Roles of serum in innate immune responses of human leukocytes to synthetic lipopeptide

[1]  T. Fukuda,et al.  Disaggregation of lipopolysaccharide by albumin, hemoglobin or high-density lipoprotein, forming complexes that prime neutrophils for enhanced release of superoxide. , 2016, Pathogens and disease.

[2]  H. L. Wright,et al.  Human filarial Wolbachia lipopeptide directly activates human neutrophils in vitro , 2014, Parasite immunology.

[3]  R. Tapping,et al.  Human Lipopolysaccharide-binding Protein (LBP) and CD14 Independently Deliver Triacylated Lipoproteins to Toll-like Receptor 1 (TLR1) and TLR2 and Enhance Formation of the Ternary Signaling Complex* , 2013, The Journal of Biological Chemistry.

[4]  M. Oosting,et al.  Activation of innate host defense mechanisms by Borrelia. , 2010, European cytokine network.

[5]  A. Ulmer,et al.  Hemoglobin enhances the biological activity of synthetic and natural bacterial (endotoxic) virulence factors: a general principle. , 2008, Medicinal chemistry (Shariqah (United Arab Emirates)).

[6]  D. Webb,et al.  Oxidized Phospholipid Inhibition of Toll-like Receptor (TLR) Signaling Is Restricted to TLR2 and TLR4 , 2008, Journal of Biological Chemistry.

[7]  John R. Ledford,et al.  TLR2-Mediated Activation of Neutrophils in Response to German Cockroach Frass1 , 2008, The Journal of Immunology.

[8]  G. Jung,et al.  The activity of lipopeptide TLR2 agonists critically depends on the presence of solubilizers , 2007, European journal of immunology.

[9]  G. Jung,et al.  Physicochemical and biological analysis of synthetic bacterial lipopeptides : Validity of the concept of ’ endotoxic conformation ’ 1 , 2003 .

[10]  Hidehiko Sano,et al.  CD14 directly binds to triacylated lipopeptides and facilitates recognition of the lipopeptides by the receptor complex of Toll‐like receptors 2 and 1 without binding to the complex , 2006, Cellular microbiology.

[11]  M. Pabst,et al.  Local anesthetics inhibit priming of neutrophils by lipopolysaccharide for enhanced release of superoxide: suppression of cytochrome b558 expression by disparate mechanisms , 2005, Journal of leukocyte biology.

[12]  E. Moore,et al.  Structural organization of the neutrophil NADPH oxidase: phosphorylation and translocation during priming and activation , 2005, Journal of leukocyte biology.

[13]  S. Dower,et al.  The expression and roles of Toll‐like receptors in the biology of the human neutrophil , 2005, Journal of leukocyte biology.

[14]  H. Heine,et al.  Binding of lipopeptide to CD14 induces physical proximity of CD14, TLR2 and TLR1 , 2005, European journal of immunology.

[15]  P. Detmers,et al.  TLR2 Recognizes a Bacterial Lipopeptide through Direct Binding1 , 2004, The Journal of Immunology.

[16]  U. Göbel,et al.  Lipopolysaccharide Binding Protein Binds to Triacylated and Diacylated Lipopeptides and Mediates Innate Immune Responses , 2004 .

[17]  A. Bennaceur-Griscelli,et al.  From bloodjournal.hematologylibrary.org at PENN STATE UNIVERSITY on February 21, 2013. For personal use only. , 2002 .

[18]  S. Foster,et al.  Selective Roles for Toll-Like Receptor (TLR)2 and TLR4 in the Regulation of Neutrophil Activation and Life Span1 , 2003, The Journal of Immunology.

[19]  J. Weiss,et al.  An Essential Role for Albumin in the Interaction of Endotoxin with Lipopolysaccharide-binding Protein and sCD14 and Resultant Cell Activation* , 2002, The Journal of Biological Chemistry.

[20]  C. Harding,et al.  Mycobacterium tuberculosis 19-kDa Lipoprotein Promotes Neutrophil Activation1 , 2001, The Journal of Immunology.

[21]  J. Peterson,et al.  Neutrophil Activation by Bacterial Lipoprotein Versus Lipopolysaccharide: Differential Requirements for Serum and CD141 , 2000, The Journal of Immunology.

[22]  R. M. Wooten,et al.  Cutting edge: inflammatory signaling by Borrelia burgdorferi lipoproteins is mediated by toll-like receptor 2. , 1999, Journal of immunology.

[23]  J. Radolf,et al.  Activation of human monocytic cells by Borrelia burgdorferi and Treponema pallidum is facilitated by CD14 and correlates with surface exposure of spirochetal lipoproteins. , 1999, Journal of immunology.

[24]  K. Kusumoto,et al.  Neutrophils responded to immobilized lipopolysaccharide in the absence of lipopolysaccharide‐binding protein , 1998, Journal of leukocyte biology.

[25]  J. Radolf,et al.  Treponema pallidum and Borrelia burgdorferi lipoproteins and synthetic lipopeptides activate monocytic cells via a CD14-dependent pathway distinct from that used by lipopolysaccharide. , 1998, Journal of immunology.

[26]  R. M. Wooten,et al.  The role of CD14 in signaling mediated by outer membrane lipoproteins of Borrelia burgdorferi. , 1998, Journal of immunology.

[27]  F. DeLeo,et al.  Neutrophils exposed to bacterial lipopolysaccharide upregulate NADPH oxidase assembly. , 1998, The Journal of clinical investigation.

[28]  T. B. Morrison,et al.  Borrelia burgdorferi outer surface protein A (OspA) activates and primes human neutrophils. , 1997, Journal of immunology.

[29]  S. Wright,et al.  Catalytic Properties of Lipopolysaccharide (LPS) Binding Protein , 1996, The Journal of Biological Chemistry.

[30]  M. Pabst,et al.  An analogue of lipid A and LPS from Rhodobacter sphaeroides inhibits neutrophil responses to LPS by blocking receptor recognition of LPS and by depleting LPS‐binding protein in plasma , 1995, Journal of leukocyte biology.

[31]  A. Robidoux,et al.  E5531, a pure endotoxin antagonist of high potency. , 1995, Science.

[32]  M. Pabst,et al.  Lipopolysaccharides from periodontal pathogens prime neutrophils for enhanced respiratory burst: differential effect of a synthetic lipid a precursor IVA (LA-14-PP). , 1995, Journal of periodontal research.

[33]  M. Pabst,et al.  Neutrophil responses to lipopolysaccharide. Effect of adherence on triggering and priming of the respiratory burst. , 1991, Journal of immunology.

[34]  S. James,et al.  Human neutrophils release the Leu-8 lymph node homing receptor during cell activation. , 1990, Blood.

[35]  M. Pabst,et al.  Priming of neutrophils by lipopolysaccharide for enhanced release of superoxide. Requirement for plasma but not for tumor necrosis factor-alpha. , 1990, Journal of immunology.

[36]  S. Minakami,et al.  Monoclonal antibody 7D5 raised to cytochrome b558 of human neutrophils: immunocytochemical detection of the antigen in peripheral phagocytes of normal subjects, patients with chronic granulomatous disease, and their carrier mothers. , 1987, Blood.

[37]  G. Barlow,et al.  Studies on a lipopolysaccharide from Escherichia coli. Heterogeneity and mechanism of reversible inactivation by sodium deoxycholate. , 1969, Biochemistry.

[38]  M. Ikawa,et al.  STUDIES ON A LIPOPOLYSACCHARIDE FROM ESCHERICHIA COLI * , 1966, Annals of the New York Academy of Sciences.