PINK1/Parkin mediated mitophagy ameliorates palmitic acid-induced apoptosis through reducing mitochondrial ROS production in podocytes.
暂无分享,去创建一个
Hua Gan | Ting Liu | Xu-shun Jiang | Xue-mei Chen | Jiang-min Wan | Xiao-Gang Du | Wei Hua | Jun-ling He | Xun-jia Li
[1] Lijun Xu,et al. Berberine Protects Glomerular Podocytes via Inhibiting Drp1-Mediated Mitochondrial Fission and Dysfunction , 2019, Theranostics.
[2] Z. Dong,et al. PINK1/Parkin-mediated mitophagy is activated in cisplatin nephrotoxicity to protect against kidney injury , 2018, Cell Death & Disease.
[3] Yuehua Wu,et al. Palmitic Acid-Induced Podocyte Apoptosis via the Reactive Oxygen Species-Dependent Mitochondrial Pathway , 2018, Kidney and Blood Pressure Research.
[4] G. Qin,et al. FoxO1 Promotes Mitophagy in the Podocytes of Diabetic Male Mice via the PINK1/Parkin Pathway , 2017, Endocrinology.
[5] Y. Kanwar,et al. Disruption of renal tubular mitochondrial quality control by Myo-inositol oxygenase in diabetic kidney disease. , 2015, Journal of the American Society of Nephrology : JASN.
[6] V. D’Agati,et al. Deficient Autophagy Results in Mitochondrial Dysfunction and FSGS. , 2015, Journal of the American Society of Nephrology : JASN.
[7] W. Ding,et al. Mitochondrial dynamics and mitochondrial quality control , 2014, Redox biology.
[8] D. Koya,et al. Lipid mediators in diabetic nephropathy , 2014, Fibrogenesis & tissue repair.
[9] Jianhua Zhang,et al. Mitophagy mechanisms and role in human diseases. , 2014, The international journal of biochemistry & cell biology.
[10] Songming Huang,et al. Mitochondrial dysfunction in the pathophysiology of renal diseases. , 2014, American journal of physiology. Renal physiology.
[11] T. Horino,et al. Sestrin-2 and BNIP3 regulate autophagy and mitophagy in renal tubular cells in acute kidney injury. , 2013, American journal of physiology. Renal physiology.
[12] Lin Sun,et al. Mitochondrial dynamics: regulatory mechanisms and emerging role in renal pathophysiology , 2012, Kidney international.
[13] T. Schwarz,et al. The pathways of mitophagy for quality control and clearance of mitochondria , 2012, Cell Death and Differentiation.
[14] P. Kim,et al. ROS-induced mitochondrial depolarization initiates PARK2/PARKIN-dependent mitochondrial degradation by autophagy , 2012, Autophagy.
[15] Hyun Soon Lee. Mechanisms and consequences of hypertriglyceridemia and cellular lipid accumulation in chronic kidney disease and metabolic syndrome. , 2011, Histology and histopathology.
[16] D. Selkoe,et al. The mitochondrial intramembrane protease PARL cleaves human Pink1 to regulate Pink1 trafficking , 2011, Journal of neurochemistry.
[17] R. Youle,et al. Targeting mitochondrial dysfunction: role for PINK1 and Parkin in mitochondrial quality control. , 2011, Antioxidants & redox signaling.
[18] N. Wood,et al. Mitophagy and Parkinson's disease: The PINK1–parkin link , 2011, Biochimica et biophysica acta.
[19] A. Whitworth,et al. PINK1 cleavage at position A103 by the mitochondrial protease PARL , 2010, Human molecular genetics.
[20] C. Mammucari,et al. Signaling pathways in mitochondrial dysfunction and aging , 2010, Mechanisms of Ageing and Development.
[21] N. Hattori,et al. PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy , 2010, The Journal of cell biology.
[22] N. Hattori,et al. PINK1 is recruited to mitochondria with parkin and associates with LC3 in mitophagy , 2010, FEBS letters.
[23] Fabienne C. Fiesel,et al. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 , 2010, Nature Cell Biology.
[24] Ji Zhang,et al. Role of BNIP3 and NIX in cell death, autophagy, and mitophagy , 2009, Cell Death and Differentiation.
[25] R. Youle,et al. Parkin is recruited selectively to impaired mitochondria and promotes their autophagy , 2008, The Journal of cell biology.
[26] L. Scorrano,et al. High levels of Fis1, a pro-fission mitochondrial protein, trigger autophagy. , 2008, Biochimica et biophysica acta.
[27] Min Wu,et al. Fission and selective fusion govern mitochondrial segregation and elimination by autophagy , 2008, The EMBO journal.
[28] P. Schrauwen,et al. Oxidative capacity, lipotoxicity, and mitochondrial damage in type 2 diabetes. , 2004, Diabetes.
[29] S. Emr,et al. Autophagy as a regulated pathway of cellular degradation. , 2000, Science.
[30] T. Rabelink,et al. Early mechanisms of renal injury in hypercholesterolemic or hypertriglyceridemic rats. , 2000, Journal of the American Society of Nephrology : JASN.
[31] T. Meyer,et al. Podocyte loss and progressive glomerular injury in type II diabetes. , 1997, The Journal of clinical investigation.
[32] D. Wheeler,et al. Lipid abnormalities in the nephrotic syndrome: causes, consequences, and treatment. , 1994, American journal of kidney diseases : the official journal of the National Kidney Foundation.
[33] Z. Varghese,et al. LIPID NEPHROTOXICITY IN CHRONIC PROGRESSIVE GLOMERULAR AND TUBULO-INTERSTITIAL DISEASE , 1982, The Lancet.