Ingestion of partially hydrolyzed whey protein suppresses epicutaneous sensitization to β‐lactoglobulin in mice
暂无分享,去创建一个
[1] J. Roh,et al. Oral tolerance inhibits atopic dermatitis‐like type 2 inflammation in mice by modulating immune microenvironments , 2017, Allergy.
[2] J. Garssen,et al. A Specific Mixture of Fructo-Oligosaccharides and Bifidobacterium breve M-16V Facilitates Partial Non-Responsiveness to Whey Protein in Mice Orally Exposed to β-Lactoglobulin-Derived Peptides , 2017, Front. Immunol..
[3] C. Flohr,et al. Does atopic dermatitis cause food allergy? A systematic review , 2016, The Lancet.
[4] Y. Takeda,et al. Evaluation of the antigenicity of hydrolyzed cow's milk protein formulas using the mouse basophil activation test. , 2016, Toxicology letters.
[5] Y. Takeda,et al. Decreased expression of CD200R3 on mouse basophils as a novel marker for IgG1-mediated anaphylaxis , 2015, Immunity, inflammation and disease.
[6] S. Ziegler,et al. The role of basophils and proallergic cytokines, TSLP and IL-33, in cutaneously sensitized food allergy. , 2014, International immunology.
[7] A. Panchaud,et al. Low-Allergenic Hydrolyzed Egg Induces Oral Tolerance in Mice , 2014, International Archives of Allergy and Immunology.
[8] M. Kubo,et al. Exposure to food allergens through inflamed skin promotes intestinal food allergy through the thymic stromal lymphopoietin-basophil axis. , 2014, The Journal of allergy and clinical immunology.
[9] Kirsten Beyer,et al. A global survey of changing patterns of food allergy burden in children , 2013, The World Allergy Organization journal.
[10] J. Garssen,et al. Oral treatment with β‐lactoglobulin peptides prevents clinical symptoms in a mouse model for cow's milk allergy , 2013, Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology.
[11] R. Geha,et al. Epicutaneous sensitization results in IgE-dependent intestinal mast cell expansion and food-induced anaphylaxis. , 2013, The Journal of allergy and clinical immunology.
[12] G. Lack. Update on risk factors for food allergy. , 2012, The Journal of allergy and clinical immunology.
[13] S. Ziegler,et al. Langerhans cells are critical in epicutaneous sensitization with protein antigen via thymic stromal lymphopoietin receptor signaling. , 2012, The Journal of allergy and clinical immunology.
[14] J. Garssen,et al. Oral tolerance induction by partially hydrolyzed whey protein in mice is associated with enhanced numbers of Foxp3+ regulatory T‐cells in the mesenteric lymph nodes , 2011, Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology.
[15] J. Wal,et al. Oral tolerance and Treg cells are induced in BALB/c mice after gavage with bovine β‐lactoglobulin , 2011, Allergy.
[16] H. Weiner. Oral tolerance: immune mechanisms and the generation of Th3-type TGF-beta-secreting regulatory cells. , 2001, Microbes and infection.
[17] T. Chatila,et al. IgE-mediated systemic anaphylaxis and impaired tolerance to food antigens in mice with enhanced IL-4 receptor signaling. , 2011, The Journal of allergy and clinical immunology.
[18] R. Cardona,et al. Repeated episodes of anaphylaxis after the first consumption of egg. , 2011, Allergologia et immunopathologia.
[19] S. Ziegler,et al. Mechanical injury polarizes skin dendritic cells to elicit a T(H)2 response by inducing cutaneous thymic stromal lymphopoietin expression. , 2010, The Journal of allergy and clinical immunology.
[20] J. Garssen,et al. Acute allergic skin response as a new tool to evaluate the allergenicity of whey hydrolysates in a mouse model of orally induced cow’s milk allergy , 2009, Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology.
[21] M. Torrero,et al. CD200R surface expression as a marker of murine basophil activation , 2009, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.
[22] R. Geha,et al. IL-21R is essential for epicutaneous sensitization and allergic skin inflammation in humans and mice. , 2008, The Journal of clinical investigation.
[23] A. Burks,et al. Oral tolerance, food allergy, and immunotherapy: implications for future treatment. , 2008, The Journal of allergy and clinical immunology.
[24] F. Finkelman. Anaphylaxis: lessons from mouse models. , 2007, The Journal of allergy and clinical immunology.
[25] M. Jordana,et al. Th2 Differentiation in Distinct Lymph Nodes Influences the Site of Mucosal Th2 Immune-Inflammatory Responses1 , 2007, The Journal of Immunology.
[26] H. Sampson,et al. Role of TLR4 in allergic sensitization to food proteins in mice , 2006, Allergy.
[27] B. Niggemann,et al. Sensitizing Capacity and Residual Allergenicity of Hydrolyzed Cow’s Milk Formulae: Results from a Murine Model , 2001, International Archives of Allergy and Immunology.
[28] A. Pfeifer,et al. Induction of systemic immunologic tolerance to beta-lactoglobulin by oral administration of a whey protein hydrolysate. , 1997, The Journal of allergy and clinical immunology.
[29] Li-Fang Wang,et al. Epicutaneous exposure of protein antigen induces a predominant Th2-like response with high IgE production in mice. , 1996, Journal of immunology.
[30] H. Weiner,et al. Peripheral deletion of antigen-reactive T cells in oral tolerance , 1995, Nature.
[31] P. Askenase,et al. Transforming growth factor-beta 1 inhibits murine immediate and delayed type hypersensitivity. , 1992, Journal of immunology.
[32] S. Yoshida. Isolation of β-Lactogobulin and α-Lactalbumin by Gel Filtration Using Sephacryl S-200 and Purification by Diethylaminoethyl Ion-Exchange Chromatography , 1990 .
[33] M. Kubo,et al. Basophil‐derived IL‐4 promotes epicutaneous antigen sensitization concomitant with the development of food allergy , 2018, The Journal of allergy and clinical immunology.
[34] P. Hener,et al. TSLP produced by keratinocytes promotes allergen sensitization through skin and thereby triggers atopic march in mice. , 2013, The Journal of investigative dermatology.
[35] J. Hourihane. Peanut allergy. , 2011, Pediatric clinics of North America.