ApoE-Isoform-Dependent SARS-CoV-2 Neurotropism and Cellular Response
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Qi Cui | Guihua Sun | Yanhong Shi | Jinhui Wang | Xianwei Chen | V. Arumugaswami | G. Garcia | E. Tian | Cheng Wang | Mingzi Zhang
[1] S. Goldman,et al. Cell‐Based Therapy for Canavan Disease Using Human iPSC‐Derived NPCs and OPCs , 2020, Advanced science.
[2] Silva Kasela,et al. Identification of Required Host Factors for SARS-CoV-2 Infection in Human Cells , 2020, Cell.
[3] A. Helenius,et al. Neuropilin-1 facilitates SARS-CoV-2 cell entry and infectivity , 2020, Science.
[4] Li Li,et al. When glia meet induced pluripotent stem cells (iPSCs) , 2020, Molecular and Cellular Neuroscience.
[5] Madeline A. Lancaster,et al. SARS-CoV-2 Infects the Brain Choroid Plexus and Disrupts the Blood-CSF Barrier in Human Brain Organoids , 2020, Cell Stem Cell.
[6] Jay Gopalakrishnan,et al. SARS‐CoV‐2 targets neurons of 3D human brain organoids , 2020, The EMBO journal.
[7] X. Mao,et al. iPSCs-Derived Platform: A Feasible Tool for Probing the Neurotropism of SARS-CoV-2 , 2020, ACS chemical neuroscience.
[8] J. Chan,et al. SARS-CoV-2 infects human neural progenitor cells and brain organoids , 2020, Cell Research.
[9] Catherine Z. Chen,et al. Human Pluripotent Stem Cell-Derived Neural Cells and Brain Organoids Reveal SARS-CoV-2 Neurotropism , 2020, bioRxiv.
[10] C. Svendsen,et al. Human iPSC-Derived Cardiomyocytes Are Susceptible to SARS-CoV-2 Infection , 2020, Cell Reports Medicine.
[11] L. Smirnova,et al. Infectability of human BrainSphere neurons suggests neurotropism of SARS-CoV-2. , 2020, ALTEX.
[12] S. Farhadian,et al. Neuroinvasion of SARS-CoV-2 in human and mouse brain , 2020, bioRxiv.
[13] P. Kane,et al. Regulation of V-ATPase Activity and Organelle pH by Phosphatidylinositol Phosphate Lipids , 2020, Frontiers in Cell and Developmental Biology.
[14] Duc-Huy T. Nguyen,et al. A Human Pluripotent Stem Cell-based Platform to Study SARS-CoV-2 Tropism and Model Virus Infection in Human Cells and Organoids , 2020, Cell Stem Cell.
[15] K. Blennow,et al. Neurochemical evidence of astrocytic and neuronal injury commonly found in COVID-19 , 2020, Neurology.
[16] Pardis C Sabeti,et al. Neuropathological Features of Covid-19 , 2020, The New England journal of medicine.
[17] S. Chanda,et al. Sofosbuvir protects human brain organoids against SARS-CoV-2 , 2020, bioRxiv.
[18] Bin Zhang,et al. CD49f Is a Novel Marker of Functional and Reactive Human iPSC-Derived Astrocytes , 2020, Neuron.
[19] Victor G. Puelles,et al. Multiorgan and Renal Tropism of SARS-CoV-2 , 2020, The New England journal of medicine.
[20] Cezmi A Akdis,et al. Immune response to SARS‐CoV‐2 and mechanisms of immunopathological changes in COVID‐19 , 2020, Allergy.
[21] D. Melzer,et al. APOE e4 Genotype Predicts Severe COVID-19 in the UK Biobank Community Cohort , 2020, medRxiv.
[22] Marco Tullio Liuzza,et al. More than smell – COVID-19 is associated with severe impairment of smell, taste, and chemesthesis , 2020, medRxiv.
[23] J. Sejvar,et al. Neurological associations of COVID-19 , 2020, The Lancet Neurology.
[24] M. Fowkes,et al. Central nervous system involvement by severe acute respiratory syndrome coronavirus‐2 (SARS‐CoV‐2) , 2020, Journal of medical virology.
[25] S. Kremer,et al. Neurologic Features in Severe SARS-CoV-2 Infection , 2020, The New England journal of medicine.
[26] L. Mao,et al. Neurologic Manifestations of Hospitalized Patients With Coronavirus Disease 2019 in Wuhan, China. , 2020, JAMA neurology.
[27] Q. Ye,et al. The pathogenesis and treatment of the `Cytokine Storm' in COVID-19 , 2020, Journal of Infection.
[28] R. Carlier,et al. Olfactory and gustatory dysfunctions as a clinical presentation of mild-to-moderate forms of the coronavirus disease (COVID-19): a multicenter European study , 2020, European Archives of Oto-Rhino-Laryngology.
[29] N. Enomoto,et al. A first case of meningitis/encephalitis associated with SARS-Coronavirus-2 , 2020, International Journal of Infectious Diseases.
[30] Giuliano Rizzardini,et al. Self-reported Olfactory and Taste Disorders in Patients With Severe Acute Respiratory Coronavirus 2 Infection: A Cross-sectional Study , 2020, Clinical Infectious Diseases: An Official Publication of the Infectious Diseases Society of America.
[31] F. Gage,et al. Modeling Human Cytomegalovirus-Induced Microcephaly in Human iPSC-Derived Brain Organoids , 2020, Cell reports. Medicine.
