Diabetes in Mice With Selective Impairment of Insulin Action in Glut4-Expressing Tissues
暂无分享,去创建一个
G. Shulman | D. Accili | Hui-Young Lee | V. Samuel | Hongxia Ren | Taylor Y. Lu | H. Lin
[1] T. Horvath,et al. Divergent Regulation of Energy Expenditure and Hepatic Glucose Production by Insulin Receptor in Agouti-Related Protein and POMC Neurons , 2009, Diabetes.
[2] Gabriel M. Belfort,et al. Cerebellar Neurons Possess a Vesicular Compartment Structurally and Functionally Similar to Glut4-Storage Vesicles from Peripheral Insulin-Sensitive Tissues , 2009, The Journal of Neuroscience.
[3] Kathryn Moynihan Ramsey,et al. High-fat diet disrupts behavioral and molecular circadian rhythms in mice. , 2007, Cell metabolism.
[4] B. Kahn,et al. Tissue-specific Alterations of Glucose Transport and Molecular Mechanisms of Intertissue Communication in Obesity and Type 2 Diabetes , 2007, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.
[5] R. DePinho,et al. Impaired regulation of hepatic glucose production in mice lacking the forkhead transcription factor Foxo1 in liver. , 2007, Cell metabolism.
[6] X. Papademetris,et al. The role of skeletal muscle insulin resistance in the pathogenesis of the metabolic syndrome , 2007, Proceedings of the National Academy of Sciences.
[7] R. Mirmira. Faculty Opinions recommendation of Insulin receptors in beta-cells are critical for islet compensatory growth response to insulin resistance. , 2007 .
[8] F. Ashcroft,et al. Insulin action in AgRP-expressing neurons is required for suppression of hepatic glucose production. , 2007, Cell metabolism.
[9] M. Stoffel,et al. Insulin receptors in β-cells are critical for islet compensatory growth response to insulin resistance , 2007, Proceedings of the National Academy of Sciences.
[10] C. Sosnowski,et al. [Commentary to the article: Kahn SE, Haffner SM, Heise MA, et al. ADOPT Study Group. Glycemic durability of rosiglitazone, metformin, or glyburide monotherapy. N Engl J Med 2006; 355: 2427-43]. , 2007, Kardiologia polska.
[11] B. Zinman,et al. Glycemic durability of rosiglitazone, metformin, or glyburide monotherapy. , 2006, The New England journal of medicine.
[12] R. Klein,et al. Role of brain insulin receptor in control of body weight and reproduction. , 2006, Science.
[13] D. Accili,et al. FoxO1 protects against pancreatic beta cell failure through NeuroD and MafA induction. , 2005, Cell metabolism.
[14] K. Nanjo,et al. Subcellular localization of glucose transporter 4 in the hypothalamic arcuate nucleus of ob/ob mice under basal conditions , 2005, Brain Research.
[15] Fred W. Turek,et al. Obesity and Metabolic Syndrome in Circadian Clock Mutant Mice , 2005, Science.
[16] G. Wilcox. Insulin and insulin resistance. , 2005, The Clinical biochemist. Reviews.
[17] D. Porte,et al. Diabetes, Obesity, and the Brain , 2005, Science.
[18] O. Boss,et al. GLUT4 glucose transporter deficiency increases hepatic lipid production and peripheral lipid utilization. , 2004, The Journal of clinical investigation.
[19] A. Goldfine,et al. The cellular fate of glucose and its relevance in type 2 diabetes. , 2004, Endocrine reviews.
[20] V. Routh,et al. Neuronal glucosensing: what do we know after 50 years? , 2004, Diabetes.
[21] V. Routh,et al. The regulation of glucose-excited neurons in the hypothalamic arcuate nucleus by glucose and feeding-relevant peptides. , 2004, Diabetes.
[22] D. Accili,et al. Transgenic rescue of insulin receptor-deficient mice. , 2004, The Journal of clinical investigation.
[23] D. Accili. Lilly lecture 2003: the struggle for mastery in insulin action: from triumvirate to republic. , 2004, Diabetes.
[24] B. McEwen,et al. Glucose transporter expression in the central nervous system: relationship to synaptic function. , 2004, European journal of pharmacology.
