Physical evidence of the coupling of solubilized 5-HT1A binding sites with G regulatory proteins.
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M. Hamon | S. El Mestikawy | M. Emerit | S. Mestikawy | M Hamon | B. Rouot | M B Emerit | H Gozlan | S el Mestikawy | B Rouot | H. Gozlan | M. Hamon | Bruno Rouot
[1] Y. Kitamura,et al. Effects of pertussis toxin on the alpha 2-adrenoceptor-inhibitory GTP-binding protein-adenylate cyclase system in rat brain: pharmacological and neurochemical studies. , 1987, European Journal of Pharmacology.
[2] M. Caron,et al. The D2-dopamine receptor of anterior pituitary is functionally associated with a pertussis toxin-sensitive guanine nucleotide binding protein. , 1987, The Journal of biological chemistry.
[3] M. Hamon,et al. [3H]8‐Hydroxy‐2‐(Di‐n‐Propylamino)Tetralin Binding to Pre‐ and Postsynaptic 5‐Hydroxytryptamine Sites in Various Regions of the Rat Brain , 1985, Journal of neurochemistry.
[4] L. Iversen,et al. REGIONAL STUDIES OF CATECHOLAMINES IN THE RAT BRAIN‐I , 1966, Journal of neurochemistry.
[5] D. Hoyer,et al. 5-HT1D receptor-mediated inhibition of forskolin-stimulated adenylate cyclase activity in calf substantia nigra. , 1988, European journal of pharmacology.
[6] R. Sekura,et al. Altered activity of the inhibitory guanyl nucleotide-binding component (Ni) induced by pertussis toxin. Uncoupling of Ni from receptor with continued coupling of Ni to the catalytic unit. , 1984, The Journal of biological chemistry.
[7] M. Hamon,et al. Ca2+‐Guanine Nucleotide Interactions in Brain Membranes. II. Characteristics of [3H]Guanosine Triphosphate and [3H]β, γ‐Imidoguanosine 5′‐Triphosphate Binding and Catabolism in the Rat Hippocampus and Striatum , 1982, Journal of neurochemistry.
[8] M. De Vivo,et al. Characterization of the 5-hydroxytryptamine1a receptor-mediated inhibition of forskolin-stimulated adenylate cyclase activity in guinea pig and rat hippocampal membranes. , 1986, The Journal of pharmacology and experimental therapeutics.
[9] J. Metcalfe,et al. Transient complexes. A new structural model for the activation of adenylate cyclase by hormone receptors (guanine nucleotides/irradiation inactivation). , 1979, The Biochemical journal.
[10] M. Caron,et al. The mammalian beta 2-adrenergic receptor: reconstitution of functional interactions between pure receptor and pure stimulatory nucleotide binding protein of the adenylate cyclase system. , 1984, Biochemistry.
[11] A. Gilman,et al. Guanine nucleotide-binding regulatory proteins; mediators of transmembrane signaling , 1987 .
[12] A. Herbet,et al. Multiple receptors for serotonin in the rat brain. , 1980, Advances in biochemical psychopharmacology.
[13] P. Bradley,et al. Proposals for the classification and nomenclature of functional receptors for 5-hydroxytryptamine , 1986, Neuropharmacology.
[14] O. H. Lowry,et al. Protein measurement with the Folin phenol reagent. , 1951, The Journal of biological chemistry.
[15] N. Mahy,et al. Pertussis toxin modifies the characteristics of both the inhibitory GTP binding proteins and the somatostatin receptor in anterior pituitary tumor cells. , 1988, The Journal of pharmacology and experimental therapeutics.
[16] A. Herbet,et al. Characteristics of central 5-HT receptors and their adaptive changes following intracerebral 5,7-dihydroxytryptamine administration in the rat. , 1978, Molecular pharmacology.
[17] R. North,et al. 5-HT3 receptors are membrane ion channels , 1989, Nature.
[18] R. Nicoll,et al. A G protein couples serotonin and GABAB receptors to the same channels in hippocampus. , 1986, Science.