[32] Xin Zhou,et al. Risk Factors Associated With Acute Respiratory Distress Syndrome and Death in Patients With Coronavirus Disease 2019 Pneumonia in Wuhan, China , 2020, The Journal of Emergency Medicine.
[33] Catherine M. Brown,et al. First 12 patients with coronavirus disease 2019 (COVID-19) in the United States , 2020, medRxiv.
[34] G. Herrler,et al. SARS-CoV-2 Cell Entry Depends on ACE2 and TMPRSS2 and Is Blocked by a Clinically Proven Protease Inhibitor , 2020, Cell.
[35] A. M. Leontovich,et al. The species Severe acute respiratory syndrome-related coronavirus: classifying 2019-nCoV and naming it SARS-CoV-2 , 2020, Nature Microbiology.
[36] E. Holmes,et al. A new coronavirus associated with human respiratory disease in China , 2020, Nature.
[37] G. Gao,et al. A Novel Coronavirus from Patients with Pneumonia in China, 2019 , 2020, The New England journal of medicine.
[38] R. Zorec,et al. Astrocytes in Flavivirus Infections , 2019, International journal of molecular sciences.
[39] Neville E. Sanjana,et al. GFAP Mutations in Astrocytes Impair Oligodendrocyte Progenitor Proliferation and Myelination in an hiPSC Model of Alexander Disease. , 2018, Cell stem cell.
[40] L. Tsai,et al. APOE4 Causes Widespread Molecular and Cellular Alterations Associated with Alzheimer’s Disease Phenotypes in Human iPSC-Derived Brain Cell Types , 2018, Neuron.
[41] Elizabeta Gjoneska,et al. APOE4 Causes Widespread Molecular and Cellular Alterations Associated with Alzheimer’s Disease Phenotypes in Human iPSC-Derived Brain Cell Types , 2018, Neuron.
[42] W. Banks,et al. Neuroimmune Axes of the Blood–Brain Barriers and Blood–Brain Interfaces: Bases for Physiological Regulation, Disease States, and Pharmacological Interventions , 2018, Pharmacological Reviews.
[43] A. Fagan,et al. ApoE4 markedly exacerbates tau-mediated neurodegeneration in a mouse model of tauopathy , 2017, Nature.
[44] Shinya Yamanaka,et al. Induced pluripotent stem cell technology: a decade of progress , 2016, Nature Reviews Drug Discovery.
[45] David W. Nauen,et al. Brain-Region-Specific Organoids Using Mini-bioreactors for Modeling ZIKV Exposure , 2016, Cell.
[46] R. Jaenisch,et al. Induced Pluripotent Stem Cells Meet Genome Editing. , 2016, Cell stem cell.
[47] William A. Lee,et al. Therapeutic efficacy of the small molecule GS-5734 against Ebola virus in rhesus monkeys , 2016, Nature.
[48] Jürgen Winkler,et al. Directly Reprogrammed Human Neurons Retain Aging-Associated Transcriptomic Signatures and Reveal Age-Related Nucleocytoplasmic Defects. , 2015, Cell stem cell.
[49] Elsa Vera,et al. Programming and Reprogramming Cellular Age in the Era of Induced Pluripotency. , 2015, Cell stem cell.
[50] David A. Scott,et al. Double Nicking by RNA-Guided CRISPR Cas9 for Enhanced Genome Editing Specificity , 2013, Cell.
[51] Madeline A. Lancaster,et al. Cerebral organoids model human brain development and microcephaly , 2013, Nature.
[52] Huaxi Xu,et al. Apolipoprotein E and Alzheimer disease: risk, mechanisms and therapy , 2013, Nature Reviews Neurology.
[53] F. Gage,et al. Induced pluripotent stem cells (iPSCs) and neurological disease modeling: progress and promises. , 2011, Human molecular genetics.
[54] R. Tanzi,et al. Thirty years of Alzheimer's disease genetics: the implications of systematic meta-analyses , 2008, Nature Reviews Neuroscience.
[55] M. Katze,et al. Genomic Analysis Reveals Age-Dependent Innate Immune Responses to Severe Acute Respiratory Syndrome Coronavirus , 2008, Journal of Virology.
[56] Jeff E. Mold,et al. Apolipoprotein (apo) E4 enhances HIV-1 cell entry in vitro, and the APOE ε4/ε4 genotype accelerates HIV disease progression , 2008, Proceedings of the National Academy of Sciences.
[57] Shulan Tian,et al. Induced Pluripotent Stem Cell Lines Derived from Human Somatic Cells , 2007, Science.
[58] T. Ichisaka,et al. Induction of Pluripotent Stem Cells from Adult Human Fibroblasts by Defined Factors , 2007, Cell.
[59] J. Burgos,et al. Effect of Apolipoprotein E on the Cerebral Load of Latent Herpes Simplex Virus Type 1 DNA , 2006, Journal of Virology.
[60] B. Hyman,et al. Endocytic Pathway Abnormalities Precede Amyloid β Deposition in Sporadic Alzheimer’s Disease and Down Syndrome , 2000 .
[61] J. Haines,et al. Effects of Age, Sex, and Ethnicity on the Association Between Apolipoprotein E Genotype and Alzheimer Disease: A Meta-analysis , 1997 .
[62] Li Li,et al. Modeling neurological diseases using iPSC-derived neural cells , 2017, Cell and Tissue Research.
[63] F. Gage,et al. Orphan nuclear receptor TLX activates Wnt/β-catenin signalling to stimulate neural stem cell proliferation and self-renewal , 2010, Nature Cell Biology.
[64] F. Gage,et al. Expression and function of orphan nuclear receptor TLX in adult neural stem cells , 2004, Nature.