Retrograde neurotrophin signaling: Trk-ing along the axon
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[1] Scott L Pomeroy,et al. High-resolution imaging demonstrates dynein-based vesicular transport of activated Trk receptors. , 2002, Journal of neurobiology.
[2] R. Campenot,et al. Retrograde Support of Neuronal Survival Without Retrograde Transport of Nerve Growth Factor , 2002, Science.
[3] C. Howe,et al. NGF Signaling from Clathrin-Coated Vesicles Evidence that Signaling Endosomes Serve as a Platform for the Ras-MAPK Pathway , 2001, Neuron.
[4] Barbara L. Hempstead,et al. Regulation of Cell Survival by Secreted Proneurotrophins , 2001, Science.
[5] L. Goldstein,et al. Disruption of Axonal Transport and Neuronal Viability by Amyloid Precursor Protein Mutations in Drosophila , 2001, Neuron.
[6] Rosalind A. Segal,et al. Neurotrophins use the Erk5 pathway to mediate a retrograde survival response , 2001, Nature Neuroscience.
[7] P. Rakic,et al. Requirement of the JIP1 scaffold protein for stress-induced JNK activation. , 2001, Genes & development.
[8] Ahmad Salehi,et al. Failed retrograde transport of NGF in a mouse model of Down's syndrome: Reversal of cholinergic neurodegenerative phenotypes following NGF infusion , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[9] W. Mobley,et al. Nerve Growth Factor Activates Persistent Rap1 Signaling in Endosomes , 2001, The Journal of Neuroscience.
[10] P. McPherson,et al. Signaling on the Endocytic Pathway , 2001, Traffic.
[11] S. Bartlett,et al. Prolonged recycling of internalized neurotrophins in the nerve terminal. , 2001, Cytometry.
[12] C. Sung,et al. Association of Trk Neurotrophin Receptors with Components of the Cytoplasmic Dynein Motor , 2001, The Journal of Neuroscience.
[13] S. Poser,et al. Differential Regulation of Mitogen-Activated Protein Kinases ERK1/2 and ERK5 by Neurotrophins, Neuronal Activity, and cAMP in Neurons , 2001, The Journal of Neuroscience.
[14] E. Mccleskey,et al. Role of Phosphoinositide 3-Kinase and Endocytosis in Nerve Growth Factor-Induced Extracellular Signal-Regulated Kinase Activation via Ras and Rap1 , 2000, Molecular and Cellular Biology.
[15] D. Ginty,et al. Spatially and Functionally Distinct Roles of the PI3-K Effector Pathway during NGF Signaling in Sympathetic Neurons , 2000, Neuron.
[16] D. Kaplan,et al. The TrkB-Shc Site Signals Neuronal Survival and Local Axon Growth via MEK and PI3-Kinase , 2000, Neuron.
[17] Bevil R. Conway,et al. Cell Surface Trk Receptors Mediate NGF-Induced Survival While Internalized Receptors Regulate NGF-Induced Differentiation , 2000, The Journal of Neuroscience.
[18] David R Kaplan,et al. Neurotrophin signal transduction in the nervous system , 2000, Current Opinion in Neurobiology.
[19] S. Schmid,et al. Regulation of signal transduction by endocytosis. , 2000, Current opinion in cell biology.
[20] D. Ginty,et al. Characterization of an NGF–P-TrkA Retrograde-Signaling Complex and Age-Dependent Regulation of TrkA Phosphorylation in Sympathetic Neurons , 1999, The Journal of Neuroscience.
[21] F. L. Watson,et al. Rapid Nuclear Responses to Target-Derived Neurotrophins Require Retrograde Transport of Ligand–Receptor Complex , 1999, The Journal of Neuroscience.
[22] J. Priestley,et al. Axonal Transport of Activating Transcription Factor-2 Is Modulated by Nerve Growth Factor in Nociceptive Neurons , 1999, The Journal of Neuroscience.
[23] S. Bartlett,et al. Evidence for Phosphatidylinositol 4‐Kinase and Actin Involvement in the Regulation of 125I‐β‐Nerve Growth Factor Retrograde Axonal Transport , 1999, Journal of neurochemistry.
[24] S. Bartlett,et al. Signalling events regulating the retrograde axonal transport of 125 I−βNerve growth factor in vivo , 1998, Brain Research.
[25] F. L. Watson,et al. Trk Receptors Function As Rapid Retrograde Signal Carriers in the Adult Nervous System , 1997, The Journal of Neuroscience.
[26] W. Mobley,et al. A signaling organelle containing the nerve growth factor-activated receptor tyrosine kinase, TrkA. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[27] A. Riccio,et al. An NGF-TrkA-mediated retrograde signal to transcription factor CREB in sympathetic neurons. , 1997, Science.
[28] Robert B. Campenot,et al. Rapid Retrograde Tyrosine Phosphorylation of trkA and Other Proteins in Rat Sympathetic Neurons in Compartmented Cultures , 1997, The Journal of cell biology.
[29] J. Lavail,et al. Endocytosis of Activated TrkA: Evidence that Nerve Growth Factor Induces Formation of Signaling Endosomes , 1996, The Journal of Neuroscience.
[30] E. Dı́az-Rodrı́guez,et al. TrkA receptor ectodomain cleavage generates a tyrosine-phosphorylated cell-associated fragment , 1996, The Journal of cell biology.
[31] M. Ehlers,et al. NGF-stimulated retrograde transport of trkA in the mammalian nervous system , 1995, The Journal of cell biology.
[32] R. Jaenisch,et al. Differential role of the low affinity neurotrophin receptor (p75) in retrograde axonal transport of the neurotrophins , 1995, Neuron.
[33] M. Greenberg,et al. Membrane depolarization and calcium influx stimulate MEK and MAP kinase via activation of Ras , 1994, Neuron.
[34] B. Hempstead,et al. Overexpression of the trk tyrosine kinase rapidly accelerates nerve growth factor-induced differentiation , 1992, Neuron.
[35] K. Oiwa,et al. The force-velocity relationship for microtubule sliding in demembranated sperm flagella of the sea urchin. , 1988, Cell structure and function.
[36] L L Iversen,et al. The retrograde axonal transport of nerve growth factor. , 1974, Brain research.
[37] M. Sofroniew,et al. Nerve growth factor signaling, neuroprotection, and neural repair. , 2001, Annual review of neuroscience.
[38] E. Huang,et al. Neurotrophins: roles in neuronal development and function. , 2001, Annual review of neuroscience.
[39] D. Cleveland,et al. Slowing of axonal transport is a very early event in the toxicity of ALS–linked SOD1 mutants to motor neurons , 1999 .
[40] C. Howe,et al. A signaling endosome hypothesis to explain NGF actions: potential implications for neurodegeneration. , 1996, Cold Spring Harbor symposia on quantitative biology.