Discordance of P53 Mutations of Synchronous Colorectal Carcinomas

[1]  N. Kinukawa,et al.  p53 mutations in multiple urothelial carcinomas: a molecular analysis of the development of multiple carcinomas. , 1997, Modern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc.

[2]  G. Kang,et al.  Genetic evidence for the multicentric origin of synchronous multiple gastric carcinoma. , 1997, Laboratory investigation; a journal of technical methods and pathology.

[3]  P. Vihko,et al.  Conserved region mutations of the p53 gene are concentrated in distal colorectal cancers , 1997, International journal of cancer.

[4]  R. Scully,et al.  Ovarian, peritoneal, and endometrial serous carcinoma: clonal origin of multifocal disease. , 1996, Modern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc.

[5]  M. Kelley,et al.  TP53 and RAS mutations in metachronous tumors from patients with cancer of the upper aerodigestive tract , 1995, International journal of cancer.

[6]  C. Harris,et al.  Mutations in the p53 tumor suppressor gene: clues to cancer etiology and molecular pathogenesis. , 1994, Cancer research.

[7]  S. Noguchi,et al.  Discrimination between multicentric and multifocal carcinomas of the breast through clonal analysis , 1994, Cancer.

[8]  S. Groshen,et al.  p53 nuclear protein accumulation correlates with mutations in the p53 gene, tumor grade, and stage in bladder cancer. , 1993, The American journal of pathology.

[9]  J. Farndon,et al.  Correlation between p53 mutations and antibody staining in breast carcinoma , 1993 .

[10]  W. Hong,et al.  Discordant p53 gene mutations in primary head and neck cancers and corresponding second primary cancers of the upper aerodigestive tract. , 1993, Cancer research.

[11]  R. Knapp,et al.  Unifocal origin of advanced human epithelial ovarian cancers. , 1992, Cancer research.

[12]  R. Lothe,et al.  Molecular genetic studies of tumor suppressor gene regions on chromosomes 13 and 17 in colorectal tumors. , 1992, Journal of the National Cancer Institute.

[13]  M. Seki,et al.  Genetic changes of both p53 alleles associated with the conversion from colorectal adenoma to early carcinoma in familial adenomatous polyposis and non-familial adenomatous polyposis patients. , 1992, Cancer research.

[14]  S. Hirohashi,et al.  Mutation pattern of the p53 gene as a diagnostic marker for multiple hepatocellular carcinoma. , 1992, Cancer research.

[15]  D. Schaid,et al.  Expression of p53 and 17p allelic loss in colorectal carcinoma. , 1992, Cancer research.

[16]  S. Hirohashi,et al.  Different pattern of chromosomal allele loss in multiple hepatocellular carcinomas as evidence of their multifocal origin. , 1992, Cancer research.

[17]  B. Vogelstein,et al.  Clonal origin of bladder cancer. , 1992, The New England journal of medicine.

[18]  D. Neal,et al.  c-erbB-2 amplification and identical p53 mutations in concomitant transitional carcinomas of renal pelvis and urinary bladder , 1992, The Lancet.

[19]  B. Vogelstein,et al.  p53 mutations in human cancers. , 1991, Science.

[20]  A. Levine,et al.  The p53 tumour suppressor gene , 1991, Nature.

[21]  J. Mecklin,et al.  The International Collaborative Group on Hereditary Non-Polyposis Colorectal Cancer (ICG-HNPCC) , 1991, Diseases of the colon and rectum.

[22]  B. Vogelstein,et al.  p53 gene mutations occur in combination with 17p allelic deletions as late events in colorectal tumorigenesis. , 1990, Cancer research.

[23]  G. Fleuren,et al.  Flow cytometric DNA‐ploidy analysis of synchronously occurring multiple malignant tumors of the female genital tract , 1990, Cancer.

[24]  F. Collins,et al.  Mutations in the p53 gene occur in diverse human tumour types , 1989, Nature.

[25]  M. Makuuchi,et al.  Multicentric independent development of hepatocellular carcinoma revealed by analysis of hepatitis B virus integration pattern. , 1989, The American journal of surgical pathology.

[26]  T. Sekiya,et al.  Rapid and sensitive detection of point mutations and DNA polymorphisms using the polymerase chain reaction. , 1989, Genomics.

[27]  A. Feinberg,et al.  MULTIPLE GENETIC ALTERATIONS IN DISTAL AND PROXIMAL COLORECTAL CANCER , 1989, The Lancet.

[28]  G. Bolis Randomised comparison of cisplatin with Cyclophosphamide/Cisplatin and with Cyclophosphamide/Doxorubicin/Cisplatin in advanced ovarian cancer , 1988 .

[29]  J. Coon,et al.  Origin of multiple “primary” colon carcinomas: A retrospective flow cytometric study , 1986, Cancer.

[30]  D. Sachar,et al.  A Comparison of Multiple Synchronous Colorectal Cancer in Ulcerative Colitis, Familial Polyposis Coli, and de Novo Cancer , 1986, Annals of surgery.

[31]  B. Vogelstein,et al.  Purification of DNA from formaldehyde fixed and paraffin embedded human tissue. , 1985, Biochemical and biophysical research communications.

[32]  F. R. Watson,et al.  Cancer of the colon: the influence of the no-touch isolation technic on survival rates. , 1967, Annals of surgery.

[33]  B. Henderson,et al.  A Prospective Study” , 2007 .

[34]  T. Sekiya Single-Strand Conformation Polymorphism Analysis , 1996 .

[35]  P. Radice,et al.  Genetic evidence for an independent origin of multiple preneoplastic and neoplastic lung lesions. , 1995, Cancer research.

[36]  T. Sekiya,et al.  Allele-specific polymerase chain reaction: a method for amplification and sequence determination of a single component among a mixture of sequence variants. , 1991, Analytical biochemistry.

[37]  L. Sobin,et al.  Histological typing of intestinal tumours , 1976 .

[38]  J. A. Bargen,et al.  Multiple carcinomas of the large intestine: a review of the literature and a study of 261 cases. , 1958, Gastroenterology.