[25] V. Routh,et al. Physiological and molecular characteristics of rat hypothalamic ventromedial nucleus glucosensing neurons. , 2004, Diabetes.
[26] C. Kahn,et al. Insulin receptor knockout mice. , 2003, Annual review of physiology.
[27] Z. Bloomgarden,et al. Inflammation and insulin resistance. , 2003, Diabetes care.
[28] Y. Kido,et al. The forkhead transcription factor Foxo1 links insulin signaling to Pdx1 regulation of pancreatic beta cell growth. , 2002, The Journal of clinical investigation.
[29] L. Rossetti,et al. Hypothalamic insulin signaling is required for inhibition of glucose production , 2002, Nature Medicine.
[30] R. Sankar,et al. Insulin-responsive glucose transporters-GLUT8 and GLUT4 are expressed in the developing mammalian brain. , 2002, Brain research. Molecular brain research.
[31] Y. Kido,et al. Defective insulin secretion in pancreatic beta cells lacking type 1 IGF receptor. , 2002, The Journal of clinical investigation.
[32] D. Accili,et al. Regulation of insulin action and pancreatic β-cell function by mutated alleles of the gene encoding forkhead transcription factor Foxo1 , 2002, Nature Genetics.
[33] C. Kahn,et al. Adipose tissue selective insulin receptor knockout protects against obesity and obesity-related glucose intolerance. , 2002, Developmental cell.
[34] Jochen Roeper,et al. ATP-sensitive K+ channels in the hypothalamus are essential for the maintenance of glucose homeostasis , 2001, Nature Neuroscience.
[35] W. Lee,et al. Insulin-like growth factor 1 regulates developing brain glucose metabolism. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[36] C. Kahn,et al. Redistribution of substrates to adipose tissue promotes obesity in mice with selective insulin resistance in muscle. , 2000, The Journal of clinical investigation.
[37] Y. Kido,et al. Tissue-specific insulin resistance in mice with mutations in the insulin receptor, IRS-1, and IRS-2. , 2000, The Journal of clinical investigation.
[38] R. Schliebs,et al. Insulin‐sensitive GLUT4 glucose transporters are colocalized with GLUT3‐expressing cells and demonstrate a chemically distinct neuron‐specific localization in rat brain , 1999, Journal of neuroscience research.
[39] Y. Kido,et al. Impaired glucose tolerance in mice with a targeted impairment of insulin action in muscle and adipose tissue , 1998, Nature Genetics.
[40] L. Rossetti,et al. Type 2 diabetes — who is conducting the orchestra? , 1998, Nature Genetics.
[41] C. Kahn,et al. A muscle-specific insulin receptor knockout exhibits features of the metabolic syndrome of NIDDM without altering glucose tolerance. , 1998, Molecular cell.
[42] L. Pénicaud,et al. Immunocytochemical localization of the insulin‐responsive glucose transporter 4 (Glut4) in the rat central nervous system , 1998, The Journal of comparative neurology.
[43] C. B. Smith,et al. Glucose Transporter Expression in Brain: Relationship to Cerebral Glucose Utilization , 1998, Developmental Neuroscience.
[44] S. Vannucci,et al. GLUT4 glucose transporter expression in rodent brain: effect of diabetes , 1998, Brain Research.
[45] S. Vannucci,et al. Glucose transporter proteins in brain: Delivery of glucose to neurons and glia , 1997, Glia.
[46] C. Kahn,et al. Development of a Novel Polygenic Model of NIDDM in Mice Heterozygous for IR and IRS-1 Null Alleles , 1997, Cell.
[47] S. Vannucci,et al. Glucose transporter proteins in brain , 1994, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[48] G. Bell,et al. Hormonal/metabolic regulation of the human GLUT4/muscle-fat facilitative glucose transporter gene in transgenic mice. , 1993, The Journal of biological chemistry.
[49] Y. Kido,et al. Defective insulin secretion in pancreatic beta cells lacking type 1 IGF receptor. , 2002, The Journal of clinical investigation.
[50] S. Vannucci,et al. Alterations in brain glucose transporter proteins, GLUT1 and GLUT3, in streptozotocin diabetic rats. , 1993, Advances in experimental medicine and biology.