[19] D. Kendall,et al. 5-Hydroxytryptamine-stimulated inositol phospholipid hydrolysis in rat cerebral cortex slices: pharmacological characterization and effects of antidepressants. , 1985, The Journal of pharmacology and experimental therapeutics.
[20] J. Bockaert,et al. 5-HT1B receptors are negatively coupled with adenylate cyclase in rat substantia nigra. , 1988, European journal of pharmacology.
[21] M. Vivo,et al. Serotonin decreases population spike amplitude in hippocampal cells through a pertussis toxin substrate , 1987, Brain Research.
[22] P. Conn,et al. A unique serotonin receptor in choroid plexus is linked to phosphatidylinositol turnover. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[23] P. Sternweis,et al. Isolation of two proteins with high affinity for guanine nucleotides from membranes of bovine brain. , 1984, The Journal of biological chemistry.
[24] M. Hamon,et al. Pharmacological and Biochemical Characterization of Rat Hippocampal 5‐Hydroxytryptamine1A Receptors Solubilized by 3–[3–(Cholamidopropyl)dimethylammonio]‐1‐Propane Sulfonate (CHAPS) , 1988, Journal of neurochemistry.
[25] W. Schlegel,et al. Activation of adenylate cyclase in hepatic membranes involves interactions of the catalytic unit with multimeric complexes of regulatory proteins. , 1979, The Journal of biological chemistry.
[26] L. Limbird,et al. Solubilization of human platelet alpha-adrenergic receptors: evidence that agonist occupancy of the receptor stabilizes receptor--effector interactions. , 1981, Proceedings of the National Academy of Sciences of the United States of America.
[27] 5-Hydroxytryptamine receptor subtypes. , 1990, Pharmacology & toxicology.
[28] M. Jackson,et al. 5-HT3 receptors mediate rapid responses in cultured hippocampus and a clonal cell line , 1988, Neuron.
[29] T. Pugsley,et al. A rapid filtration assay for soluble receptors using polyethylenimine-treated filters. , 1983, Analytical biochemistry.
[30] A. Couvineau,et al. Solubilization of the liver vasoactive intestinal peptide receptor. Hydrodynamic characterization and evidence for an association with a functional GTP regulatory protein. , 1986, The Journal of biological chemistry.
[31] Differential regulation of two molecular forms of a mu-opioid receptor type by sodium ions, manganese ions and by guanyl-5'-yl imidodiphosphate. , 1986, Journal of receptor research.
[32] M. Caron,et al. The genomic clone G-21 which resembles a β-adrenergic receptor sequence encodes the 5-HT1A receptor , 1988, Nature.
[33] R. Ciaranello,et al. Differential inactivation and G protein reconstitution of subtypes of [3H]5-hydroxytryptamine binding sites in brain. , 1988, Molecular pharmacology.
[34] G. Aghajanian,et al. Pertussis toxin blocks 5-HT1A and GABAB receptor-mediated inhibition of serotonergic neurons. , 1987, European journal of pharmacology.
[35] David E. Clapham,et al. Roles of G protein subunits in transmembrane signalling , 1988, Nature.
[36] J. Bockaert,et al. Pharmacology of 5-hydroxytryptamine-1A receptors which inhibit cAMP production in hippocampal and cortical neurons in primary culture. , 1988, Molecular pharmacology.
[37] T. Asano,et al. Prevention of the agonist binding to gamma-aminobutyric acid B receptors by guanine nucleotides and islet-activating protein, pertussis toxin, in bovine cerebral cortex. Possible coupling of the toxin-sensitive GTP-binding proteins to receptors. , 1985, The Journal of biological chemistry.
[38] M. Hamon,et al. Chromatographic Analyses of the Serotonin 5‐HT1A Receptor Solubilized from the Rat Hippocampus , 1989, Journal of neurochemistry.
[39] M. Caron,et al. Effector coupling mechanisms of the cloned 5-HT1A receptor. , 1989, The Journal of biological chemistry.
[40] M. Caron,et al. An intronless gene encoding a potential member of the family of receptors coupled to guanine nucleotide regulatory proteins , 1987, Nature.
[41] B. Roth,et al. Multiple mechanisms of serotonergic signal transduction. , 1987, Life sciences.