Poly(I:C) promotes the production of IL-17A by murine CD1d-driven invariant NKT cells in airway inflammation.
暂无分享,去创建一个
[1] D. Chaussabel,et al. Herpes simplex virus encephalitis in a patient with complete TLR3 deficiency: TLR3 is otherwise redundant in protective immunity , 2011, The Journal of experimental medicine.
[2] P. Rogers,et al. Invariant NKT cells are required for airway inflammation induced by environmental antigens , 2011, The Journal of experimental medicine.
[3] G. Besra,et al. Innate and cytokine-driven signals, rather than microbial antigens, dominate in natural killer T cell activation during microbial infection , 2011, The Journal of experimental medicine.
[4] S. Su,et al. Toll-like receptor 3 ligand polyinosinic:polycytidylic acid enhances autoimmune disease in a retinal autoimmunity model. , 2011, International immunopharmacology.
[5] U. Švajger,et al. Mechanism of Endosomal TLR Inhibition by Antimalarial Drugs and Imidazoquinolines , 2011, The Journal of Immunology.
[6] F. Annunziato,et al. The TLR7 Ligand 9-Benzyl-2-Butoxy-8-Hydroxy Adenine Inhibits IL-17 Response by Eliciting IL-10 and IL-10–Inducing Cytokines , 2011, The Journal of Immunology.
[7] A. Fryer,et al. Toll-like receptor 7 agonists are potent and rapid bronchodilators in guinea pigs. , 2011, The Journal of allergy and clinical immunology.
[8] Y. Gho,et al. A viral PAMP double‐stranded RNA induces allergen‐specific Th17 cell response in the airways which is dependent on VEGF and IL‐6 , 2010, Allergy.
[9] S. S. Jensen,et al. Differential induction of inflammatory cytokines by dendritic cells treated with novel TLR-agonist and cytokine based cocktails: targeting dendritic cells in autoimmunity , 2010, Journal of Inflammation.
[10] V. Aidinis,et al. Toll-like receptor 7-triggered immune response in the lung mediates acute and long-lasting suppression of experimental asthma. , 2010, American journal of respiratory and critical care medicine.
[11] T. Wynn,et al. Bleomycin and IL-1β–mediated pulmonary fibrosis is IL-17A dependent , 2010, The Journal of experimental medicine.
[12] Jiuhong Kang,et al. MicroRNA miR-326 regulates TH-17 differentiation and is associated with the pathogenesis of multiple sclerosis , 2009, Nature Immunology.
[13] S. Paludan,et al. TLR3 Ligand Polyinosinic:Polycytidylic Acid Induces IL-17A and IL-21 Synthesis in Human Th Cells1 , 2009, The Journal of Immunology.
[14] Kai-ping Yan,et al. iNKT cell development is orchestrated by different branches of TGF-β signaling , 2009, The Journal of experimental medicine.
[15] D. Bani,et al. Modified Adenine (9-Benzyl-2-Butoxy-8-Hydroxyadenine) Redirects Th2-Mediated Murine Lung Inflammation by Triggering TLR71 , 2009, The Journal of Immunology.
[16] T. Chiba,et al. Human TSLP and TLR3 ligands promote differentiation of Th17 cells with a central memory phenotype under Th2‐polarizing conditions , 2008, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.
[17] J. Alcorn,et al. TH17 Cells Mediate Steroid-Resistant Airway Inflammation and Airway Hyperresponsiveness in Mice1 , 2008, The Journal of Immunology.
[18] M. Smyth,et al. Diverse cytokine production by NKT cell subsets and identification of an IL-17–producing CD4−NK1.1− NKT cell population , 2008, Proceedings of the National Academy of Sciences.
[19] M. Nyegaard,et al. Association analysis identifies TLR7 and TLR8 as novel risk genes in asthma and related disorders , 2008, Thorax.
[20] L. Cosmi,et al. Human interleukin 17–producing cells originate from a CD161+CD4+ T cell precursor , 2008, The Journal of experimental medicine.
[21] Angeliki Xagorari,et al. Toll-Like Receptors and Viruses: Induction of Innate Antiviral Immune Responses , 2008, The open microbiology journal.
[22] A. Rachitskaya,et al. Cutting Edge: NKT Cells Constitutively Express IL-23 Receptor and RORγt and Rapidly Produce IL-17 upon Receptor Ligation in an IL-6-Independent Fashion1 , 2008, The Journal of Immunology.
[23] D. Radzioch,et al. Chronic asthma-induced airway remodeling is prevented by toll-like receptor-7/8 ligand S28463. , 2007, American journal of respiratory and critical care medicine.
[24] Yasushi Saito,et al. T-bet inhibits both TH2 cell-mediated eosinophil recruitment and TH17 cell-mediated neutrophil recruitment into the airways. , 2007, The Journal of allergy and clinical immunology.
[25] P. Scholtes,et al. Isolation of CD4+ T cells from murine lungs: a method to analyze ongoing immune responses in the lung , 2006, Nature Protocols.
[26] L. Cosmi,et al. Redirection of allergen-specific TH2 responses by a modified adenine through Toll-like receptor 7 interaction and IL-12/IFN release. , 2006, The Journal of allergy and clinical immunology.
[27] G. Berry,et al. Glycolipid activation of invariant T cell receptor+ NK T cells is sufficient to induce airway hyperreactivity independent of conventional CD4+ T cells. , 2006, Proceedings of the National Academy of Sciences of the United States of America.
[28] Sergio Romagnani,et al. Role for Interferon‐γ in the Immunomodulatory Activity of Human Bone Marrow Mesenchymal Stem Cells , 2006 .
[29] S. Johnston,et al. Asthmatic bronchial epithelial cells have a deficient innate immune response to infection with rhinovirus , 2005, The Journal of experimental medicine.
[30] D. Umetsu,et al. Essential role of NKT cells producing IL-4 and IL-13 in the development of allergen-induced airway hyperreactivity , 2003, Nature Medicine.
[31] Q. Hamid,et al. IL-17 is increased in asthmatic airways and induces human bronchial fibroblasts to produce cytokines. , 2001, The Journal of allergy and clinical immunology.
[32] Hyun-Jeong Ko,et al. A distinct subset of natural killer T cells produces IL-17, contributing to airway infiltration of neutrophils but not to airway hyperreactivity. , 2008, Cellular immunology.
[33] H. Hoymann,et al. Invasive and noninvasive lung function measurements in rodents. , 2007, Journal of pharmacological and toxicological methods.
[34] J. Kolls,et al. Interleukin-17: an emerging role in lung inflammation. , 2003, American journal of respiratory cell and molecular biology.
[35] E. Maggi. The TH1/TH2 paradigm in allergy. , 1998, Immunotechnology : an international journal of immunological engineering.