Treatment of episodes of hereditary angioedema with C1 inhibitor: serial assessment of observed abnormalities of the plasma bradykinin-forming pathway and fibrinolysis.
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[1] P. Keith,et al. 2010 International consensus algorithm for the diagnosis, therapy and management of hereditary angioedema , 2010, Allergy, asthma, and clinical immunology : official journal of the Canadian Society of Allergy and Clinical Immunology.
[2] A. Kaplan,et al. Factor XII-independent cleavage of high-molecular-weight kininogen by prekallikrein and inhibition by C1 inhibitor. , 2009, Journal of Allergy and Clinical Immunology.
[3] B. Zuraw. Clinical practice. Hereditary angioedema. , 2008, The New England journal of medicine.
[4] A. Kaplan,et al. Studies of the mechanisms of bradykinin generation in hereditary angioedema plasma. , 2008, Annals of Allergy, Asthma & Immunology.
[5] P. Schlattmann,et al. Treatment of acute edema attacks in hereditary angioedema with a bradykinin receptor-2 antagonist (Icatibant). , 2007, The Journal of allergy and clinical immunology.
[6] M. Cicardi,et al. Anaphylaxis in Response to C1 Esterase Inhibitor in a Patient with Hereditary Angioedema , 2007 .
[7] M. Frank. Hereditary angioedema: the clinical syndrome and its management in the United States. , 2006, Immunology and allergy clinics of North America.
[8] William H. Yang,et al. Canadian 2003 International Consensus Algorithm For the Diagnosis, Therapy, and Management of Hereditary Angioedema. , 2004, The Journal of allergy and clinical immunology.
[9] A. Tordai,et al. Hereditary and acquired angioedema: Problems and progress: Proceedings of the third C1 esterase inhibitor deficiency workshop and beyond , 2004, Journal of Allergy and Clinical Immunology.
[10] P. Patston,et al. The reaction between plasmin and C1-inhibitor results in plasmin inhibition by the serpin mechanism , 2002, Blood coagulation & fibrinolysis : an international journal in haemostasis and thrombosis.
[11] M. Silverberg,et al. Pathways for bradykinin formation and inflammatory disease. , 2002, The Journal of allergy and clinical immunology.
[12] B. Zuraw,et al. Detection of C1 inhibitor mutations in patients with hereditary angioedema. , 2000, The Journal of allergy and clinical immunology.
[13] M. Cicardi,et al. Local bradykinin generation in hereditary angioedema. , 1999, The Journal of allergy and clinical immunology.
[14] M. Tosi. Molecular genetics of C1 inhibitor. , 1998, Immunobiology.
[15] J. Karsh,et al. C1-esterase inhibitor transfusions in patients with hereditary angioedema. , 1998, Annals of allergy, asthma & immunology : official publication of the American College of Allergy, Asthma, & Immunology.
[16] C. Feighery,et al. Degradation of C1-Inhibitor by Plasmin: Implications for the Control of Inflammatory Processes , 1997, Molecular medicine.
[17] M. Cicardi,et al. Activation of factor XII and cleavage of high molecular weight kininogen during acute attacks in hereditary and acquired C1-inhibitor deficiencies. , 1996, Immunopharmacology.
[18] A. Davis,et al. Hereditary angioneurotic oedema: characterization of plasma kinin and vascular permeability‐enhancing activities , 1994, Clinical and experimental immunology.
[19] A. Eerenberg,et al. Generation of plasmin during acute attacks of hereditary angioedema. , 1993, The Journal of laboratory and clinical medicine.
[20] V. Donaldson,et al. Comparison of human high molecular weight kininogen digestion by plasma kallikrein and by plasmin. A revised method of purification of high molecular weight kininogen. , 1987, The Journal of laboratory and clinical medicine.
[21] M. Silverberg,et al. The effect of C1 inhibitor upon Hageman factor autoactivation. , 1986, Blood.
[22] K. Fujikawa,et al. The activation of pro-urokinase by plasma kallikrein and its inactivation by thrombin. , 1986, The Journal of biological chemistry.
[23] M. Gilbert,et al. Tissue plasminogen activator release in vivo in response to vasoactive agents. , 1985, Blood.
[24] A. Kaplan,et al. Distribution of plasma kallikrein between C-1 inactivator and alpha 2-macroglobulin in plasma utilizing a new assay for alpha 2-macroglobulin-kallikrein complexes. , 1985, The Journal of biological chemistry.
[25] R. Colman,et al. The regulation of human factor XIIa by plasma proteinase inhibitors. , 1985, The Journal of biological chemistry.
[26] A. Kaplan,et al. Kinin formation in hereditary angioedema plasma: evidence against kinin derivation from C2 and in support of "spontaneous" formation of bradykinin. , 1983, The Journal of allergy and clinical immunology.
[27] R. Colman,et al. Prekallikrein activation and high-molecular-weight kininogen consumption in hereditary angioedema. , 1983, The New England journal of medicine.
[28] J. Dunn,et al. Mechanisms of activation of the classical pathway of complement by Hageman factor fragment. , 1983, The Journal of clinical investigation.
[29] A. Sheffer,et al. Clinical and biochemical effects of stanozolol therapy for hereditary angioedema. , 1981, The Journal of allergy and clinical immunology.
[30] M. Silverberg,et al. Activation of the classical pathway of complement by Hageman factor fragment , 1981, The Journal of experimental medicine.
[31] J. Gelfand,et al. Replacement therapy in hereditary angioedema: successful treatment of acute episodes of angioedema with partly purified C1 inhibitor. , 1980, The New England journal of medicine.
[32] A. Kaplan,et al. Hageman factor substrates. Human plasma prekallikrein: mechanism of activation by Hageman factor and participation in hageman factor-dependent fibrinolysis. , 1977, The Journal of biological chemistry.
[33] D. Alling,et al. Treatment of hereditary angioedema with danazol. Reversal of clinical and biochemical abnormalities. , 1976, The New England journal of medicine.
[34] I. Clemmensen,et al. The primary inhibitor of plasmin in human plasma. , 1976, The Biochemical journal.
[35] K.,et al. Inhibition by C1INH of Hagemann factor fragment activation of coagulation, fibrinolysis, and kinin generation. , 1973, The Journal of clinical investigation.
[36] A. Sheffer,et al. Tranexamic acid therapy in hereditary angioneurotic edema. , 1972, The New England journal of medicine.
[37] D. Alling,et al. Epsilon aminocaproic acid therapy of hereditary angioneurotic edema. A double-blind study. , 1972, The New England journal of medicine.
[38] K. Austen,et al. A PREALBUMIN ACTIVATOR OF PREKALLIKREIN , 1971, The Journal of experimental medicine.