The hSNM1B/Apollo variant rs11552449 is associated with cellular sensitivity towards mitomycin C and ionizing radiation.

[1]  J. Wu,et al.  Single nucleotide polymorphisms of nucleotide excision repair pathway are significantly associated with outcomes of platinum-based chemotherapy in lung cancer , 2017, Scientific Reports.

[2]  Minoru Takata,et al.  Biallelic mutations in the ubiquitin ligase RFWD3 cause Fanconi anemia , 2017, The Journal of clinical investigation.

[3]  I. Demuth,et al.  SNM1B/Apollo in the DNA damage response and telomere maintenance , 2017, Oncotarget.

[4]  J. Witte,et al.  Telomere structure and maintenance gene variants and risk of five cancer types , 2016, International journal of cancer.

[5]  F. Nielsen,et al.  Hereditary breast and ovarian cancer: new genes in confined pathways , 2016, Nature Reviews Cancer.

[6]  A. D’Andrea,et al.  The Fanconi anaemia pathway: new players and new functions , 2016, Nature Reviews Molecular Cell Biology.

[7]  Tian Liu,et al.  Genetic Burden Analyses of Phenotypes Relevant to Aging in the Berlin Aging Study II (BASE-II) , 2016, Gerontology.

[8]  E. Steinhagen-Thiessen,et al.  Relative Leukocyte Telomere Length, Hematological Parameters and Anemia - Data from the Berlin Aging Study II (BASE-II) , 2016, Gerontology.

[9]  U. Lindenberger,et al.  Editorial , 2016, Gerontology.

[10]  E. Ziv,et al.  Multiple breast cancer risk variants are associated with differential transcript isoform expression in tumors , 2015, Human molecular genetics.

[11]  J. Wu,et al.  Heterozygote advantage of methylenetetrahydrofolate reductase polymorphisms on clinical outcomes in advanced non-small cell lung cancer (NSCLC) patients treated with platinum-based chemotherapy , 2014, Tumor Biology.

[12]  Shu-Chen Li,et al.  Cohort profile: The Berlin Aging Study II (BASE-II). , 2014, International journal of epidemiology.

[13]  D. Bonatto,et al.  New features on Pso2 protein family in DNA interstrand cross-link repair and in the maintenance of genomic integrity in Saccharomyces cerevisiae. , 2013, Fungal genetics and biology : FG & B.

[14]  Jaana M. Hartikainen,et al.  Large-scale genotyping identifies 41 new loci associated with breast cancer risk , 2013, Nature Genetics.

[15]  I. Demuth,et al.  The nuclease hSNM1B/Apollo is linked to the Fanconi anemia pathway via its interaction with FANCP/SLX4. , 2012, Human molecular genetics.

[16]  S. Chanock,et al.  Genetic variants in DNA repair genes and the risk of cutaneous malignant melanoma in melanoma‐prone families with/without CDKN2A mutations , 2012, International journal of cancer.

[17]  J. Sekiguchi,et al.  Snm1B/Apollo functions in the Fanconi anemia pathway in response to DNA interstrand crosslinks. , 2011, Human molecular genetics.

[18]  Sandy Chang,et al.  The telomeric protein SNM1B/Apollo is required for normal cell proliferation and embryonic development , 2010, Aging cell.

[19]  T. de Lange,et al.  Apollo contributes to G overhang maintenance and protects leading-end telomeres. , 2010, Molecular cell.

[20]  E. Gilson,et al.  SNMIB/Apollo protects leading‐strand telomeres against NHEJ‐mediated repair , 2010, The EMBO journal.

[21]  G. Feldman,et al.  Hereditary breast and ovarian cancer due to mutations in BRCA1 and BRCA2 , 2010, Genetics in Medicine.

[22]  I. Demuth,et al.  Evidence for hSNM1B/Apollo functioning in the HSP70 mediated DNA damage response , 2009, Cell cycle.

[23]  Brian D. Freibaum,et al.  The Protein hSnm1B Is Stabilized When Bound to the Telomere-binding Protein TRF2* , 2008, Journal of Biological Chemistry.

[24]  J. Bae,et al.  Snm1B/Apollo mediates replication fork collapse and S Phase checkpoint activation in response to DNA interstrand cross-links , 2008, Oncogene.

[25]  I. Demuth,et al.  Endogenous hSNM1B/Apollo interacts with TRF2 and stimulates ATM in response to ionizing radiation. , 2008, DNA repair.

[26]  T. Lange,et al.  Apollo, an Artemis-Related Nuclease, Interacts with TRF2 and Protects Human Telomeres in S Phase , 2006, Current Biology.

[27]  H. Arakawa,et al.  Multiple repair pathways mediate tolerance to chemotherapeutic cross-linking agents in vertebrate cells. , 2005, Cancer research.

[28]  H. Arakawa,et al.  DNA Cross-Link Repair Protein SNM1A Interacts with PIAS1 in Nuclear Focus Formation , 2004, Molecular and Cellular Biology.

[29]  I. Demuth,et al.  Human SNM1B is required for normal cellular response to both DNA interstrand crosslink-inducing agents and ionizing radiation , 2004, Oncogene.

[30]  R. Cawthon Telomere measurement by quantitative PCR. , 2002, Nucleic acids research.

[31]  U. Lindenberger,et al.  The Berlin Aging Study II: An overview [Special section] , 2016 .

[32]  W Bounds,et al.  Seattle, Wa , 2022 .