et al. QT interval variability and spontaneous ventricular tachycardia or fibrillation in the Multicenter Automatic Defibrillator Implantation Trial (MADIT) II patients. , 2004, Journal of the American College of Cardiology.
[16] M. Esler,et al. Relation Between Cardiac Sympathetic Activity and Hypertensive Left Ventricular Hypertrophy , 2003, Circulation.
[17] A. Diedrich,et al. Modulation of QT Interval During Autonomic Nervous System Blockade in Humans , 2002, Circulation.
[18] R. Quaglione,et al. QT-interval variability and autonomic control in hypertensive subjects with left ventricular hypertrophy. , 2002, Clinical science.
[19] Richard Balon,et al. Increased QT variability in patients with panic disorder and depression , 2000, Psychiatry Research.
[20] Richard Balon,et al. Effect of Posture and Isoproterenol on Beat-to-Beat Heart Rate and QT Variability , 2000, Neuropsychobiology.
[21] F. Messerli. Hypertension and sudden cardiac death. , 1999, American journal of hypertension.
[22] D. Roden,et al. Abnormalities of the QT interval in primary disorders of autonomic failure. , 1998, American heart journal.
[23] I. Jonsdottir,et al. Influence of voluntary exercise on hypothalamic norepinephrine. , 1998, Journal of applied physiology.
[24] H. Calkins,et al. Beat-to-beat QT interval variability: novel evidence for repolarization lability in ischemic and nonischemic dilated cardiomyopathy. , 1997, Circulation.
[25] Richard P. Lewis,et al. ACC/AHA Guidelines for the Clinical Application of Echocardiography. A report of the American College of Cardiology/American Heart Association Task Force on Practice Guidelines (Committee on Clinical Application of Echocardiography). Developed in collaboration with the American Society of Echocardio , 1997, Circulation.
[26] J. Alpert,et al. ACC/AHA guidelines for the clinical application of echocardiography: executive summary. A report of the American College of Cardiology/American Heart Association Task Force on practice guidelines (Committee on Clinical Application of Echocardiography). Developed in collaboration with the American So , 1997, Journal of the American College of Cardiology.
[27] Capelle,et al. Dispersion of refractoriness in canine ventricular myocardium. Effects of sympathetic stimulation. , 1991, Circulation research.
[28] P. Schwartz,et al. Sympathetic modulation of the relation between ventricular repolarization and cycle length. , 1991, Circulation research.
[29] I. Meredith,et al. Overflow of catecholamine neurotransmitters to the circulation: source, fate, and functions. , 1990, Physiological reviews.
[30] G. Jennings,et al. Noradrenaline release and the pathophysiology of primary human hypertension. , 1989, American journal of hypertension.
[31] M. Franz,et al. Cycle length dependence of human action potential duration in vivo. Effects of single extrastimuli, sudden sustained rate acceleration and deceleration, and different steady-state frequencies. , 1988, The Journal of clinical investigation.
[32] N. Reichek,et al. Echocardiographic assessment of left ventricular hypertrophy: comparison to necropsy findings. , 1986, The American journal of cardiology.
[33] Jean-Pierre Valentin,et al. I(Ks) restricts excessive beat-to-beat variability of repolarization during beta-adrenergic receptor stimulation. , 2010, Journal of molecular and cellular cardiology.
[34] A. Malliani,et al. Quantifying electrocardiogram RT-RR variability interactions , 2006, Medical and Biological Engineering and Computing.