Effect of mineral trioxide aggregate on dentin bridge formation and expression of dentin sialoprotein and heme oxygenase-1 in human dental pulp.

[1]  A. Reis,et al.  Evaluation of Mineral Trioxide Aggregate and Calcium Hydroxide Cement as Pulp-capping Agents in Human Teeth Materials and Methods , 2007 .

[2]  Eun-Cheol Kim,et al.  The induction of heme oxygenase-1 modulates bismuth oxide-induced cytotoxicity in human dental pulp cells. , 2007, Journal of endodontics.

[3]  C. Pameijer,et al.  Clinical and histological evaluation of white ProRoot MTA in direct pulp capping. , 2006, American journal of dentistry.

[4]  M. Plotkowski,et al.  Cytotoxicity of MTA and Portland cement on human ECV 304 endothelial cells. , 2005, International endodontic journal.

[5]  J. Gutmann,et al.  Histological and scanning electron microscopy assessment of various vital pulp-therapy materials. , 2003, Journal of endodontics.

[6]  M. Ghanbariha,et al.  Mineral trioxide aggregate (MTA) and calcium hydroxide as pulp-capping agents in human teeth: a preliminary report. , 2003, International endodontic journal.

[7]  R. Holland,et al.  Response of the pulp of dogs to capping with mineral trioxide aggregate or a calcium hydroxide cement. , 2001, Dental traumatology : official publication of International Association for Dental Traumatology.

[8]  L. Gibbs,et al.  Inhibition of inducible nitric oxide synthase in the human intestinal epithelial cell line, DLD-1, by the inducers of heme oxygenase 1, bismuth salts, heme, and nitric oxide donors , 2000, Gut.

[9]  R. Foresti,et al.  The heme oxygenase pathway and its interaction with nitric oxide in the control of cellular homeostasis. , 1999, Free radical research.

[10]  M. Torabinejad,et al.  Osteoblast biocompatibility of mineral trioxide aggregate. , 1999, Biomaterials.

[11]  E. T. Koh,et al.  Cellular response to Mineral Trioxide Aggregate. , 1998, Journal of endodontics.

[12]  J. Hoidal,et al.  Effect of tumor necrosis factor-α and interleukin-1α on heme oxygenase-1 expression in human endothelial cells. , 1998, American journal of physiology. Heart and circulatory physiology.

[13]  H. Stanley,et al.  The disastrous effects of the "total etch" technique in vital pulp capping in primates. , 1998, American journal of dentistry.

[14]  M. Torabinejad,et al.  Using mineral trioxide aggregate as a pulp-capping material. , 1996, Journal of the American Dental Association.

[15]  A. Choi,et al.  Hemoglobin provides protection against lethal endotoxemia in rats: the role of heme oxygenase-1. , 1995, American journal of respiratory cell and molecular biology.

[16]  M. Torabinejad,et al.  Cytotoxicity of four root end filling materials. , 1995, Journal of endodontics.

[17]  M. Torabinejad,et al.  Physical and chemical properties of a new root-end filling material. , 1995, Journal of endodontics.

[18]  M. Torabinejad,et al.  Comparative investigation of marginal adaptation of mineral trioxide aggregate and other commonly used root-end filling materials. , 1995, Journal of endodontics.

[19]  R. D'Souza,et al.  Characterization of Cellular Responses Involved in Reparative Dentinogenesis in Rat Molars , 1995, Journal of dental research.

[20]  W. Butler.  Dentin matrix proteins and dentinogenesis. , 1995, Connective tissue research.

[21]  H. Stanley.  Pulp capping: conserving the dental pulp--can it be done? Is it worth it? , 1989, Oral surgery, oral medicine, and oral pathology.

[22]  U. Schröder.  Effects of Calcium Hydroxide-containing Pulp-capping Agents on Pulp Cell Migration, Proliferation, and Differentiation , 1985, Journal of dental research.

[23]  L. Tronstad,et al.  Reaction of the exposed pulp to Dycal treatment. , 1974, Oral surgery, oral medicine, and oral pathology.

[24]  T. Lundy,et al.  Dycal therapy for pulp exposures. , 1972, Oral surgery, oral medicine, and oral pathology.