The effects of chronic treatment with amitriptyline and MDL 72394 on the control of 5-HT release in vivo

[1]  W. Lovenberg,et al.  Chronic MAO A and MAO B inhibition decreases the 5-HT1A receptor-mediated inhibition of forskolin-stimulated adenylate cyclase. , 1988, European journal of pharmacology.

[2]  A. Sleight,et al.  Relationship between extracellular 5‐hydroxytryptamine and behaviour following monoamine oxidase inhibition and l‐tryptophan , 1988, British journal of pharmacology.

[3]  A. R. Green,et al.  The effects of single and repeated electroconvulsive shock administration on the release of 5‐hydroxytryptamine and noradrenaline from cortical slices of rat brain , 1987, British journal of pharmacology.

[4]  N. Maidment,et al.  In Vivo Voltammetry , 1987 .

[5]  D. Middlemiss,et al.  Monoamine receptor sensitivity changes following chronic administration of MDL 72394, a site-directed inhibitor of monoamine oxidase. , 1986, European journal of pharmacology.

[6]  P. Bradley,et al.  Proposals for the classification and nomenclature of functional receptors for 5-hydroxytryptamine , 1986, Neuropharmacology.

[7]  C. Marsden,et al.  In vivo voltammetry in the suprachiasmatic nucleus of the rat: effects of RU24969, methiothepin and ketanserin. , 1986, European journal of pharmacology.

[8]  A. Green Anti-depressant treatments and serotonin receptor number and function. , 2009, Acta pharmacologica et toxicologica.

[9]  C. Marsden,et al.  Recent advances in high-performance liquid chromatographic analysis of small molecules. Application of high-performance liquid chromatography with electrochemical detection to the study of neurotransmitters in vivo. , 1985, Biochemical Society transactions.

[10]  J. Palacios,et al.  Quantitative autoradiographic mapping of serotonin receptors in the rat brain. I. Serotonin-1 receptors , 1985, Brain Research.

[11]  C. Routledge,et al.  The 5‐HT1 receptor agonist RU‐24969 decreases 5‐hydroxytryptamine (5‐HT) release and metabolism in the rat frontal cortex in vitro and in vivo , 1985, British journal of pharmacology.

[12]  M. Hamon,et al.  Presynaptic 5-HT autoreceptors on serotonergic cell bodies and/or dendrites but not terminals are of the 5-HT1A subtype. , 1985, European journal of pharmacology.

[13]  C. Montigny,et al.  Electrophysiologically-identified serotonin receptors in the rat CNS Effect of antidepressant treatment , 1984, Neuropharmacology.

[14]  J. Palacios,et al.  The binding of serotonergic ligands to the porcine choroid plexus: characterization of a new type of serotonin recognition site. , 1984, European journal of pharmacology.

[15]  C. Gardner,et al.  The behavioural effects of RU 24969, a suggested 5-HT1 receptor agonist in rodents and the effect on the behaviour of treatment with antidepressants , 1984, Neuropharmacology.

[16]  D. Middlemiss Stereoselective blockade at [3H]5-HT binding sites and at the 5-HT autoreceptor by propranolol. , 1984, European journal of pharmacology.

[17]  M. Palfreyman,et al.  (E)-.beta.-(Fluoromethylene)-m-tyrosine: a substrate for aromatic L-amino acid decarboxylase liberating an enzyme-activated irreversible inhibitor of monoamine oxidase , 1984 .

[18]  N. Shorstein,et al.  Rapid down-regulation of serotonin2 receptor binding during combined administration of tricyclic antidepressant drugs and α 2 antagonists , 1983, Neuropharmacology.

[19]  M. F. Sugrue Do antidepressants possess a common mechanism of action? , 1983, Biochemical pharmacology.

[20]  Y. Nomura,et al.  Modifications of central 5-hydroxytryptamine binding sites in synaptic membranes from rat brain after long-term administration of tricyclic antidepressants. , 1979, European journal of pharmacology.