Synaptic vesicle mobility and presynaptic F-actin are disrupted in a N-ethylmaleimide-sensitive factor allele of Drosophila.
暂无分享,去创建一个
[1] J. Henley,et al. Novel putative targets of N‐ethylmaleimide sensitive fusion protein (NSF) and α/β soluble NSF attachment proteins (SNAPs) include the Pak‐binding nucleotide exchange factor βPIX , 2006 .
[2] T. Uemura,et al. Lim kinase regulates the development of olfactory and neuromuscular synapses. , 2006, Developmental biology.
[3] Bryan A Stewart,et al. Disruption of synaptic development and ultrastructure by Drosophila NSF2 alleles , 2005, The Journal of comparative neurology.
[4] Toshiro Aigaki,et al. A Genetic Screen for Suppressors of Drosophila NSF2 Neuromuscular Junction Overgrowth , 2005, Genetics.
[5] P. Greengard,et al. Structural Domains Involved in the Regulation of Transmitter Release by Synapsins , 2005, The Journal of Neuroscience.
[6] M. Heisenberg,et al. Flies lacking all synapsins are unexpectedly healthy but are impaired in complex behaviour , 2004, The European journal of neuroscience.
[7] P. Verstreken,et al. Dap160/Intersectin Acts as a Stabilizing Scaffold Required for Synaptic Development and Vesicle Endocytosis , 2004, Neuron.
[8] W. Betz,et al. Effects of wortmannin and latrunculin A on slow endocytosis at the frog neuromuscular junction , 2004, The Journal of physiology.
[9] L. Lagnado,et al. High Mobility of Vesicles Supports Continuous Exocytosis at a Ribbon Synapse , 2004, Current Biology.
[10] Sunil Q. Mehta,et al. Synaptojanin Is Recruited by Endophilin to Promote Synaptic Vesicle Uncoating , 2003, Neuron.
[11] T. Uemura,et al. Control of Growth Cone Motility and Morphology by LIM Kinase and Slingshot via Phosphorylation and Dephosphorylation of Cofilin , 2003, The Journal of Neuroscience.
[12] T. A. Ryan,et al. Actin has a molecular scaffolding, not propulsive, role in presynaptic function , 2003, Nature Neuroscience.
[13] E. Neher,et al. Involvement of Actin Polymerization in Vesicle Recruitment at the Calyx of Held Synapse , 2003, The Journal of Neuroscience.
[14] Gabriel Fenteany,et al. Small-molecule inhibitors of actin dynamics and cell motility. , 2003, Current topics in medicinal chemistry.
[15] L. Brodin,et al. Impaired recycling of synaptic vesicles after acute perturbation of the presynaptic actin cytoskeleton , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[16] M. Bretscher,et al. Cell polarity and locomotion, as well as endocytosis, depend on NSF. , 2002, Development.
[17] Kendal Broadie,et al. Living synaptic vesicle marker: Synaptotagmin‐GFP , 2002, Genesis.
[18] R. Newman,et al. Sequential SNARE disassembly and GATE-16–GOS-28 complex assembly mediated by distinct NSF activities drives Golgi membrane fusion , 2002, The Journal of cell biology.
[19] Bryan A Stewart,et al. Dominant-negative NSF2 disrupts the structure and function of Drosophila neuromuscular synapses. , 2002, Journal of neurobiology.
[20] I. Meinertzhagen,et al. Endophilin Mutations Block Clathrin-Mediated Endocytosis but Not Neurotransmitter Release , 2002, Cell.
[21] Liaoyuan A. Hu,et al. Binding of the β2 Adrenergic Receptor toN-Ethylmaleimide-sensitive Factor Regulates Receptor Recycling* , 2001, The Journal of Biological Chemistry.
[22] W. Trimble,et al. SNARE-dependent signaling at the Drosophila wing margin. , 2001, Developmental biology.
[23] J. A. Golby,et al. Partitioning of N-ethylmaleimide-sensitive fusion (NSF) protein function in Drosophila melanogaster: dNSF1 is required in the nervous system, and dNSF2 is required in mesoderm. , 2001, Genetics.
[24] W. Trimble,et al. SNARE proteins contribute to calcium cooperativity of synaptic transmission. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[25] S. Han,et al. Identification of Rab6 as an N-ethylmaleimide-sensitive fusion protein-binding protein. , 2000, The Biochemical journal.
[26] C. Guatimosim,et al. Two Endocytic Recycling Routes Selectively Fill Two Vesicle Pools in Frog Motor Nerve Terminals , 2000, Neuron.
[27] Y. Goda,et al. Actin-Dependent Regulation of Neurotransmitter Release at Central Synapses , 2000, Neuron.
[28] A. Dunaevsky,et al. F-Actin Is Concentrated in Nonrelease Domains at Frog Neuromuscular Junctions , 2000, The Journal of Neuroscience.
[29] R. S. Wilkinson,et al. Disruption of actin impedes transmitter release in snake motor terminals , 2000, The Journal of physiology.
[30] G. Augustine,et al. The actin cytoskeleton and neurotransmitter release: an overview. , 2000, Biochimie.
[31] M. Ramaswami,et al. Synaptic Localization and Restricted Diffusion of a Drosophila Neuronal Synaptobrevin - Green Fluorescent Protein Chimera in Vivo , 2000, Journal of neurogenetics.
[32] F. Braet,et al. New anti‐actin drugs in the study of the organization and function of the actin cytoskeleton , 1999, Microscopy research and technique.
[33] W. Almers,et al. Endocytic vesicles move at the tips of actin tails in cultured mast cells , 1999, Nature Cell Biology.
[34] R. Lefkowitz,et al. Identification of NSF as a β-Arrestin1-binding Protein , 1999, The Journal of Biological Chemistry.
[35] F. Kawasaki,et al. Synaptic physiology and ultrastructure in comatose mutants defines an in vivo role for NSF in neurotransmitter release , 1998, Neuroscience Research.
[36] H. Bellen,et al. Synaptic Vesicle Size and Number Are Regulated by a Clathrin Adaptor Protein Required for Endocytosis , 1998, Neuron.
[37] R. Huganir,et al. Interaction of the N-Ethylmaleimide–Sensitive Factor with AMPA Receptors , 1998, Neuron.
[38] G. Collingridge,et al. NSF Binding to GluR2 Regulates Synaptic Transmission , 1998, Neuron.
[39] W. Wickner,et al. LMA1 Binds to Vacuoles at Sec18p (NSF), Transfers upon ATP Hydrolysis to a t-SNARE (Vam3p) Complex, and Is Released during Fusion , 1998, Cell.
[40] Y. Kidokoro,et al. Two Distinct Pools of Synaptic Vesicles in Single Presynaptic Boutons in a Temperature-Sensitive Drosophila Mutant, shibire , 1998, Neuron.
[41] K. Edwards,et al. GFP-moesin illuminates actin cytoskeleton dynamics in living tissue and demonstrates cell shape changes during morphogenesis in Drosophila. , 1997, Developmental biology.
[42] W. Saxton,et al. Kinesin mutations cause motor neuron disease phenotypes by disrupting fast axonal transport in Drosophila. , 1996, Genetics.
[43] P. De Camilli,et al. Mobility of Synaptic Vesicles in Nerve Endings Monitored by Recovery from Photobleaching of Synaptic Vesicle-Associated Fluorescence , 1996, The Journal of Neuroscience.
[44] W. Betz,et al. Synaptic Vesicle Movements Monitored by Fluorescence Recovery after Photobleaching in Nerve Terminals Stained with FM1-43 , 1996, The Journal of Neuroscience.
[45] M. Poo,et al. Effects of cytochalasin treatment on short‐term synaptic plasticity at developing neuromuscular junctions in frogs. , 1996, The Journal of physiology.
[46] L. Pallanck,et al. Distinct Roles for N-Ethylmaleimide-sensitive Fusion Protein (NSF) Suggested by the Identification of a Second Drosophila NSF Homolog (*) , 1995, The Journal of Biological Chemistry.
[47] W. Trimble,et al. Identification of a second homolog of N-ethylmaleimide-sensitive fusion protein that is expressed in the nervous system and secretory tissues of Drosophila. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[48] T. Südhof,et al. Essential functions of synapsins I and II in synaptic vesicle regulation , 1995, Nature.
[49] A. Bershadsky,et al. Swinholide A Is a Microfilament Disrupting Marine Toxin That Stabilizes Actin Dimers and Severs Actin Filaments (*) , 1995, The Journal of Biological Chemistry.
[50] J. Rothman,et al. N-ethylmaleimide-sensitive fusion protein: a trimeric ATPase whose hydrolysis of ATP is required for membrane fusion , 1994, The Journal of cell biology.
[51] J. Renger,et al. Improved stability of Drosophila larval neuromuscular preparations in haemolymph-like physiological solutions , 1994, Journal of Comparative Physiology A.
[52] Mark K. Bennett,et al. A protein assembly-disassembly pathway in vitro that may correspond to sequential steps of synaptic vesicle docking, activation, and fusion , 1993, Cell.
[53] Paul Tempst,et al. SNAP receptors implicated in vesicle targeting and fusion , 1993, Nature.
[54] J. Rothman,et al. Soluble N-ethylmaleimide-sensitive fusion attachment proteins (SNAPs) bind to a multi-SNAP receptor complex in Golgi membranes. , 1992, The Journal of biological chemistry.
[55] J. Rothman,et al. Purification of an N-ethylmaleimide-sensitive protein catalyzing vesicular transport. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[56] David J. Fleet,et al. Optical Flow Estimation , 2006, Handbook of Mathematical Models in Computer Vision.
[57] J. Henley,et al. Novel putative targets of N-ethylmaleimide sensitive fusion protein (NSF) and alpha/beta soluble NSF attachment proteins (SNAPs) include the Pak-binding nucleotide exchange factor betaPIX. , 2006, Journal of cellular biochemistry.
[58] R. Lefkowitz,et al. Identification of NSF as a beta-arrestin1-binding protein. Implications for beta2-adrenergic receptor regulation. , 1999, The Journal of biological chemistry.
[59] J. Bamburg,et al. Actin disassembles reversibly during electrically induced recycling of synaptic vesicles in cultured neurons. , 1998, Brain research. Molecular brain research.